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Name: Why is the mutation rate so muchhigher in mitochondria than in thenucl
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Description: Why is the mutation rate so muchhigher in mitochondria than in thenucleus (and chloroplasts)? Could thishigh rate have been useful to the cell?

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Biology

Why is the mutation rate so muchhigher in mitochondria than in thenucleus (and chloroplasts)? Could thishigh rate have been useful to the cell?

Biology Today and Tomorrow without Physiology (MindTap Course List)

5th Edition

ISBN:9781305117396

Author:Cecie Starr, Christine Evers, Lisa Starr

Publisher:Cecie Starr, Christine Evers, Lisa Starr

Chapter6: Dna Structure And Function

Section: Chapter Questions

Problem 1CT

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Similar questions

Some bacteria might be able to respond to environmental stress by increasing the rate at which mutations occur during cell division. How might this be accomplished? Do you think there would be an evolutionary advantage of this ability? Explain.
Why do mitochondria need genes that encode rRNAs andtRNAs?
How can the same modification of the standard genetic code happen in two organelle genomes (chloroplasts and mitochondria) and in the same organism?
The more cells divide, the more likely it is that mutationswill accumulate in their genomes. Explain how ?
In the replica plate experiment, it shows that mutations are random. However, certain environmental stresses (such as high temperature, high salt, and low pH) can increase the mutation rate. QUESTIONS: Does this indicate that mutations are nonrandom, since they increase in response to cell stressors?
X-rays strike a chromosome in a living cell and ultimately cause the cell to die. Did the X-rays produce a mutation? Explain why or why not.
Why is it reasonable that eukaryotes have aDNA polymerase (Pol ) that operates only in mitochondria?
why would a mutation complex 1 usually not result in immediate death ? And while blocking complex IV can result in immediate death ?
Antibiotics such as chloramphenicol, tetracycline, and erythromycin inhibit protein synthesis in bacteria, but have no effect on the synthesis of proteins encoded by eukaryotic nuclear genes. Cycloheximide inhibits the synthesis of proteins encoded by nuclear genes, but has no effect on bacterial protein synthesis. How might these compounds be used to determine which proteins are encoded by mitochondrial and chloroplast genomes?
Why would a mutation in a somatic cell of a multicellular organism not necessarily result in a detectable phenotype?
why does a clone of cells produceonly one type of G-6-PD enzyme? What would you expect to happen if a clone was derived from an early embryonic cell? Whydoes the initial sample of tissue produce both forms of G-6-PD?
Is each of these statements true of chloroplast or mitochondrial genomes, both, or neither? a.contain tRNA genes