The subject who was diagnosed with hyperthyroidism was given propylthiouracil (prevents T4 production and conversion of T4 to T3 in the tissues). After 2 months of treatment, her TSH levels increased by 371.15 % and her T4 levels decreased by 42.7 %. Explain why her TSH levels increased and her T4 levels decreased.
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The subject who was diagnosed with hyperthyroidism was given propylthiouracil (prevents T4 production and conversion of T4 to T3 in the tissues). After 2 months of treatment, her TSH levels increased by 371.15 % and her T4 levels decreased by 42.7 %. Explain why her TSH levels increased and her T4 levels decreased.
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- The subject who was diagnosed with primary hypothyroidism was given levothyroxine (synthetic T4). After 2 months of treatment, her TSH values decreased by 96.38 % and her T4 levels increased by 879.4 %. Explain why her TSH levels decreased and T4 increased.The subject who was diagnosed with secondary hypothyroidism was given levothyroxine (synthetic T4). After 6 weeks of treatment, her TSH values did not change and her T4 levels increased by 778.62 %. Explain the why TSH did not change and why T4 increased.A 24-year-old woman presents 2 months postpartum with symptoms of hyperthyroidism. She does not have evidence of Graves' ophthalmopathy. Her TSH level is undetectable, and free T4 is two times the upper limit of normal. What are the possible causes for her thyrotoxicosis? What tests would be useful to sort out the cause of her thyrotoxicosis?
- In a patient with Hashimoto thyroiditis due to autoimmune destruction of the thyroid gland, serum T4, T3 resin uptake and basal metabolic rate will be decreased while plasma TSH levels will be increased. Group of answer choices True FalseList the disadvantages of insulin obtained from the pancreas of salughtered cows and pigs:Compare and contrast formulation of long acting insulin and short acting insulin. How do their properties help control the rate at which the Delivered insulin enters bloodstream?
- Identify the partial sequence of events that occurs after the insulin binds to its receptor. 1. Phosphorylation of insulin receptor substrate (IRS-1) 2. Activation of the receptor tyrosine kinase activity 3. Activation of phosphodiesterase 4. Activation of phospholipase C 5. Phosphorylation of phosphatidylinositol 6. Activation of phosphatidylinositol-3 -kinase (P1-3 kinase) 7. Binding of Grb 2 8. Activation of a phosphatase 9. Dephosphorylation of pyruvate kinase 10. phosphorylation of pyruvate kinase 2-7-1-4-3-9 1-7-3-5-6-8 2-1-6-5-8-9 2-7-1-6-5-8-9 2-7-4-6-1-8-10Consider the communication between the hypothalamus, pituitary and various target organs when answering this question. For each condition discussed, state the change you would expect to find for each hormone/compound addressed - INCREASED, DECREASED, UNAFFECTED, CAN'T INTERPRET: A patient suffering from untreated Graves disease: A) TSH B) Calcitonin C) Thyroxine(T4/T3) D) TRH A patient suffering from untreated goiter: A) TSH B) Calcitonin C) Thyroxine(T4/T3) D) TRH A patient suffering from pituitary tumor secreting ACTH: A) CRH B) ACTH C) Cortisol D) Plasma potassium level Now, let's consider calcium metabolism and balance. For each condition discussed, state the change you would expect to find for hormone/calcium levels - INCREASED, DECREASED, UNAFFECTED, CAN'T INTERPRET: A patient with…Because of prolonged stress created by the COVID 19 pandemic, many people have experienced adrenal fatigue created by the General Adaptation Syndrome. Please explain, using COVID-19 as an example, what this is and how this process develops into adrenal fatigue or organ collapse. Be sure to explain each stage of GAS and which/how hormones are involved in your answer.
- Diabetes insipidus has been identified in two males. One person did not have the condition until he had a stroke. The other had lived with the condition his whole life, and despite the existence of normal ADH receptors, it had never reacted to exogenous ADH. What might be the cause of the two men's diabetes insipidus? Provide your references.Grave's disease is caused by the overproduction of immunoglobulins that mimic the shape of TSH and thereby bind to the TSH receptors. What is the expected hormonal change to the overproduction of TSH mimicking immunoglobulins? Select one: a. a decrease in the production of TSH, leading to a decrease in the metabolic rate b. an increase in metabolic rate, leading to a decrease in the production of TRH c. a decrease in metabolic rate, leading to an increase in the production of TSH d. an increase in the production of TRH, leading to an increase in the metabolic rateDr. Ryan, Dr. Shapiro, and Dr. Lakey are pioneers in an islet transplant program at the University of Alberta in Edmonton. The Edmonton Protocol is a transplant procedure developed by them where donor islet cells are transplanted into the pancreas of Type 1 diabetic patients in combination with a glucocorticoid free immunosuppressive regimen. One year after transplant, none of the patients had returned to requiring insulin injections. Select the FOUR statements below that describe a patient who does not require insulin injections (insulin independent). Beta cells secrete insulin in response to increased blood glucose levels. Beta cells secrete insulin in response to decreased blood glucose levels. Urine samples indicate high levels of glucose. Urine samples indicate no glucose. Muscle, liver cells, and adipose tissue are permeable to glucose. Muscle, liver, cells and adipose tissue are impermeable to glucose. Water excretion is normal. Extra water is excreted.