Select the correct statements An oncogene is a cancer producing gene A proto-oncogene is only found in cancer cells Tumor suppressor genes are genes whose normal products inhibit cell division A proto-oncogene is a normal cellular gene that has the potential to become an oncogene

Select the correct statements An oncogene is a cancer producing gene A proto-oncogene is only found in cancer cells Tumor suppressor genes are genes whose normal products inhibit cell division A proto-oncogene is a normal cellular gene that has the potential to become an oncogene

Human Heredity: Principles and Issues (MindTap Course List)

11th Edition

ISBN:9781305251052

Author:Michael Cummings

Publisher:Michael Cummings

Chapter12: Genes And Cancer

Section: Chapter Questions

Problem 18QP: You are in charge of a new gene therapy clinic. Two cases have been referred to you for review and...

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Please answer fast and all question other wise I will give downvote. QUESTION 1 Why is cancer a disease of old age ? A. Older DNA is more easily damaged B. There are fewer stem cells to repair tissue in older people C. DNA mutations accumulate with age QUESTION 2 The frequency of this molecular change in cancer cells makes it readily detectable by liquid biopsy. A. Point mutations B. Gene amplification C. DNA methylation D. PSA antigen QUESTION 3 Can surgery (with conventional chemotherapy if necessary) ever cure cancer? A. Yes, but typically only when cancer is detected at an early stage B. No, it simply prolongs life, usually by a few months C. Yes, even advanced cancer can frequently be cured by chemotherapy D. Only when it is combined with a targeted therapy
Please answer fast Gain-of-function mutations could contribute to cancer progression if the mutant gene is, a) p53 b) Rb c) cdks d) Wee1 2. Loss-of-function mutations could contribute to cancer progression if the mutant gene is, a) Rb b) Ras-GEF c) cdc25 d) cdks Help please! and explain if possible
Please help me to answer this question thank you! (Multiple choice) Select the correct answer 1. The function of mitosis is? a. To distribute the cell's DNA equally between the daughters b. To divide the cytoplasm equally between the daughter cells c. To distribute the parent cell's organelles evenly d. All of the given e. None of the give 2. The chromosomes of daughter cells formed during mitosis a. Are double the number of those in the parent cell b. Are identical to the chromosomes of the other daughter c. Are half the number of those in the parent cell d. b & c e. All 3. Which of these cells is likely to have been formed using mitosis? a. Skin cell b. Sperm cell c. Egg cell d. Heart muscle cell e. a & d 4. A certain stain colors DNA violet. When stained, which of these should appear violet? a. Chromatin b. Chromosomes c. Chromatids d. a & b e. All of the given 5. Two DNA molecules, joined at a centromere, have coiled to form tiny bodies.…
Cervical Cancer Incidence In HPV-Positive Women In 2003, Michelle Khan and her coworkers published their findings on a 10-year study in which they followed cervical cancer incidence and HPV status in 20,514 women. All women who participated in the study were free of cervical cancer when the test began. Pap tests were taken at regular intervals, and the researchers used a DMA probe hybridization test (Section 15.3) to detect specific types of HPV in the women's cervical cells. The results are shown in FIGURE 37.26 as a graph of the incidence rate of cervical cancer by HPV type. HPV- positive women are often infected by more than one type, so the data were sorted into groups based on the women's HPV status ranked by type: either positive for HPV16; or negative for HPV16 and positive for HPV18; or negative for HPV16 and HPV18 and positive for any other cancer-causing HPV; or negative for all cancer-causing HPV. Follow-up time (months) FIGURE 37.26 Cumulative incidence rate of cervical cancer correlated with HPV status in, 20,514 women aged 16 years and cider. The data were grouped as follows: HPV16 positive HFV16 negative and HPV18 positive All other cancer-causing HPV Types combined No cancer-causing HPV type was detected. 2. In which group would women infected with both HPV16 and HPV18 fall?
Cervical Cancer Incidence In HPV-Positive Women In 2003, Michelle Khan and her coworkers published their findings on a 10-year study in which they followed cervical cancer incidence and HPV status in 20,514 women. All women who participated in the study were free of cervical cancer when the test began. Pap tests were taken at regular intervals, and the researchers used a DMA probe hybridization test (Section 15.3) to detect specific types of HPV in the women's cervical cells. The results are shown in FIGURE 37.26 as a graph of the incidence rate of cervical cancer by HPV type. HPV- positive women are often infected by more than one type, so the data were sorted into groups based on the women's HPV status ranked by type: either positive for HPV16; or negative for HPV16 and positive for HPV18; or negative for HPV16 and HPV18 and positive for any other cancer-causing HPV; or negative for all cancer-causing HPV. Follow-up time (months) FIGURE 37.26 Cumulative incidence rate of cervical cancer correlated with HPV status in, 20,514 women aged 16 years and cider. The data were grouped as follows: HPV16 positive HFV16 negative and HPV18 positive All other cancer-causing HPV Types combined No cancer-causing HPV type was detected. 1. At 110 months into the study, what percentage of women who were not infected with any type of cancer-causing HPV had cervical cancer? What percentage of women who were infected with HPV16 also had cervical cancer?
