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Q: Explain how mismatch repair fixes incorrectly matched base pair
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A: Tuberculosis It is a serious illness of lung if not treated properly. Tuberculosis is caused by…
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Q: What are the parts and function of the digestive system of a milkfish
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Q: D. What are STICHIOCYTES? What are its uses?
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Q: Explain how mismatch repair fixes incorrectly matched base pairs.
A: During DNA synthesis, most DNA polymerases use a process is called proofreading to double-check…
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A: Gel electrophoresis is a laboratory method used to separate mixtures of DNA, RNA, or proteins…
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A: ANSWER:
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Q: 2. Discuss the procedure of Knott's Concentration technique. What are its advantages?
A: Knott's concentration technique procedure and advantages.
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A: Melanin is a kind of pigmentation that usually produces hair, eye and in dark skin.
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Q: explain how beta and gamma radiation can be used to produce diagnostic images.
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- TLCK and TPCK both use Histadine residues in the active site as inhibitors of serine proteases. Why is TLCK specific for trypsin and TPCK is specific for chymotrypsin?Describe the intracellular, biochemical downstream signal cascade that results from the binding of menthol to TRPM8the key features between glycosaminoglycan-binding proteins and lectins?
- CK1-41 is a non-competitive inhibitor of caspase-1. Explain.Neuraminidase (NA) catalyzes a hydrolysis of the glycosidic linkages of sialic acids using a mechanism shown in Fig. (a). and Fig. (b) shows competitive inhibitors of NA Considering the mechanism of NA, why do zanamivir and oseltamivir inhibit NA so potently?Explain why people with a hereditary deficiency of carnitine acyltransferase II have muscle weakness. Why are the symptoms more severe during fasting?
- What would be the effect on the activity of phosphofructokinase of the mutation of Asp103 to the unusual amino acid shown below? Explain in terms of actual structures of the side chains of Asp and this unusual amino acid.Individuals with phenylketonuria cannot convert phenylalanine to tyrosine. Why don’t these individuals exhibit a deficiency of tyrosine?(Note: The alpha subunit of the heterotrimeric G-protein complex becomes activated upon activation of a GPCR. ) What is the mechanism used to turn off the alpha subunit ?