Alcohol affects the central nervous system by enhancing the effect ofGABA at its receptor. GABA binds to GABA receptors and opensligand-gated Cl–channels. However, chronic consumption of alcoholmakes the GABA receptor less sensitive to both alcohol and GABA,which increases alcohol dependence as well as alcohol withdrawalsymptoms, such as anxiety, tremors, and insomnia. Benzodiazepinesenhance the binding of GABA molecules to their receptors and thus aresometimes used to treat people with alcohol withdrawal symptoms. Forsynapses involving GABA, predict the effect of alcohol on the postsynaptic membranes; compare the effect of chronic alcohol consumption onthe postsynaptic membranes in these synapses; and predict the effect ofbenzodiazepine treatment on the degree of polarization of postsynapticmembranes in people who are experiencing alcohol withdrawalsymptoms.
Alcohol affects the central nervous system by enhancing the effect of
GABA at its receptor. GABA binds to GABA receptors and opens
ligand-gated Cl–
channels. However, chronic consumption of alcohol
makes the GABA receptor less sensitive to both alcohol and GABA,
which increases alcohol dependence as well as alcohol withdrawal
symptoms, such as anxiety, tremors, and insomnia. Benzodiazepines
enhance the binding of GABA molecules to their receptors and thus are
sometimes used to treat people with alcohol withdrawal symptoms. For
synapses involving GABA, predict the effect of alcohol on the postsynaptic membranes; compare the effect of chronic alcohol consumption on
the postsynaptic membranes in these synapses; and predict the effect of
benzodiazepine treatment on the degree of polarization of postsynaptic
membranes in people who are experiencing alcohol withdrawal
symptoms.
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