Ketogenic Diets Rationale

when dieters reach their weight loss goal. Dieters are allowed to eat more carbohydrates, but

H, who had come into the emergency department two days prior for diabetic ketoacidosis (DKA) and was diagnosed with type 1 diabetes. The Mayo Clinic explains that diabetic ketoacidosis occurs when the body cannot produce insulin. Insulin is the key that allows glucose to enter cells, and provide the cells with energy. Without glucose for energy, the body begins to breakdown fats that produce harmful substances known as ketones. DKA is characterized by hyperglycemia, metabolic acidosis and ketones in the urine, (2017). That had since resolved, so for the most part of the day me and the secondary nurse were providing diabetic education. She was educated on the use of the glucometer, how to give insulin injections, and symptoms of hypoglycemia, I felt confident in this education and was surprised by how much I remembered from previous lectures. I did not have time to provide teaching on diet, exercise induced hypoglycemia, or patient specific teaching based upon her report, because we switched our roles. I can see how education can get swept under the rug in practice when you are so busy throughout the day. Instead of saving education until the end of the day, when I become a new nurse I will make sure I am taking advantage of every patient encounter as an opportunity to educate. Later in the simulation, when I had transitioned roles, Ms. H experienced a hypoglycemic episode after receiving a dose of insulin and not consuming any of her breakfast. According to Silvestri, hypoglycemia is often caused by the following, too much insulin, too little food, or excessive activity (2014, p. 653). In Dr. Johanson’s lectures I learned that carbohydrates are the main source of energy for the body and are metabolized to form glucose for the body to use. When there is no consumption of carbohydrates, there is no glucose and this results in low blood glucose known as hypoglycemia. A type 1 diabetic lacks the ability to make insulin, which is the key that

According to Mark Sisson (2009), longer-term ketosis can result from fasting or staying on a low-carbohydrate diet, and ketosis that’s deliberately induced serves as a medical intervention for epilepsy. In glycolysis, higher levels of insulin block release of fat from adipose tissues and promote storage of body fat, while the body is in ketosis, fat reserves are continually released and consumed as energy (Sisson, 2009, p.47). This is why ketosis is referred to as

Insulin is a hormone that is produced from what is known as the “islets of Langerhans”, discovered by German Histologist Paul Langerhans, and is required for the utilization of glucose in muscle cells for energy. If the muscles are deprived of glucose for energy conversion, the muscles begin to utilize fat for energy (Roth). This however has toxic side effects, such as the production of high blood levels of Ketone bodies or otherwise known as Acetone. In high quantities, Acetone will accumulate in the blood, leading to brain damage and the possibility of brain death (Roth).

This study was performed to study the effects on what would happen to the mass of a bone from a chicken wing when the amount of sugar is increased in a liquid. The hypothesis for this lab was that when more sugar was added to the liquid, the more the mass of the bone would decrease over time. It was predicted that the gatorade would make the mass decrease the most and that the water would decrease it the least. It is known that sugar can increase body weight in fat (Greenfield), but sugar can also act as an acid in some cases (Goldschmidt). Our hypothesis was formed thinking that the sugar may act as an acid in this experiment. In this experiment the independent variable was the liquids and the dependent variable was the three chicken bones.

I, a normal seven year old boy, was about to have my life changed forever. At first I didn’t realize how my life was being dramatically altered. I thought that the doctors would give me medicine, like they usually do, and I’ll continue my life as always. However, Diabetes is much more complicated. I was shocked to discover that for the rest of my life I had to receive shots and count these, previously unknown, sugars called Carbohydrates, which determines how big the shot will be.

Energy is essential for survival, and the human body is remarkably efficient at utilizing different energy sources. The body can obtain energy from glucose when consuming a normal carbohydrate diet, or from ketone bodies when consuming a ketogenic diet. This is an alternative source of energy that the body will use when 5% (or less) of total kcal come from carbohydrates. Metabolic processes differ depending on the energy source. One application of understanding the metabolic processes is the imprevment of glycemic control and stop of medications use under a low carbohydrate ketogenic diet. with type II diabetes can

Purpose Statement: To inform my audience of the effect sugar has on the human body.

The spike in blood glucose levels after ingestion of simple sugars is thought to be related to some of the heart and vascular diseases which have become more frequent in recent times. Simple sugars form a greater part of modern diets than formerly, perhaps leading to more cardiovascular disease. The degree of causation is still not clear, however.

