Symptoms And Treatment Of Copd

According to the provider, the claimant's cough has been improved. His review of systems was positive for fatigue, malaise, sleep difficulty, shortness of breath, wheezes, and a cough. His blood pressure was 115/71 mmHg and his BMI was 30.35 kg/m2. The physical examination revealed wheezes. Clonazepam was prescribed for agitation. Atorvastatin, Nystatin, Citalopram, and a probiotic were prescribed. Continued use of Aspirin and a regular inhaler were suggested. Further, a follow-up visit with Endocrinology, Cardiology, and Pulmonology. As it relates to a spot in his lung, a repeat CT scan was recommended. The bronchial washes were negative for

Data: Pulmonary function testing dated 2010 showed moderately severe obstruction with positive bronchodilators response. Normal lung volumes. Evidence of air trapping. Severely reduced diffusing capacity for carbon monoxide.

Have you ever known a person who smokes and has a hard time doing every day activities, due to difficulty of breath, or constantly coughing. He or she may have Chronic Obstructive Pulmonary Disease, or COPD. COPD is a progressive and treatable lung disease that causes shortness of breath due to obstruction of air way (COPD, 2013). Progressive means that is gradually gets worse over time. It is a combination of chronic bronchitis and emphysema (Causes,2014). Chronic bronchitis is inflammation of the bronchioles, which causes mucus build up (Davis,2016). Emphysema is when the air sacs get enlarged (Smoking, 2016). Since the disease does not have a cure yet it is important to know pathology (path of disease), epidemiology (who is effected in a population), ethology (who is effected genetically), manifestation (symptoms), treatment, and outcome.

History of Present Illness: Mr. Magnuson is a very pleasant 77-year-old gentleman who was previously seen in this office by Elvira Aguila, MD for COPD and hypoxic respiratory failure. He is here today for routine followed up. He was last evaluated in January 2015. Since that time, he states that his dyspnea is worse. He feels that it is related to the weather. He does state that he works around the house, although he does have significant functional limitations because of shortness of breath. His wife also confirms that he is able to do less and less. He has a stable, minimal cough. He is using 4L of oxygen at night as well as, as needed throughout the day. He continues to smoke three to four cigarettes on

COPD is a chronic inflammation of the airways leading to fixed airflow obstruction and restricted gas exchange. The term COPD includes two basic respiratory pathologies: chronic bronchitis and emphysema (GOLD, 2006) that are described below. The condition is not reversible and may progressively worsen with time (NICE, 2010).

Chronic obstructive pulmonary disease (COPD) is progressive disease affecting the lungs. It is characterized by airflow limitation that is not fully reversible, as well as the abnormal inflammatory response of the lungs to noxious particles and gases (Girod & King, 2005). Smoking is a major risk factor for both the mortality and morbidity of COPD. Other causes may include: bronchial hyper-responsiveness, indoor and outdoor air pollution, allergy, biomass fuels and smoke exposure for women cooking in poorly ventilated conditions (Mclvor, Tunks & Todd, 2010 and Calverley & Walker, 2003). Current treatment approaches include: prevention of disease progression, management of stable disease and management of exacerbations (Calverley et al., 2003).

Chronic obstructive pulmonary disease (COPD) is a broad term for people who suffer the chronic respiratory diseases chronic bronchitis and emphysema. Chronic bronchitis is inflammation of the small airway passages that leads to reduced airflow. Emphysema is the destruction of the alveolar walls resulting in minimised gas exchange and limiting airflow through the airways11. COPD is a progressive disease that can be attributed to an abnormal inflammatory response from the lungs as a result of long term exposure to noxious particles and gasses, primarily caused by cigarette smoking.

COPD is characterized by chronic inflammation found in the airways lung parenchyma, and pulmonary vasculature (Huether and McCance, 2012). The pathogenesis of COPD is complex and involves many mechanisms. However, the primary process is inflammation (Huether and McCance, 2012). The inflammatory process starts with inhalation of toxic particles and gases. The airways become inflamed, resulting in excess mucus production; Peripheral ways undergo repeated cycles of injury and repair of the airway walls with resultant structural remodeling (Huether and McCance, 2012). The lungs can be inflated quickly but can only partially deflate.

Chronic Obstructive Pulmonary Disease, also known as COPD, is a disease affecting the respiratory system that causes obstructed airflow from the lungs. It is caused by long-term exposure to irritating gasses, with cigarette smoke being the most common cause. Contributing factors of COPD include emphysema, a condition where the alveoli at the end of the bronchioles are destroyed as a result of exposure to irritating gasses, and chronic bronchitis, which is an inflammation of the bronchial tubes, which carry air to and from the alveoli. As well, those who develop COPD are at a higher risk for heart disease, lung cancer, and various other conditions than those without the disease.

On January, 31st, Patient F.F. arrived to the emergency room in the hospital with her brother due to an increased temperature for ‘the past 3 days,’ fatigue, and was ‘unable to catch [her] breath.’ A focused assessment revealed crackles and wheezes in the lower lobes of the lungs. The patient was leaning over in a tripod position and breathing heavily between words. The patient’s heart sounds were normal with a regular S1 and S2. The patient denied having chest pain and edema was not present. The patient reported having a productive cough with green sputum for the past 3 days. Vital signs were taken and the patient’s oxygen saturation was 88%. The doctor ordered 2 liters of oxygen by nasal cannula for the patient with a continuous

Patients present with shortness of breath, cough, sputum production, airflow limitation, wheezing, chest tightness, dyspnea during exertion, fatigue, dizziness, elevated shoulders, overuse of accessory breathing

Patient TM is a 61-year-old female patient with PMH of COPD, Hypertension, asthma and type II Diabetic who presents to the clinic with a complaint of Shortness of breath for the past 2 Days. The patient states that the SOB was sudden in onset and progressive. It was 8/10 in severity. Occurs with minimal activity. The patient states that he has been using his rescue inhalers but is not getting any relief. The patient states she has severe exacerbations of COPD around once or twice a year. She states that she is coughing up a small amount of clear sputum with no foul smell. Denies fever. Denies chest pain no palpitations. The patient TM is a chronic smoker. She has three pack years history of

On examination, he was saturating at 96% on room air. His lung fields revealed reduced air entry but no significant wheeze or

Chronic obstructive pulmonary disease (COPD) is a chronic lung disease that causes obstructed airflow from the lungs. Symptoms of COPD patients are shortness of breath (SOB) that causes limiting of the patient’s activities and lack of energy, hard cough, massive sputum production, blue lips and fingernails (cyanosis), losing weight, fatigue, swelling in feet (edema), and wheezing. COPD is a disease caused by a long history of smoking cigarettes. COPD patients are more at risk for diseases such as heart disease, lung cancer, and other conditions and diseases. The two most common diseases related to COPD are emphysema and chronic bronchitis. Tobacco smoking is the main cause of COPD in developed countries because people are close to chemical fumes, dust, and smoke from fuel in poorly ventilated homes. Many

On the initial presentation to Emergency department with worsening dyspnoea, dry cough and neck tightness. The patient was afebrile, acyanotic, tachypnoiec (respiratory rate of 36 per minute), tachycardic (heart rate of 166 per