Which step(s) of cellular respiration would be directly inhibited by high concentrations of NADH? How would this affect other steps of cellular respiration?
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- In cellular respiration, can NADH and oxygen levels increase/decrease at the same time or not? What is the reason on why they increase/decrease at the same time or at diffrent times?What is the term for the process whereby the synthesis of ATP is coupled to the conversion of NADH or FADH2 to NAD+ or FAD, respectively?Can you please explain the relationship of ATP concentration and NADH concentration in this graph (cellular respiration)? At different treatments of ATP (E.g. At 25 ATP, 50 ATP, 100 ATP), why did the NADH concentration change?
- When rotenone is added to actively respiring mitochondria,the ratio of NADH to NAD+ increases, but the FADH-/FADratio remains unchanged. What step in the system is beinginhibited?At which site in the mitochondrion and chloroplast do the ATPmolecules collect after being made by ATP synthases, and what is theadvantage of this?How come high levels of NADH indicate reduced coenzymes rather than oxidized coenzymes? Isn't pyruvate oxidized by the reduction of NAD+ to NADH? Why is the answer reduced then?
- What effect would cyanide have on NADH and NAD+ concentrations in a cell? In your response, you must utilize the phrase "oxidized." Based on that answer, what effect would cyanide have on carbon dioxide production during cell respiration?Where in cellular respiration does feedback inhibition occur? What would happen if this feedback inhibition were removed?What would be the effect on ATP and NADH production (how many ATP and NADH could be produced from one molecule of glucose) if pyruvate transport into the mitochondrion were inhibited?
- The citric acid cycle generates NADH and FADH2, which are then used in the process of oxidative phosphorylation to make ATP. If the citric acid cycle (which does not use oxygen) and oxidative phosphorylation are separate processes, as they are, then why is it that the citric acid cycle stops almost immediately when O2 is removed?In the krebs cycle, if the enzyme used succinate dehydrogenase used NAD+ as the oxidizing agent, what impact would it have on the overall production of ATP in the call?Why does mitochondrial NADH yields more ATPs than cytosolic NADH?