Biopsychosocial Model Of Childhood Obesity

Genetic research of obesity was partly successful in establishing obesity in model organisms – rodents where obesity occurs spontaneously together with other pathological aspects (insulin resistance, …). The main cause of monogenic obesity in these model organisms are common mutations always present in only one gene. Results of research on model organisms allowed us to understand biological mechanisms of calorie intake and regulation and maintenance of body weight. The most important insight into obesity was achieved in 1994 after discovery of ob gene encoding for leptin. In two years period, using screening method, candidate homologous genes, selected on genetic study basis on mice, another five genes were identified. Mutations on these genes were found to be the cause of autosomal recessive or dominant monogenic obesity. Products of these genes are leptin and its receptor, proopiomelanocortin (POMC) melanocortin receptor 4 (MC4R) and

There have been studies conducted to find out what has caused or what the leading factors to obesity are. Researchers are currently still doing research to find out what causes or what may be the lead to obesity. Childhood obesity is a serious medical condition which considers a child to be obese if their Body Mass Index (BMI) is at or above the 95th percentile for children and teens of the same age and sex. (Rendall., Weden, Lau, Brownell, Nazarov & Fernandes, 2014). Obesity is on a rise in the Unites States and all over the world and can lead or result to other health complications later in life. The crucial breakdown serves as an implication of outlining childhood obesity, collaborating problems of the disease and resolutions, as well as applying critical thinking to give a complete approach to deliver information on childhood obesity. This will be done through citation of scholarly articles, samples and other modes of supporting details.

Genes control almost every aspect of human life, and when it comes to weight there is no exception. Little is known, however, how much of an individual’s genes actually control the weight of certain individuals. Body fat can vary from person to person, yet some people have always carried more weight than others. Often times, when one person is overweight in the family, most of the other individuals in the same family are overweight as well. However, more than just genetics can go into being overweight, and a person’s genes are not the end all be all of obesity. Many times the environment surrounding people who are obese contributes to the overall weight of the individual, and when obesity promoting genes are mixed

There are many people who believe that obesity is a matter of personal responsibility. Many people think it is Americans are the ones who to blame and not the fast food industry. Many people may say that it is easy to blame obesity on what Americans eat. However, these people do not realize that many Americans cannot afford healthy foods. So, since fast foods are cheap and affordable, it may be why many people choose them. In the article “Physical activity and childhood obesity” Green, Riley, and Hargrove explain “a greater percentage of adolescents from families living in poverty are obese (23%), compared with those from families of a higher socioeconomic status (14%)” (915). Additionally, this statement demonstrates that although obesity may be caused by genes or psychological influences, the environment is the most important factor. When families are in poverty, they are not looking for what is healthy and what is unhealthy. They are looking for food to survive, and so many times fast food is the only option. Following this future, there is indication that obesity, extremely affects certain minority youth populations. The National Health and Nutrition Examination survey found that African American and Hispanic adolescents ages 12-19 were more likely to be overweight at 21 and 23 percent, respectively, as compared to non-Hispanic White adolescents (14%) (915). Furthermore, these statistics show that there is a strong relationship among race or ethnicity, poverty, and

concludes that there are a multitude of factors that have contributed to the recent childhood obesity growth rates, including changes in the environment where children are raised. "In the built environment and urban lifestyle have resulted in reduced physical activities and, consequently, have played a role in the growth of childhood obesity." Consumers generally lack credible nutritional information.

These factors or social determinants of health can be behavioral, social, psychological, technological, environmental, economical, and cultural, operating at all levels from the individual to the family to society as a whole. The circumstances are different from person to person even when the society is the same. Therefore, it is hard to constrain and minimize the factors to a small number. However, this paper provided two evidence based approaches (socioeconomic and cultural) as causal associations for childhood obesity among African Americans in Louisville, Kentucky. (Figure

indicate that inherited genetic variation is an important risk factor for obesity. It was also pointed

Childhood obesity is a major problem in the United States. There are multiple reasons for this problem including the child’s school system, the access to technology in young children and the child’s genes. It is sometimes inevitable for a child to be obese due to their genes. There are some precautions being made to help this problem. Some might work but others might fail. Childhood obesity is a major problem and needs to be fixed because it causes major health issues for the child in the future.

