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Pharmacological Treatment Paper

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Attention-deficit hyperactivity disorder, also known as ADHD, is one of the most frequent psychiatric conditions recognized in children (Am, 2007). There are concerns with over-diagnosing children with ADHD and over-treating them with medication, even among some professionals. Society is often unaware of the effects of pharmacological treatment for children with this disorder and early health interventions. Through research, this paper will address the long-term effects of pharmacological treatment for children with ADHD. Despite the fact that symptoms are relieved by medication, the question that still remains is how health and educational outcomes are affected by pharmacological treatment? Conversely, several medications and their side …show more content…

The long-term effects of children with ADHD persist into adulthood with co-morbidities and damaged psychosocial implementation (Europe Child Adolescence Psychiatry, 2011). There are over a hundred trials concerning drugs that treat ADHD, signifying that they are linked with an array of adverse effects. Generally, the adverse effects of the drugs are acceptable in society because they are labeled as mild or temporary. However, the specialized and public concern has been brought aware that these mild and temporary effects have the potential to cause severe effects that can include death and misuse of drugs. Children with ADHD are being exposed to drugs that have unreasonable hazards and effects that can harm their development and sleep.
Effects on Sleep Sangal et al. (2006) conducted a study on how methylphenidate and atomoxetine effects sleep in children with ADHD. Compared to methylphenidate, children and parents documented that atomoxetine helped them fall asleep faster, sleep better, and get up easier in the morning. The significant differences in both medications were that methylphenidate decreased children’s appetite, both had differing effects on sleep, and methylphenidate had greater occurrences of insomnia. Methylphenidate’s sleep-onset latency was more than atomoxetine’s, drastically. Even though the treatments enhance performances on tasks, they cause sympathomimetic effects from the increases

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