According to the experiment results the hypothesis was proven to be correct. The diabetics did indeed have a higher absorbance level than the non-diabetics. It was made clear that the rate at which a diabetic digests their meal is slower than a non-diabetic. It proves that fact that without sugar, the body could not function properly because there would not be enough glucose in the body to provide energy in order to make ATP. Without the main ingredient to make ATP the body would die. This fact leads to the understanding as to why people with diabetes in third world countries are dying because they cannot get the insulin needed in order to help their bodies make ATP and function (Khunti).

When insulin is at high levels in the body, the food you eat can readily get converted into body fat, in the form of triglycerides. Thus, if lower amounts of carbohydrates are consumed, the body naturally produces less insulin and looks to other sources for fuel, namely FAT! For this reason, the Atkins diet restricts processed and refined carbohydrates and limits intake to 15-60 grams per day, encouraging protein and fat consumption (http://www.ivillage.com/diet/features/dietsaz). When the body lacks its primary energy source (carbohydrates) it will naturally turn to its next source (fat) to burn for energy. When the body breaks down fat, ketones are formed and the appetite is naturally suppressed. One reaches ketosis when his or her carbohydrate intake is less than 40 grams per day. The average person not on this diet consumes 300+ grams of carbohydrates per day.

The Ketogenic Diet is a way of eating that is becoming increasingly popular within fitness and health communities. The Ketogenic Diet began as a way of eating to combat Alzheimer’s disease along with neurological disorders such as Multiple Sclerosis and Parkinson’s Disease (Davis, n.d.). In the past decade it has become widely known for its weight loss benefits. As of recent, many famous public figures and professional athletes have further popularized the concept of the Ketogenic Diet. Many years ago, the Ketogenic Diet began as a way to combat diseases and neurological disorders, but has since then become a much more popular tool for weight loss.

Reactive hypoglycemia, a rare form of hypoglycemia, increases insulin levels after the consumption of excess carbohydrates, leading to a drop in blood glucose levels. This differs from conventional hypoglycemia where blood glucose drops several hours after a meal, but can easily be returned to normal by the consumption of food. Reactive hypoglycemia can cause fatigue, dizziness, shakiness, and in extreme cases, a coma. Although no effective treatments exist, glucagon, a peptide hormone derived from pancreatic alpha cells, seems to reduce symptoms. In the proposed experiment, the effectiveness of glucagon relative to a regimen of dietary control, exercise, and Acarbose will be tested on Zucker-diabetic-fatty (ZDF) rats (Rattus rattus). Three

Hypoglycemia is an expression used to describe a below average glucose level. Due to young age, piglets have trouble walking and feeding off their mother (or sow) to get essential nutrients. One of these essential nutrients is glucose (or sugar). This nutrient is essential because it give the animal energy to become mobile. When born piglets already have low levels of glucose; however, the animal body is supposed to compensate by using the low glycogen reserves located in the piglets liver. The glycogen reserves are supposed to give the piglets a sufficient amount of glucose until the piglets are able to move towards and to feed off the sow. The piglet is dependent on the sow for energy provided by the lactose in the sows milk; however, if the piglet is deficient in not getting enough lactose to maintain the piglets energy, the level of the piglet’s energy will suffer from Hypoglycemia because… The piglet body temperature will drop below a stainable level, go into a coma and die.

During HS, the utilization of carbohydrates (i.e. glucose) as an energy source is altered (Streffer, 1988), and this has been demonstrated by our lab in pigs (Pearce et al., 2013) and cattle (Wheelock et al., 2010). Acute HS was originally thought to be responsible for reduced worker productivity in summer months (Baumgard and Rhoads, 2013), and was first reported to cause hypoglycemia in cats (Lee and Scott, 1916). In addition, athletes exercising during times of HS have increased hepatic glucose output and glucose oxidation at the expense of lipids (Fink et al., 1975; Febbraio, 2001). Moreover, exogenous glucose is unable to blunt the formation of hepatic glucose (Angus et al., 2001) likely due to increased glycogenolysis (Febbraio, 2001), and gluconeogenesis (Collins et al., 1980). A proposed mechanism for the enhanced hepatic glucose output is increased pyruvate carboxylase expression (a rate limiting enzyme that controls lactate and alanine entry into the gluconeogenic pathway) during times of HS (O’Brien et al., 2008; White et al., 2009) likely resulting from increased plasma lactate (presumably due to an increase in muscle lactate production). This indicates that peripheral tissues increase aerobic glycolysis, and thus resembles the Warburg effect (used in cancer cells) where aerobic glycolysis in skeletal muscle is accelerated and lipolysis is reduced (Baumgard and Rhoads, 2013).