Introduction: In 1998 the World Health Organization announced that obesity had become an epidemic in both adults and children. This epidemic is a major concern in public health yet many countries are failing to address the issue. The purpose of this paper is to discuss the causes contributing to this increasing epidemic of childhood obesity. There are many causes that factor into childhood obesity but I will focus specifically on genetics, overeating, and the lack of exercise.

Childhood obesity is an important issue in the American culture because of the staggering results that this disease has reached in the past years. Certain gene factors connected with the change of culture and lifestyle has produced kids and adults who are now not as healthy as most people were just a few decades ago. The widespread of obesity has been the biggest result of these specific changes.

This paper will explore and discuss the rising issue of childhood obesity by using published books and articles. I will explore five different factors that correlate to possible beliefs of childhood obesity. The factors that I will discuss include: parenting, education, technology, marketing, and the Fat-Gene theory. There has not been one specific cause to childhood obesity but there are underlying effects that can be harmful to children from 2-19 years of age. The

The cause of childhood obesity is widely debated. There are some who believe childhood obesity is caused by socioeconomic factors while others believe it is caused by media marketing junk food to children. As a response to this ongoing debate, food markers, mainly fast food, try to defend themselves by blaming childhood obesity on physical inactivity and the lack of parental influence on a healthy diet. Despite what many researchers may believe, childhood obesity is caused by a combination of key factors. According to a well-written article by Erica Roth, Childhood Obesity, she along with other doctors believes that obesity could be caused by many different factors. Some may include “family history, many psychological factors, and lifestyle” (Childhood Obesity). However, the causes of childhood obesity are not limited to these life threatening factors, childhood obesity is almost always

The topic that I have chosen to look at is obesity. The aim of this topic is to find out whether “the ignorance of genetics as a contra factor of obesity”. I have chosen to look at this topic because obesity is concerns me. Obesity has always been a bid debate for a long time now and I was intrigued to find out more about the topic and the cause of obesity. The fact that obesity has made deadlines news in recent years. There are a lot of misconceptions about obesity such as overweight is caused by a sow metabolism. There have been a lot of beliefs and myths about obesity in society today and he above myths is on of them. According to Donnellan (1998) ‘ ‘people who are overweight have told their doctors that they eat no more and

There are more than one billion overweigh adults in the world with at least three hundred of these adults being obese. In North America, obesity has increasingly become one of the most important public health problems (Flegal, Graubard, Williamson, & Gail 2005). With cases of obesity increasing, there has also been more exportation of research being done into the topic. Moreover, social, psychological and biological factors are being investigated to determine aspects that may influence the onset of obesity. This paper will focus on two specific domains of the biopsychosocial model of health with a focus on psychological and social factors that affect obesity.

Everybody knows the child that can eat any type of food all day and never seem to gain weight. This occurrence has led researchers to investigate the role that genetics plays in childhood obesity. Not all children who are inactive or who eat poorly are obese, much in the same way that some obese children eat fairly healthy, and exercise moderately. Heredity has recently been shown to influence body fat percentage, regional fat storage, and the body?s response to overeating (Rush, 1). Children who have obese parents are 80% more likely to be obese than their lean parented counterparts (Buffington, 16). This familial correlation is contributed to genetics as well as the parents eating habits. Children with obese parents typically aren?t taught the correct way to choose when and what food to eat, leading to poor eating habits and eventually obesity. Many genetic defects can have a significant effect on obesity such as variable thyroid activity and pituitary defects. Abnormalities in any one of these regulators could be responsible for appetite abnormalities and weight gain. Furthermore, obesity leads to defects in appetite regulation, hormone production, and metabolic events (Oklahoma Cooperative, 4) that are responsible for further weight gain,