Respiratory Examination I then needed to carry out a respiratory assessment. I observed Mr Brown’s chest for any visible signs of scars or trauma. This appeared normal. His chest was normal shape, with no signs of barrel or pigeon chest. With a barrel chest, the shape is abnormally round and bulging. This can be the result of a patient suffering from COPD. A Patient with a pigeon chest has a sternum that protrudes beyond the front of the abdomen (Publications, 2007). I also observed his ease of breathing there was no sign of him using his accessory muscles to aid breathing (IBID). The frequent use of accessory muscles can indicate a respiratory problem especially if the patient purses their lips and flare their nostrils when breathing (Publications, 2007). When carrying …show more content…
On palpation there was no sign of sacral or peripheral oedema. This is assessed by looking for the size and colour of his ankles an also seeing if they are bilaterally equal size, then gently pressing the skin with your finger to see if an indentation is left, which will slowly refill as the fluid returns (IBID). Percussion The reason for percussing a chest is to set up vibrations which then become audible. This can then aid to assess areas of varying density. The presence of fluids, solids or air will produce different resonances. Resonant is normal lung sound (Cross and Rimmer, 2007). Dull sound can mean fluid or tissue filled cavity Hyper resonant can indicate air trapping in lung cavity or pleura space (IBID). I found nothing abnormal I then went onto percuss his chest in the appropriate places (see appendix 2 for places for percussion) using my two fingers to tap on my second finger, which I had placed over his intercostal spaces. I was observing for Resonance, hyper resonance dull and very dull sounds Nothing abnormal
A chest radiograph showed a rounded, well-defined soft tissue density measuring 4.5 x 4.2 cm located in the inferior aspect of the left hilum at the level of the mid chest. The chest x-ray was otherwise unremarkable. Computed tomography of the chest revealed a left hilar 3.2 x
Lungs: Diminished breath sounds in all lung fields. Resonant to percussion. No wheezes, rales, or rhonchi. Symmetric chest expansion. Breathing nonlabored.
According to Egan’s, ventilation is the process of moving gas (usually air) in and out of the lungs Egan’s 225). If a patient lungs sounds are normal, the respiratory care practitioner is not concerned due to decrease in obstruction in the airway. Contrary, if the respiratory care practitioner listens to sounds like wheezes, stridor, rhonchi, rales, the practitioner is mostly concerned due to the fact that an increase in obstruction is either in the upper or lower airway. This increase obstruction could lead to decrease tissue oxygenation by causing tissue hypoxia, cyanosis, stagnant or a disease such as chronic bronchitis.
Lungs: Diminished breath sounds in all lung fields. Resonant to percussion. No wheezes, rales, or rhonchi. Symmetric chest expansion. Breathing nonlabored.
Lungs: Clear to auscultation bilaterally. No wheezes, rales, or rhonchi. Symmetric chest expansion. Breathing nonlabored. Diminished breath sounds in all lung fields. Resonant to percussion.
Lungs: Clear to auscultation bilaterally. No wheezes, rales, or rhonchi. Symmetric chest expansion. Breathing nonlabored.
Percussion: I would put my two fingers on the area of my patient and tap directly on the body part and ask my patient if they are in pain, and I would also focus on their verbal and non-verbal to determine how painful the area is. This technique would help me notice any tenderness, shape, and position of the organs. I have checked the abdomen and is not solid and it is not full of gas or fluid. My finding would be documented.
Both of his lung fields appear clear, with normal breath sounds, no signs of pulmonary consolidation and other abnormalities detected. His apex beat show an extra heart sound (ectopic heartbeat) which is due to the narrowing of his blood vessels that connects to the heart and to the lungs, which often occurs without a clear cause and are harmless, with no signs of murmur or split heart sounds. His abdomen is significantly distended with presence of bulging flanks pushed outwards with no signs of pain or rebound tenderness on deep palpation, and when testing for shifting dullness. He was placed in a lateral decubitus position to assess fluid shift and had a positive result. Upon percussion of Frank’s abdomen in the supine position for flank
One of the patient’s main injuries is the 12 cm contusion on his left axial/anterior chest with 8/10 pain.
responder should inspect the chest for any injuries and palpate the chest for any abnormalities of
Knowlton has had insidious progression of dyspnea on exertion over the course of one to two years. His pulmonary function tests do demonstrate a restrictive ventilatory defect with significant decrement to his DLCO. He has a very significant smoking history. However by imaging, the is no evidence of any emphysematous disease. Additionally, there is no evidence of obstruction on his pulmonary function tests. The differential diagnosis for restrictive ventilatory defects are quite broad to include intrinsic lung disease versus neuromuscular weakness verus non-muscular diseases of the upper thorax to include kyphoscoliosis. It should be noted that Mr. Knowlton does have a fair amount of stooped posture and kyphosis on examination, however this should not contribute to the level of decreased DLCO noted on his pulmonary function tests. At this time, it would be reasonable to interrogate the lung parenchyma with high resolution CT scanning given the pulmonary function tests
Chest X-rays are difficult to interpret, but you did a great job. The ABCD systemic approach was applied appropriately, the trachea is visible and in a correct alignment, the bones are intact, no signs of fracture, the heart is enlarged, however, the diaphragm is not visible because of the disease condition (Wells, 2013). Although, congestive heart failure could be assumed as the diagnosis looking at the radiograph, but the importance of getting the signs and symptoms, the chief complaint, and the patient’s medical history cannot be overemphasized.
My chest started looking deep around the age of ten-twelve. I hated taking my shirt off to swim, it hurt to run and felt sick more often then what I should. So I went to the doctor about it and they gave me and my mother some phone numbers to some specialist for this type of condition. We call Dr. Hunter in Chicago, Illinois at Loris Children's Hospital. The bad thing was, I walked the horrific surgery, you could hear the bones snapping in place.
Mier-Jedrzejowicz et al found that patients with the most severe orthopnea had the weakest diaphragms.44 In these patients, the physical therapist should also look for the abdominal paradox. In this clinical sign the weakened and flattened diaphragm contracts inwardly, as opposed to inferiorly, pulling the rib cage with it. It is a result of the change in the orientation of the diaphragm, the loss of the ZOA and its resultant ineffectiveness in moving the rib cage outwardly. Urmey/De Troyer/Kelly/Loring45 The abdominal paradox will most likely be identified in these patients in sitting and
thoracic cavity and lungs. Quiet breathing in an unconscious effort where it is done without