Neurocognitive Dysfunction Case Study

In addition PCA is identified by a significant concentration of plagues and neurofibrillary tangles (NFT) in the occipital lobe and the parietal lobe as well as the connection known as the occipitotemporal junction ( Tsunoda, et al., 2011). The author’s go on to explain that they hypothesise that PCA is connected to AD, dementia with Lewy bodies (DLB), corticobasal degeneration (CBD)

Beginning with the three main parts of the brain, the brain stem, the cerebellum, and the cerebrum, the website gives an overview of what I had studied in Basic Anatomy and Physiology class. The cerebrum, which controls memory, thinking, emotions, and voluntary movement, is the primary part of the brain affected by AD. In order to carry out its functions, the brain requires a rich blood supply of about 20 to 25 percent of the bodies’ blood total with each heartbeat. The whole vessel network picture demonstrates how vast the blood supply to the brain is and makes it understandable that up to 50 percent

As the progressive disease take over the body, the ability to function, understand, and comprehend becomes an issue. On a neurological level, the disease affects the brain causing senile plaques and neurofibrillary tangles to build and connect between the cells. The plaques contain proteins that are called beta-amyloid that clump together between the nerve cells. On the other hand, tangles are twisted strands that keep

The disease functions by causing neurons to degenerate and lose their synapses with other nerves, thus cutting down severely on the intercellular communication which lies at the heart of all behavior. (1).

Studies show that a network in the brain called the cortico-striato-thalamo-cortical circuit contributes to the pathogenesis of the disease.

The experiencing of the world and the way the patient reacts to it is radically influenced by this unhealthy interaction within the brain. The initial symptoms involve mild forgetfulness of recent events, activities or familiar people/things. In the process of development of the disease, skills acquired throughout the life of the patients such as brushing the teeth, easting with a fork and a knife are forgotten; thoughts are very unclear, and behavior becomes unable to be subordinated to normal human life norms. All the clues the brain has learned so as to be able to fill in the blank in everyday life have been forgotten. (3)

Brain - Scans show that there are differences in the brains of some people with

HCV with neurocognitive effects similar to those of HIV, but without the associated end-stage dementia (Laskus et al., 2005). Prevalence studies have estimated the 1-month rate of depression in HCV-infected individuals at 28% (Golden et al., 2005), but it is clear that treatment of HCV with interferon-based therapies dramatically increases the risk of depressive symptoms to near 80% (Laguno et al., 2004; Reichenberg et al., 2005; Scalori et al., 2005). These effects on mood, increased fatigue, and worsened quality of life are even greater in patients with concurrent, advanced HIV disease (Ryan et al., 2004), and appear likely to be due to a variety of factors, including biological and sociodemographic ones (Braitstein et al., 2005). Multiple

It’s unclear if people’s memories are affected in the same way as those of mice. But the new research adds to evidence suggesting that some body-wracking infections could also harm the human brain, says epidemiologist and neurologist Mitchell Elkind of Columbia University, who was not involved in the study.

While the cause of primary PD is has not been determined the neuropathology of PD is well understood. The midbrain contains the basal ganglia and the thalamus. Both of these have excitatory and inhibitory neural pathways that form two pathways: (1) and excitatory loop that is a direct path and (2) and inhibitory path that is indirect (Dauer & Przedborski, 2003). In PD there is initially a loss of dopamine producing neurons in the excitatory loop and the appearance of Lewy bodies throughout the brain. The loss of these dopaminergic neurons indicates that in PD the defining feature is a substantial decrease in the neurotransmitter dopamine (which occurs in the nirgrostriatal brain tract; (Dauer & Przedborski, 2003).

The virus causes changes in expression to nearly 1300 genes in the hippocampus, the area responsible for storing memories and helping bodies be properly oriented in the environment. The virus altered how the hippocampus responds to the neurotransmitter dopamine, which is widely used throughout the brain and integral to the immune system.

Then the disease will hit the frontal lobe and the prefrontal cortex which make us

The quantitative methods explained so far quantify the CBF (in one way or another) but in relative units, which implies that the regional CBF values are relative to the number of counts corresponding to a reference value. The reference value may be the mean value of a specific brain region (e.g., the cerebellum) or the whole brain. The reference value should not be affected by the pathology under study, so that the regional relative CBF values are due to the effect of the disease in the patient and not to the reference

A speech-language pathologist’s participated in a dominant aspect in the screening, assessment, diagnosis and treatments concerning infants, adolescents, children and adults alongside cognitive intercommunication disarrangements. For instance, an example of a diagnosis and treatment concerns might include dysphagia. This disorder can be diagnosed from a wide range from an infant to the elderly. As a speech-language pathologist teaching swallowing exercises can be beneficial to the patients (Glover, McCormack & Smith, 2015). This environment description illustrates the roles of speech-language pathologists in the interpretation and the management of peoples with language condition affiliated with cognitive disorders and identifies the amplitude and principle for the above-mentioned services. To state

Neuroscience's study of Cognitive Function is a relatively late phenomenon. As late as 1848, the accident and resulting injuries of Phineas Gage amazed the medical community. Furthermore, retrospective study of Gage's injuries continues to yield new information about brain injury, personality and rehabilitation.