Amplified music from night clubs can lead to hearing damage
Shifts in hearing thresholds can result in excessive exposure to any kind of sound. It not matters whether the sound is pleasant or not nor the music genre, the degree of noise induced hearing loss (NIHL) depends on the intensity and frequency characteristics of the noise, along with its duration and the intermittency of the exposure. Individual genetic and environmental factors also play important part(Bovo, Ciorba, & Martini, 2007).
Hetu and Fortin (1995) studied the characteristics of highly amplified music in discotheques from psychoacoustic and phenomenological points of view. They found that the amplified music is characterized by strong pulsations (≈ 2 Hz), a narrow dynamic
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“Recreational” noise exposure (nightclubs and music players) is unlikely to cause the above direct damage. Rather than direct mechanical injury, the process is a combination of metabolic and mechanical changes. The music is more damaging when it is impulsive (energy is highly concentrated in the time domain) and contains spectral energy peaks (over-stimulation of local specific frequency region of the cochlea), the stereocilla of the hair cells are the most vulnerable organ(Dobrucki et al., 2013).
Several mechanisms have been proposed for TTS. This includes increased shearing force of the hair cells, synaptic fatigue, metabolic exhaustion of the outer hair cells (OHCs) and inner hair cells (IHCs) due to overload, and changes in blood supply to the cochlea(Yamasoba, Pourbakht, Sakamoto, & Suzuki, 2005). The internal amplification system of the cochlea is thus impaired as shown in LePage and Murray (1997)’s research by studying otoacoustic emissions. Yamasoba et al. (2005) suggested that constant overstimulation of the metabolic process could result in production of by-products like reactive oxygen species and free-oxygen radicals, which triggers cell necrosis or apoptosis. They also found pathological changes of the hair cells along with TTS, such as hair cells disorganisation and fusion, floppy stereocilia, collapsing of the pillar bodies and swelling of the afferent nerve endings. For IHCs, in addition to
The neural processes required to hear a phone ring may surprise some people. The neuron’s cell body (the part of the neuron which contains the nucleus) contains branch-like structures known as dendrites, which receive information from the axon—a long tail-like portion of the neuron. This information then travels to other neurons via chemicals called neurotransmitters (Pastorino & Doyle-Portile, 2015). The axon is coated by a wax-like substance called myelin, composed of segments known as myelin sheaths. Corrosion of the myelin sheaths may adversely affect a sensory input, in this case
Hearing loss continues to linger in the elderly population of today’s society. Yet, the onset of hearing loss can occur at any age and at any point in
Earbuds are a major concern in hearing loss. Although, the real problem is not in the product, it’s in the volume. Getting a pair of earbuds for Christmas, your birthday or any other occasion is awesome, but if you don’t keep it at an appropriate volume it is dangerous. According to the World Health Organization, nearly 1.1 billion young adults and teens are globally at risk of developing major hearing loss. Also, wearing earbuds and listening to loud music is the same as the sound of motorcycles, chainsaws, and rock concerts. Earbuds are only bad for your hearing if you don’t turn the volume down.
This happens in two ways: listening at too high of a volume, or listening for too long. Studies have confirmed the obvious fact that “the louder the noise and the longer you’re hearing it, the greater the risk of hearing loss” (Siegel). This study concluded that an increasing amount of teens are suffering hearing loss and “wearing earbuds to listen to music may be to blame” (Siegel). The knowledge of loud sounds hurting the ear is well known, but not as many people are aware of the problems caused by listening to music for an extended amount of time. “Constant low-level noise might cause hearing problems, a new study in rats finds” (Cheung). Even music at a low level can damage a listener’s hearing if heard for an extended period of time, but what can losing some hearing really do to a
Long-term noise exposure is an example; this is due to the damage that the noise exposure can have on the sensory hair cells. Sensory hair cells are what allow you to hear and if damaged the ability to hear is reduced and these hair cells do not grow back. As Colin was a car mechanic this meant he will have been in contact and close proximity to loud equipment and machinery daily, this could have had a detrimental effect to his hearing and prevented him from hearing Mary. Other environmental factors include ototoxic drugs, genetic factors and cell damage and neural degeneration which are common effects of aging. Ototoxic drugs can effect hearing as they can damage the inner ear including the hair cells and also the auditory nerve, this is important as it carries the sound information to the
Music can affect the body negatively. One of these effects is the fact that music can be addictive (Severance). According a scholar, if people go to long without their preferred music they can have headaches. The same scholar later stated that this is caused by disharmonic tones. Disharmonic tones are found most often in hard rock music and popular pop music (Severance). However, it seems as though the positives of music out weighs the negatives.
