Lyme disease is also called Lyme borreliosis. It is caused by a bacterium called Borrelia burgdorferi, and transmitted by the black-legged tick, Ixodes scapularis, in the nymph stage (J.F. & Magnarelli, 1993). Therefore, the infected nymph’s density is the most specific ecological indicator of the risk of Lyme disease (J.F. & Magnarelli, 1993). It is an enzootic disease, although humans have been infected as accidental hosts, making it a zoonotic disease (G. & Fish, 1993).
The Peromyscus leucopus, the white- footed mice, is the principle reservoir for the disease transmission to the black-legged tick (Krohne, D.T., & Hoch, 1999). Where the disease originates from, why it has emerged so rapidly and what can be done to reduce the risk of exposure
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Nymph density was 0.1/m2 in average in the four smallest fragments and decreased to an average of 0.03/m2 in the larger fragments. NIP and DON were significant negative functions of the patch area. The infected nymph’s percentage decreased from 70% on average in smallest fragments to 48% in average in larger fragments.
Discussion
In highly fragmented landscapes, the nymph’s infection prevalence, the density of infected nymphs and density of nymphs were correlated inversely with forest patch area (Nupp, T.E., & Swihart, 1998). These metrics of the population of nymph stages of the ticks are ecological indicators of the risk of Lyme disease (Ostfeld, R.S.., & Keesing, 2000).
The elevated exposure risk in the smallest patches results from two phenomena. Firstly, biodiversity loss that accompanies fragmentation of forests decreases the tick host’s abundance that is poor reservoirs for the B. burgdorferi. Low biodiversity reduces the tick meal’s fraction taken from the poor reservoirs and increases NIP (M' Closkey, 1999). Secondly, from the smallest fragments, the vertebrate species lost are competitors and predators of the main reservoir host, white-footed mice (M' Closkey,
What became known as Texas tick fever probably arrived in North America sometime in the seventeenth century carried by cattle brought by Spanish colonists from the West Indies. It was transmitted by the cattle tick which required 200 or more frost free days per year for survival. Therefore, its spatial extent was the southern states. Initially, southerners were not terribly concerned about the disease since cattle was rarely a primary economic investment in the southern United States. Antebellum Texas was an exception since it dominated the national cattle industry. By the 1850s farmers in states such as Missouri were turning back cattle drives from Texas for fear of infecting their herds. In some instances there were violent confrontations, assaulting and even killing some of the cowboys. Eventually northern states intervened to protect their herds, instituting quarantine laws against southern cattle. In 1892 the federal government delineated a quarantine line that followed the northern border of permanent fever infestation. The accompanying regulations stated that cattle could only be moved to northern markets between
According to Dr. Dietrich Klinghardt (a world renowned Lyme expert), Lyme disease is considered the plague of the century due to the large number of people with chronic Lyme disease and the associated co-infections. The number one symptom of Lyme is chronic fatigue. People can have Lyme for a very long time and not be properly diagnosed because current laboratory testing comes up short with being able to detect it. One of the main reasons for this is because chronic Lyme does not live in the blood but rather in the tissues. It is an intracellular organism meaning it lives inside the cell. Lyme has different shapes: spirochete (like a spring), cyst with a hard shell, and acellular (without an outer cell membrane). Lyme likes to set up a sanctuary in the body such as in the brain, teeth (such as under a root canal or in a cavitation), vertebrae, breast tissue, and lymph nodes. It looks for a place where it can be protected and the body is vulnerable preferably in fatty tissue or under a biofilm. While traditionally Lyme disease is considered a tick-borne disease, recent findings have discovered that it is an insect-borne disease that can get transferred
They contract it when feeding on infected hosts. While deer are most commonly known for giving Lyme disease to ticks, rodents are the main threat. “Ostfeld’s research indicates that white-footed mice infect 75-95 percent of larval ticks that feed on them, while deer only infect about one percent” (Mercola). Urban sprawl and hunting has eliminated many of the mice’s natural predators allowing populations to grow and infected ticks to spread.
Lyme disease is an infection produced by bacteria called Borrelia Burgdorferi. This bacteria or germ is ordinarily found in shrews, deer, mice, and squirrels. Ixodes bugs, normally called deer ticks, often feed on the blood from an infected animal. When this happens, the tick then becomes a carrier of the bacteria that causes Lyme disease and can infect you with this germ through your skin. Infected deer ticks are normally found in the northeast and upper Midwest United States because of the climate and humidity levels, and become more active in the late spring and early summer months after the birth of new larvae. Lyme disease is known to cause a skin rash called erythema migrans and can leave you problems with your joints, brain, heart, and nerves. The
This journal article gives a broad overview of tick borne relapsing fever. It explains the causative agents and here they are respectively found and as well as the vectors and their environments. The article also shows an epidemiological spot map of the U.S. and TBRF cases.
