Alzheimer’s disease is a devastating neurodegenerative disease. Discuss approaches used to study the disease, giving examples of specific techniques and models, and evaluating their advantages and disadvantages. Discuss possible reasons for why effective treatments have not yet been developed. Use figures and diagrams to illustrate your answer. (This is a neuroscience question)
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Alzheimer’s disease is a devastating neurodegenerative disease. Discuss approaches used to study the disease, giving examples of specific techniques and models, and evaluating their advantages and disadvantages. Discuss possible reasons for why effective treatments have not yet been developed. Use figures and diagrams to illustrate your answer.
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- Scenario: You are the nurse doing home visits in a retirement community. Your client an 85-year-old man who has been diagnosed with Alzheimer’s disease (AD). His adult children are with him for the visit. They want to know about the disease and what treatment options exist. After you explain the progression of the disease, they ask if is curable. How should you answer?A. There are medications that may help delay cognitive decline, but the disease is not curableB. If treatment is started early, the disease progression ca be reversed.C. Drugs exist that can cure AD, you just have to find the one that works for youD. If treatments in initiated before complete neuronal damage, disease progression can be stopped.Discuss the concept of brain-computer interfaces (BCIs) and their applications in medical fields like neurorehabilitation and communication for individuals with severe disabilities.Histological and brain imaging methods can be used to detect or confirm Alzheimer’s disease. Select ALL that apply: Functional MRI imaging of neuronal activity during episodic memory tasks shows increased activity in the hippocampus. The sulci appear larger The gyri appear thicker The lateral ventricles are enlarged Functional imaging shows a decreased metabolic activity in the temporoparietal cortex at resting states indicating defects in episodic memories
- Alzheimer's Disease Alzheimer's disease is a progressive neurodegenerative disorder that affects almost 50 million people worldwide. The cause of Alzheimer's disease is still not well understood. It is thought to be a combination of genetics, lifestyle, and environmental causes. It is also difficult to diagnose Alzheimer's disease during the initial stages of the disease as symptoms can be mistaken for normal ageing. Currently, there is no cure for the disease. Medication can help manage the symptoms but degeneration of brain tissue cannot be stopped or reversed. Currently, the medication that manages the symptoms of Alzheimer's disease is a type of cholinesterase inhibitor. In other words, the medication inhibits the function of cholinesterase and helps in brain functioning and memory. What is the function of cholinesterase in the synapse? How does a cholinesterase inhibitor work in a synapse? Explain how cholinesterase inhibitors are able to increase brain functioning and…Should the Canadian government increase the funding for Alzheimer's research? Choose one of the following viewpoints to answer this question: social or economical Clearly state your position on this issue Give and explain two reasons to support your viewpoint. Use research based evidence to support each reason. IAlzheimer's disease is a progressive neurological degenerative disorder that affects almost 50 million people worldwide. The cause of Alzheimer's disease is still not well understood. It is thought to be a combination of genetics, lifestyle, and environmental causes. It is also difficult to diagnose Alzheimer's disease during the initial stages of the disease as symptoms can be mistaken for normal aging. Currently, there is no cure for the disease. Medication can help manage the symptoms but degeneration of brain tissue cannot be stopped or reversed. One medication used to manage the symptoms of Alzheimer's disease is a cholinesterase inhibitor. The medication helps to improve brain functioning and memory. What is the function of cholinesterase in the synapse? How does a cholinesterase inhibitor work in a synapse? Explain the function of cholinesterase inhibitor in increasing brain functioning and memory
- Could you please mention/explain some limitations and extensions (three each) associated with the following claim and research question. Claim: Gene therapy can reverse ageing. Research question: How does the injection of BDNF in the brain through gene therapy help treat Alzheimer's disease in people over the age of 65? please asapDescribe the symptoms, pathology, and treatment of Alzheimer’s Disease at the introduction level (Introduction to Neuroscience College Level). Provide diagrams to complement your explanation.If an accurate diagnostic test for Alzheimer’s disease is developed and there is no treatment for the disease, should the test be made available?
- You are the clinician in-charge of a drug development team targeting neurodegeneration. You are asked to give a presentation on your proposed research strategy. What area(s) of the brain would you recommend as the best target(s) for a neurogenic drug in adults Select one: a. Thalamus b. Hypothalamus c. Prefrontal cortex d. Hippocampus e. corpus callosumNeurological disorders are the leading cause of disability worldwide and the absolute numbers of person affected are increasing. What are the reasons for this? What effect will this have on health care systems? What do we know and not know about the burden of neurological disease and what kinds of information do we need to move forward in prevention and treatment?Scenerio: Is Cognitive Processing Therapy (CPT) more effective in treating PTSD than Prolonged Exposure therapy (PE)? Question 1: Does this question represent a correlational or experimental research design? Why? Question 2: Which of the two models would state that there is nothing interesting going on between CPT and PE in treating PTSD? Question 3: . State the alternative hypothesis for this study. Question 4: State the null hypothesis for this study. Question 5: Are your hypotheses directional or non-directional? How do you know this? Question 6: Suppose you run the study, and the results come out with a p value of .23. What can you conclude about the difference between the treatments? Question 7: In this case, would you decide to reject or fail to reject the null hypothesis? Why?