Hearing loss isn’t rare because it is estimated 50 million Americans suffer from hearing loss. The authors state this in the title of her book about the many millions of individuals affected by hearing loss. Hearing loss isn’t confined by age. The author developed her hearing loss at the early age of 30 but the significant onset of adult hearing loss occurs between the ages of 19 and 44. People usually think that hearing loss only affects the older generation, but we learn that it can affect individuals
Noise is one of the most widespread causes of hearing loss, and the most likely causes of occupational illnesses in the United States (Leigh 728). Acute or prolonged exposure to high-level, impulsive noise is an ongoing problem for the military and defense forces all over the world (Sliwinska-Kowalska and Adrian Davis 274). One shot from a big caliber firearm, perceived at a close range, may permanently or temporarily damage an individual hearing in an instant. Additionally, repeated exposures to noisy machinery may, over an extensive period of time, impose serious risks to human hearing. According to the report released by National Institute on Deafness and Other Communication Disorders (NIDCD); ten million Americans have already suffered permanent hearing damage from noise and thirty to fifty million individuals are exposed to hazardous noise levels every day (Leigh 728). Many hearing illness in the police service originate from the noise caused by gunshots. As a result of increased cased of hearing problem, mainly in the police service, many scholars have engaged in research in order to get appropriate mean to control these problem. Considering the increasing investigation from military groups for vital information on how to protect from impulse noise, it is vital to explore health effects associated with impulse noise from gunshots. This study explores the effects on hearing caused by
To do this, the researchers exposed a second group of adult quail to the same hearing-damaging sound as the first and then injected them with radiolabeled thymidine. This allowed researchers to use autoradiography that produce low levels of background issues as well as being able to see 10 to 20 grains over red blood cells. The control birds were still given the injections, however, they were not exposed to the sound. The experimental group experienced the same pattern of damage as the birds in the previous experiment and thymidine was seen over hair cells and support cells in the damaged area. There was no thymidine observed outside of the damaged area. The transition zone of the basilar membrane, where tall and short hair cells are integrated, it seemed that the short cells were more often labeled than the tall cells. Tall cells do not lack the afferent auditory-nerve fiber innervation that is seen in short cells making them a more useful candidate for labeling. The researchers failed to identify location and mechanism of the activation of the precursor cells but remained hopeful that there may exist a way to restore inner ear sensory losses that are caused from injury of hair
It is generally accepted that tinnitus is related to the changes in neural activity and maladaptive neuroplasticity of the auditory networks due to the damage in the auditory system. However, recent evidence obtained from neuroimaging studies show that both auditory and non-auditory structures are involved in the development of tinnitus (Leaver et al., 2011;
What should we know about hearing loss? The main causes for hearing loss is head trauma and exposure to loud sound. The only way we can take care of ourselves is by knowing how to.To prevent hearing loss we can turn down the volume, use earplugs or remove ourselves from situations where noise is excessive. More Importantly, Digan states,”The major source of dangerous noise is a personal media player combined with a pair of earbuds”, ( Pg.11). For example, Digan writes,”Sound is measured in
et al. used two different thyroid hormone receptor knock-in mutations, TRβΔ337T/Δ337T and TRβE457A/E457A, to test how triiodothyronine regulates cochlear development. They introduced the mutations in the same way for both trial groups and followed the same procedure for mice maintenance, surgery and cochlea tuning as well as using the same sound system of testing. Data was collected for auditory brainstem responses by subtracting the value measured from an electrode placed on the mastoid process from the ipsilateral electrode in the vertex, relative to the ground electrode placed in the neck and measuring, through direct instructions, the morphology of the cochlea. The researchers also measured compound action potential threshold in the cochlear by using a modified tracking system and threshold was defined as the level needed for a 20-µV N1/P1 amplitude at each frequency. Results show that TRβΔ337T/Δ337T mutants had significant increases in auditory brainstem responses, more than 60dB, and that TRβ E457A/E457A mutants had moderate elevations of approximately 20dB. Also, TRβE457A/E457A mutants show an increase in sound pressure required to raise compound action potential amplitude to the defined level and that gross morphology was normal in both groups. The results suggest that disrupted triiodothyronine leads to improper tectorial membrane development which allows for increased thresholds and decreased action potentials of inner hair cell potassium channels.
Different frequencies reach their peak at different positions along the tube, which allows the cochlea to distinguish them. Researchers have found that the spiral shape significantly enhances the “vibrational motions” that translate into nerve signals. However, if the cochlear is damaged it is obvious how important the development of the cochlear implant is. Essentially the cochlear implant serves as a bypass for damaged sections of the ear, replacing the function of the ineffective hair cells.
For example, having diabetes & hypertension can both reduce hearing. Hearing loss can also be hereditary and can effect someone as soon as birth. Hereditary types of hearing loss account for more than 50% of all congenital sensorineural hearing loss cases and are caused by genetic mutations (Egilmez). Another way that hear loss can be induced medical is with a condition called “Tinnitus” which is when someone hears a sound ,such as, ringing and there is no outside source that is causing the sound. Tinnitus is very common and can effect 50 million Americans from time to time according to the American Tinnitus Association, of those 12 million people complains that it bothers them and seeks medical attention. 2 million people suffer from sleep deprivation, depression, poor concentration, and thoughts of suicide due to Tinnitus (Dalebout). This shows that losing you hear can cause effect someone physically, mentally, and
only by the patient. Because the inner ear has no pain fibers, damage is not