Most Lyme disease cases are very serious. The disease creates symptoms that are similar to the flu. When you first catch the disease you may experience fever, chills, sweats, muscle aches, fatigue, nausea and general pain. Very rarely, Lyme disease may be fatal depending on which strain of the disease you caught.
“Borrelia burgdorferi, the bacterium that causes Lyme disease, was first isolated in 1982 by Willy Burgdorfer, Ph.D., a zoologist and microbiologist at NIAID’s Rocky Mountain Laboratories (RML) in Hamilton, Montana.” (NIAID, 2008.) Dr. Burgdorfer at the time was studying Rocky Mountain Fever in attempt to help Jorge Benach, Ph.D., discover why more than 100 spotted fever cases occurred in New York between 1971 and 1976. “Dr. Benach, of the New York State Department of Health had been a source of American dog ticks for Burgdorfer to study.” (NIAID, 2008.)
“Lyme disease got its name from where it was first discovered in Lyme, Connecticut in 1975” (healthychildren.org). “Since then there has been thousands of cases reported of the disease. The ticks live in low and high seasonal temperatures and high humidity” (healthychildren.org). Lyme Disease most common to occur in the Northeast, North-central states, or West Coast. On behalf of the Northeast, I can tell you that ticks are everywhere in this region. They like to live in tall grasses and woody areas. “The small insects more than often latch onto the foot or lower leg and crawl up the body, and travel up the body with a destination of the head. On their normal hosts, ticks also usually crawl up; they want to blood feed around the
Thesis Statement: “According to the Center of Disease Control, Lyme disease is the most commonly reported vector borne illness in the United States”(Lyme Disease: Data and statistics, 2016).
Your chances of acquiring Lyme disease can be greatly affected by where you live or travel to. Many outdoor activities put you at risk. Since deer ticks feed on mice and white-tailed deer, they are most commonly found in heavily wooded areas. Another risk factor of Lyme disease is having exposed skin. Since ticks attach easily to bare skin, being exposed in wooded or grassy areas will increase your risk. If you are bitten by a tick, the final chance of prevention is to remove the tick quickly and properly. In most cases, to infect it’s host, a tick must remain attached to the skin for at least thirty-six hours. If you safely remove the tick
Lyme borreliosis or Lyme disease can be devastating both physically and mentally for victims. The disease is transmitted by a vector, specifically a tick, which infects their host by biting and infecting them with pathological spirochete bacteria known as Borrelia burgdorferi (Muschart & Blommaert, 2015). There are several species of Borrelia globally, and as a group, the bacteria are referred to as Borrelia burgdorferi sensu lato (Pearson, 2015). The disease originated from, Lyme Connecticut, of which it was named, after a patient was mistakenly diagnosed with rheumatoid arthritis when the cause of their disease was B. burgdorferi or Lyme disease (Snow, 2013). Lyme disease is classified as one of the most common and growing vector-born disease in the United States as well as Europe (Snow, 2013). Understanding how Lyme disease progresses through the body as well as the disease’s signs and symptoms are essential to understanding how to treat the disease and stop it from spreading throughout the body with destructive results. With a disease as potentially devastating as Lyme disease, early treatment is required for a better outcome (Pearson, 2015). This research paper will discuss the pathophysiology of three Lyme conditions known as Lyme arthritis, Lyme neuroborreliosis, and Lyme carditis and provide an overview of the signs and symptoms of Lyme disease and treatments, and how to prevent infection.
Lyme disease has been a problem for those living in and around the New England area for the last few decades. Unfortunately, researchers have just discovered that the deer ticks are expanding their reach, with a surge over 320 percent. The ticks are slowly branching out into the connecting counties and states. It is believed that they are branching out due to climate change.
Lyme disease is transmitted to humans from a natural reservoir (rodents and deer) by ticks that feed on both sets of hosts. Most infections are caused by ticks in the nymphal stage. Ticks in this stage
Zoonotic diseases, diseases that can be passed between animals and humans, are very common and pose a significant threat to public health. Strategies to address health threats of Lyme disease, the most prevalent tick-spread disease in the northern hemisphere, often have only modest success due to an inclination to focus on the white-footed mouse, Peromyscus leucopus, as the primary host for ticks and the primary reservoir for the Lyme disease bacterium B. burgdorferi ss. Brisson and colleagues examined how shrews can play an even more important role as the primary host and reservoir than mice. Their findings suggest that the most successful Lyme disease mitigation approaches will be those that target multiple host species.1 Shrews have high reservoir potential, which makes them a primary host of Lyme disease and allows them to contribute significantly to the Lyme disease epidemic. Brisson et
The symptoms of lyme disease is normally a fever, headache, chills, fatigue, and a rash. Early symptoms might include a rash that is an expanding red area, it forms center of a bulls eye pattern type. Sometimes it is called ‘erythema migrans’, which refers to a rash. The later symptoms will include joint pain, swelling, vomiting and nausea. It might also cause some neurological problems like inflammation surrounding the brain.