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- When an embryo is homozygous mutant for the gap gene Kr, the fourth and fifth stripes of the pair-rule gene ftz (counting from the anterior end) do not form normally. When the gap gene kni is mutant, the fifth and sixth ftz stripes do not form normally. Explain these results in regard to how segment number is established in the embryoPlease summarize and make the following paragraph about treacher collins syndrome disease into important bullet points. Thank you!! A mutation in the TCOF1 gene causes up to 93% of Treacher Collins syndrome cases. This gene, which is found on chromosome 5, is in charge of facial developnent. TCOF1 changes spontaneously in around half of all cases at conception, but no one knows what causes the change. In addition, Treacher Collins syndrome is inherited as an autosomal dominant condition in some cases. This indicates that the child inherits the Treacher Collins syndrome gene from one parent and an unaffected copy from the other. Changes in the POLRIC and POLRID genes may be responsible for another 2% of instances. It's unclear how the mutations in the genes create facial abnormalities. However, during weeks three to eight of embryonic development, they are thought to trigger apoptosis (cell death) of facial bone, cartilage, and soft tissue.A person has two non-functional copies of the BLM gene. What is the location of this gene in Human genome? What is the inheritance pattern of this disease?
- Sex-specific imprinting distinguishes male and female genomes and is inherited in a parent-of-origin-specific manner. Such imprints are established in the germline during gametogenesis and remain intact throughout embryonic and postnatal development. Access the Imprinted Gene Catalogue website https://www.geneimprint.com/Links to an external site.. Click on “Search by Species Name” and then click on “Human.” Find 4 disorders that involve imprinting: 2 transmitted from the mother and 2 from the father, and use OMIM to describe the phenotypes (you may include images of phenotypes) and explain their underlying cause(s). Paternal Imprint Maternal Imprint 1 2 Include reference"CG islands are thought to have arisen during evolution because they were associated with portions of the genome that remained unmethylated in the germ line" is true or false.Pedro and Karli have a baby girl Ella by in vitro fertilization. Ella does not resemble either one of her parents, so they suspect that the clinic made a mistake during the procedure and implanted the wrong embryo. At Ella’s first well-baby checkup they insist that she get her blood typed. Baby Ella is type A-positive. Pedro is A-negative and Karli is O-positive. Given this information answer the following questions. (a) What are Ella’s possible genotypes? (b) What are Pedro’s possible genotypes? (c) What are Karli’s possible genotypes? (d) Is it possible that Ella is Pedro and Karli’s daughter? (e) Explain your answer in (d). (f) If baby Ella needed a blood transfusion, could either of his parents serve as a blood donor? ______ If so, which one? ______________________________ (g) Explain your answer in (f). ________________________________________ ______________________________________________________________ Kara is B-positive and her father has A-negative blood. Kara marries Ryan,…
- The woman in Problem 24 has had two miscarriages. She has come to you, an established genetic counselor, with these questions: Is there a genetic explanation of her frequent miscarriages? Should she abandon her attempts to have a child of her own? If not, what is the chance that she could have a normal child? Provide an informed response to her concerns.Phenylketonuria (PKU) is a human hereditary disease resulting from the inability of the body to process the chemical phenylalanine, which is contained in the protein we eat. PKU is manifest in early infancy and, if it remains untreated, generally leads to cognitive impairment. PKU is caused by a recessive allele with simple Mendelian inheritance. A couple intends to have children but consults a genetic counselor because the man has a sister with PKU and the woman has a brother with PKU. There are no other known cases in their families. They ask the genetic counselor to determine the probability that their first child will have PKU. What is this probability?how can I Identify the source (mechanism) and types of increased mutations in older mother oocytes and older father sperm.
- Several genes in humans in addition to the ABO gene(I) give rise to recognizable antigens on the surface ofred blood cells. The MN and Rh genes are two examples. The Rh locus can contain either a positive or anegative allele, with positive being dominant to negative. M and N are codominant alleles of the MN gene.The following chart shows several mothers and theirchildren. For each mother-child pair, choose the fatherof the child from among the males in the right column, assuming one child per male.Mother Child Malesa. O M Rh(pos) B MN Rh(neg) O M Rh(neg)b. B MN Rh(neg) O N Rh(neg) A M Rh(pos)c. O M Rh(pos) A M Rh(neg) O MN Rh(pos)d. AB N Rh(neg) B MN Rh(neg) B MN Rh(pos)Pedro and Karli have a baby girl Ella by in vitro fertilization. Ella does not resemble either one of her parents, so they suspect that the clinic made a mistake during the procedure and implanted the wrong embryo. At Ella’s first well-baby checkup they insist that she get her blood typed. Baby Ella is type A-positive. Pedro is A-negative and Karli is O-positive. Given this information answer the following questions. (d) Is it possible that Ella is Pedro and Karli’s daughter? (e) Explain your answer in (d). (f) If baby Ella needed a blood transfusion, could either of his parents serve as a blood donor? ______ If so, which one? ______________________________ (g) Explain your answer in (f). ________________________________________ _______________________________________In a paternity suit, a woman with type O blood claims that a man with type AB blood is the father of her type O baby. List the genetypes of the mom, man, and baby. Can the woman prove he is the father or can he is not? Why or why not? If the accused man was type A insyead of type AB, what would be his possible genotypes? Could the woman prove he is the father? Can the man prove he is not the father? Why or why not?