Ineffective Airway Cleaning: In COPD, smoke or other ecological poisons bother the aviation routes, bringing about hypersecretion of bodily fluid and aggravation. This steady disturbance causes the bodily fluid discharging organs and challis cells to increment in number, ciliary capacity is decreased, and more bodily fluid is created. The bronchial dividers get to be distinctly thickened, the bronchial lumen is limited, and bodily fluid may plug the aviation route (de souza et al., 2013). Weariness, fatigue, disquietude in the end happens and the patient's capacity to expectorate emissions is influenced. Impaired Gas Exchange: Chronic airflow restrictions (brought about by a blend of little aviation route malady) and aviation route
COPD is categorized by poorly reversible airflow obstruction and abnormal inflammatory responses in the lungs. This is primarily due to long-term exposure to noxious chemicals and gases, predominantly tobacco smoke. This augmented response to particulate exposure results in the hypersecretion of mucus, tissue destruction, as well as narrow airway inflammation and fibrosis. These pathological alterations increase resistance to airflow which increases the compliance of the lung, produces air trapping as well as gradual airflow obstruction.
As the number of smokers are rapidly increasing recently, the number of patients with COPD (Chronic Obstructive Pulmonary Disease) is also gradually increasing. It is one of the most common chronic diseases and is considered to be one of the five leading diseases following heart disease, pneumonia, HIV and AIDS worldwide (GOLD, 2004). Smoking is the main cause of COPD. However, long term exposure to chemical fumes and air pollution could also cause COPD. This essay is all about how COPD affects individual, family and society as a whole across their lifespan. Also, it discusses the role of a nurse in caring patients with COPD.
COPD is one of the greatest causes of disability and mortality in the twenty first century with future predictions painting an even graver story. Occupation, genome, and primarily smoking are the main causes of COPD. COPD is the third leading cause of death in America, claiming the lives of 134,676 Americans in 2010. Symptoms are typical of a constant smokers cough which progresses into the debilitating palliative stage of the disease; the development of co-morbidities exacerbates these symptoms. COPD has a complex pathophysiology involving hyperinflation, excessive mucus production and airway remodeling; diagnosis is through lung function tests. COPD is poorly managed with few effective treatments and a poor
First step preventing COPD is to know how it affects the body and the path taken; this is called the pathology. In normal, healthy breathing a person breathes in air that is moves through the bronchial tubes to the bronchioles. At the end of the bronchioles there are air sacs called alveoli. The alveoli are elastic air sacs that control the gas exchange of oxygen and carbon dioxide to the body (Smoking, 2016). The alveoli consist of elastic tissue so when a person breaths in the the alveoli can expand and when the carbon dioxide is released it can deflate and return to it’s normal size.
COPD is caused by smoking, other lung irritants and a deficiency of alpha-1-antitrypsin (Krishnan, Gussin, Prieto-Centurion, Sullivan, Zaidi, & Thomashow, 2013).
A. has a history of smoking for 50 years and being diagnosed with COPD 2 years ago. Development of COPD and its exacerbations may be a leading caused by bacteria, viruses, or environmental pollutants, including cigarette smoke. Coussa, et al, “Expiratory flow limitation (EFL), as a consequence of airway inflammation is the pathophysiological hallmark of COPD.” Exacerbations fundamentally reflect acute worsening of EFL and there is evidence for both increased airway inflammatory activity and worsening airway obstruction as likely explanations.
COPD can be caused by a number of things, but the biggest risk factor is smoking. Other inhalable toxins can also cause
Rationale: L.J. exhibits symptoms of COPD that include shortness of breath and productive cough. He has a smoking history of 65-pack-year and smoking has been identified as the primary cause/trigger of the disease (Nagelmann et al., 2011).
Chronic obstructive pulmonary disease (COPD) (http://www.thehealthsite.com/diseases-conditions/copd/001/) is considered one of the most common causes of death in the world today and, it appears that most of the patients are active smokers. However, according to researchers, the pulmonary disease can affect everybody, not only people who smoke.
Mr. Joseph is a 56-year-old has 30 smoking pack years. He was diagnosed 10 years ago with asthma/chronic bronchitis, arthritis of the knees, and congestive heart failure (CHF). Mr. Joseph weighs 350 pounds with a height of 6 feet, making his body mass index (BMI) of 47.5, much more than the recommended 25, and in fact his BMI places him in the morbid obesity classification. He takes medicines for his pulmonary conditions, along with a diuretic.