Cervical Cancer Incidence In HPV-Positive Women In 2003, Michelle Khan and her coworkers published their findings on a 10-year study in which they followed cervical cancer incidence and HPV status in 20,514 women. All women who participated in the study were free of cervical cancer when the test began. Pap tests were taken at regular intervals, and the researchers used a DMA probe hybridization test (Section 15.3) to detect specific types of HPV in the women's cervical cells. The results are shown in FIGURE 37.26 as a graph of the incidence rate of cervical cancer by HPV type. HPV- positive women are often infected by more than one type, so the data were sorted into groups based on the women's HPV status ranked by type: either positive for HPV16; or negative for HPV16 and positive for HPV18; or negative for HPV16 and HPV18 and positive for any other cancer-causing HPV; or negative for all cancer-causing HPV. Follow-up time (months) FIGURE 37.26 Cumulative incidence rate of cervical cancer correlated with HPV status in, 20,514 women aged 16 years and cider. The data were grouped as follows: HPV16 positive HFV16 negative and HPV18 positive All other cancer-causing HPV Types combined No cancer-causing HPV type was detected. 3. Is it possible to estimate from this graph the overall risk of cervical cancer that is associated with infection of cancer-causing HPV of any type?
Answer and explain your choice to the following multiple-choice questions about cancer-promoting mutations. The ABC gene undergoes multiple gene duplication events, producing multiple copies of this gene in the genome. This gene duplication event is associated with cancer. What would best describe the normal function of ABC gene? Inhibit cell growth and division Oncogene Inhibit apoptosis Metabolic enzyme EXPLAIN in 1-2 sentences: A mutation in the epidermal growth factor receptor (EGFR) causes it to send a positive signal along its intracellular signaling pathway, even without the EGF ligand bound to it. Knowing the function of a growth factor, how would you classify this mutation? an activating mutation in a tumor suppressor gene an activating mutation in a proto-oncogene a loss of function mutation in a tumor suppressor gene a loss of function mutation in a proto-oncogene EXPLAIN in 1-2 sentences: In an otherwise normal cell, what would be the immediate result of a…
Discuss the different types of DNA damage that can promote cancer. Be sure to discuss the effects of this damage on the cells and how the cancer cells begin to grow. Discuss how different physical agents (such as chemicals or radiation) cause DNA damage that contribute to cancer.
Sometimes, changes or “mutations” occur that prevent genes from doing their job properly. Certain mutations in the BRCA genes make cells more likely to divide and change rapidly, which can lead to cancer. What are BRCA genes? What job do these genes normally do in the human body? Citation required.
Can you please answer the following three questions (one paragraph for each question). Your answers need to be in your own words. Please make sure everything is from your own words 1. In a paragraph, explain what is cancer? 2. In a paragraph, explain why cancer exists, what causes it, and other important things that relates to cancer in general? 3. In a paragraph, explain how different cancers affect different population? Explain also the stages of cancer Remember your work should be in your own words. For anything you use, please cite it in APA.
Please answer all for the UPVOTE. Write TRUE if the statement is correct, but if false, change the underlined word or group of words to make the whole statement true. A. Facilitated diffusion provides the cell with a ready way to prevent the buildup of unwanted molecules within the cell or to take up needed molecules, such as sugars, that may be present outside the cell in high concentrations. B. Plant cells share a characteristic with bacteria that is not shared with animal cells—that is, plants have cell walls, which destroys the plant cells. C. The center of a plant cell usually contains a large, apparently empty space, called the central vacuole.
1.) Download an article related to on how Cancer Develops, how cancer affects cell cycle. 2.) Discuss your article and provide references. (not more than 250 words) 3.)Discuss your views on how to prevent cancer. (not more than 250 words)