Now in your own words, EXPLAIN (1) howa neural impulse is transferred to a muscle cellthis is the process of “muscle excitation"), and (2) how the electrical impulse travels intohe T-tubules and causes calcium to be released from the sarcoplasmic retículum (this is theprocess of “excitation-contraction coupling"):OMADidoinosreticulum5.se moS1ehospherahanism

Neural impulse or a nerve impulse refers to a signal that is transferred along the nerve fiber. It…

Answered: Now in your own words, EXPLAIN (1) howa…

BIOLOGY:CONCEPTS+APPL.(LOOSELEAF)

10th Edition

ISBN:9781305967359

Author:STARR

Publisher:STARR

Chapter32: Structural Support And Movement

Section: Chapter Questions

Problem 2CT: After death, a person no longer makes ATP, so calcium stored in the sarcoplasmic reticulum diffuses...

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Every skeletal muscle fiber is supplied by a motor neuron at the NMJ. Watch this video (http://openstaxcollege.org/l/skelmuscfiber) to learn more about what happens at the neuromuscular junction. (a) What is the definition of a motor unit? (b) What is the structural and functional difference between a large motor unit and a small motor unit? Can you give an example of each? (c) Why is the neurotransmitter acetylcholine degraded after binding to its receptor?
After death, calcium pumps no longer function and the calcium ion concentration of muscle fiber cytoplasm increases. The result is rigor mortis a state or postmortem muscle contraction. Explain why this contraction occurs and why it ends only when myosin heads begin to break down.
Watch this video (http://openstaxcollege.org/l/micromacro) to learn more about macro- and microstructures of skeletal muscles. (a) What are the names of the junction points between sarcomeres? (b) What are the names of the subunits within the myofibrils that run the length of skeletal muscle fibers? (c) What is the â€œdouble strand of pearlsâ€� described in the video? (d) What gives a skeletal muscle fiber its striated appearance?
The muscle relaxes if no new nerve signal arrives. However the neurotransmitter from the previous stimulation is still present in the synapse The activity of helps to remove this neurotransmitter. myosin action potential tropomyosin acetylcholinesterase
Which of the following step(s) about the excitation of skeletal muscle is/are incorrect? 1. Acetylcholine is released and binds to motor end plate receptors 2. An action potential is created and moves down T-tubules 3. Calcium is released from the sarcoplasmic reticulum 4. Calcium ions bind to tropomyosin to shift troponin off the binding sites for cross-bridging 5. Myosin forms cross-bridges and binds with actin to pull it towards middle of sarcomere
Why is the latent period longer during an isotonic twitch of a skeletalmuscle fiber than it is during an isometric twitch?a. Excitation–contraction coupling is slower during an isotonic twitch.b. Action potentials propagate more slowly when the fiber is shortening,so extra time is required to activate the entire fiber.c. In addition to the time for excitation-contraction coupling, it takesextra time for enough cross-bridges to attach to make the tension in themuscle fiber greater than the load.d. Fatigue sets in much more quickly during isotonic contractions, andwhen muscles are fatigued the cross-bridges move much more slowly.e. The latent period is longer because isotonic twitches only occur in slow(type I) muscle fibers.
3.4. About human skeletal muscle contraction, what are the correct statements? PHYSIOLOGY_basic (OJO) A muscle fiber is made by the parallel apposition of sarcomers. Generation of force relies on the formation of cross-bridges between actin and myosin filaments. In the process of contraction, adenosine triphosphate (ATP) is required both to drive the actin-myosin association-dissociation cycle and to recycle calcium back to the endoplasmic reticulum. Each muscle fiber is innervated by one neuron only. T tubules are plasma membrane invaginations of the sarcolemma that allow excitation-contraction coupling.
Hi, I only need the answer for this particular question, if that's ok: The swo-1 a mutation leads to worms with more body wall muscle, whereas worms with the swo-1 b mutation are not able to move. Based on these phenotypes and the findings from questions 3 and 4, describe the role thewild-type version of this protein plays in muscle function. Thank you!
While the sarcoplasmic reticulum of muscle was first described by nineteenth-century microscopists, its true significance was not ap-preciated until its intricate structure was revealed much later by the electron microscope. What could you tell a nineteenth-century microscopist to enlighten him or her about the structure of the sarcoplasmic reticulum and its role in the coupling of excitation and contraction?
Explain in detail how the of relaxion towards Smooth muscle contraction is done by the antagonsists atropine and mepyramine. Provide intracelluar mechanism of relaxation.
Calcium is the trigger for contraction of all muscle types. How does its binding site differ in skeletal and smooth muscle fibers?
The disease poliomyelitis (po′le-o-mi′˘e-li′tis) destroys motor neurons, causingloss of muscle function and even flaccid paralysis. Some patients recoverbecause axon branches form from the remaining motor neurons. Thesebranches innervate the paralyzed muscle fibers to produce motor unitswith many more muscle fibers than usual. How does this reinnervation ofmuscle fibers affect the degree of muscle control in a person who hasrecovered from poliomyelitis?

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