As you already know, Chronic Obstructive Pulmonary Disease (COPD), manifests itself when the passageway of air to the lungs is severely obstructed, thus preventing sufficient flow of oxygen into the bloodstream.1 The pathophysiology of COPD is a complex process that is the result of multiple airway diseases that simultaneously contribute to the impairment of airflow in the lungs.1 Specifically, the overlapping outcome of chronic bronchitis and emphysema is the pathogenesis of COPD.1 The risk factor for the COPD is influenced by the individual’s genetics, age, gender, exposure to air pollution, socioeconomic status, and the use of tobacco products.1 The use of tobacco products can increase the development of COPD.1 However, individuals that don’t smoke can also attain COPD.1 Therefore, COPD is not exclusive to individuals that smoke on a daily basis.1 In fact, genetics and the natural aging process plays a part in the development of pulmonary issues.1 For example, it has been proven that a deficiency in the alpha -1 antitrypsin gene is correlated with the development of COPD.1 The natural deterioration of lung tissue, coupled with the long term exposure to environmental elements, explains why the risk of attaining COPD increases as one progresses to the latter stages of their lives.1 In a healthy individual, goblet cells secrete about one liter of mucous that provides a moist surface over the lungs, trachea, and esophagus.1 The cilia on the pseuodocolumnar epithelial cells continuously sweep the mucus in the lungs in an upward motion.1 The cilia sweeps the mucosal trapped debris up, and removes pathogens and other foreign particles out the pulmonary tissue.1 In individuals with COPD, the pathogenesis of the disease creates structural modifications of the lung tissue, which result in deformed and nonfunctioning cilia.2 The lack of functioning cilia leads to the buildup of mucous, pathogens, and subsequent respiratory infections.2 Furthermore, the body tries to combat
Causes of COPD are, but are not limited to, tobacco smoke, occupational working exposure, outdoor and indoor pollutants, genetic factors and early life environmental factors. This disease has become the third leading cause of death in the United States of America. The only proven way to slow the progression is to QUIT smoking. Some studies have shown that giving a bronchodilator has reversed some lung function which benefits the patient. Symptoms of COPD include, but are not limited to, dyspnea upon exertion, chronic cough with or without phlegm, fatigue and weight loss. Some common characteristics of COPD are exacerbations and episodes of acute worsening. Exacerbations are commonly caused by viral and/or bacterial infections and pollution. Multimodal treatments are used and include
Although not curable, chronic obstructive pulmonary disease (COPD) is a familiar, treatable, but avoidable disease that is still a huge health problem in the United States. According to the Global Initiative for Chronic Obstructive Lung Disease, “COPD is the fourth leading cause of chronic morbidity and mortality in the United States and is projected to rank fifth in 2020 as a worldwide problem according to a study published by the World Bank/World Health Organization.”1 Mortality from COPD is also expected to increase despite medical advances in the treatment of the disease.
It is very interesting how environmental factors and genetics can affect ones health. Throughout my experience as a nurse aid, I have observed that the use of inhalers have become very common in our society. I always questioned what health conditions cause the use of in halers other than asthma. Very surprisingly, people who don’t even smoke can be affected by just inhaling the smell around others who smoke. Chronic obstructive pulmonary disease (COPD) is one of the health conditions that are cause by inhaling environmental pollutants which can result in asthma as well. One may ask, what exactly is COPD? It is the type of pulmonary disorder such as emphysema, chronic bronchitis in which the upper airway is chronically obstructed. The condition
Chronic Obstructive Pulmonary Disease, also known as COPD, is the third leading cause of death in the United States. COPD includes extensive lungs diseases such as emphysema, non-reversible asthma, specific forms of bronchiectasis, and chronic bronchitis. This disease restricts the flow of air in and out of the lungs. Ways in which these limitations may occur include the loss of elasticity in the air sacs and throughout the airways, the destruction of the walls between air sacs, the inflammation or thickening of airway walls, or the overproduction of mucus in airways which can lead to blockage. Throughout this paper I am going to explain the main causes, symptoms, diagnosis, and ways to reduce COPD.