Death from deep vein thrombosis developed in a healthcare setting can potentially be prevented or reduced. Prophylactic measures backed by evidence based practice need to be taken to help these incidents. Awareness must be raised with nursing staff, medical doctors and other healthcare professions. Many different modalities can help prevent the development of deep vein thrombosis; this paper will concentrate a mechanical method. This research proposal examines whether the use of a pneumatic compression device reduces the incidence of developing deep vein thrombosis compared to no intervention during the period of hospitalization. The research design being utilized is a true experimental study. Participants will be placed into either the experimental group that will receive the intervention of the pneumatic compression device. The other control group will not receive any intervention. Possible implications of this study could be a change in protocol regarding pneumatic compression device practice. The study postulates that a pneumatic compression device will reduce the occurrence of deep vein thrombosis in the hospitalized patient.
Prophylactic Use of a Pneumatic Compression Device Pulmonary embolism resulting from deep vein thrombosis is the most common preventable cause of hospital death (Maynard, 2015). Consequently, the Surgeon General has called to action to prevent deep vein thrombosis. Deep vein thrombosis in the healthcare setting can be reduced through
All patients are at risk of pressure injuries. This risk is exacerbated when immobility and limited access to the surgically draped patient are a part of
Unfortunately, “VTE comprised of DVT and/or PE represent a serious public health challenge, affecting up to 600,000 Americans annually. The consequences can be deadly; VTE has been identified as the most common cause of preventable mortality in hospitalized patients, accounting for up to 10% of hospital deaths” (Shermock et al., 2013, p. 1) It is imperative that all patients admitted to the ICU should be assessed for VTE. The assessment should be done frequently. It is important to assess both subjective and objective data. Past medical history is very important. It is essential to find out if the patient has any predisposition to a VTE including any trauma to veins, any varicosities, obesity, COPD, HF. Certain medications can also put a patient at risk for VTE such as oral contraceptives, hormone therapy, tamoxifen, or raloxifene. Also, any recent surgeries such as orthopedic, gynecologic, gastric, or urologic and past surgeries involving veins or a central venous catheter can put a patient at risk. Objective data includes fear, anxiety, and pain. Monitor vital signs frequently. Check the integumentary system for symmetry; taut, shiny, warm skin, erythematous, tender to palpation. Not every patient
There are errors and hazards in care that occurred in the Mr. B scenario. One error was the emergency room physician’s failure to recognize the signs and symptoms of deep vein thrombosis (DVT) that Mr. B was presenting. If not treated early, a DVT can become a pulmonary embolism, a fatal condition that Mr. B unfortunately developed. Another error in care that happened in the Mr. B scenario is the nurses’ failure to monitor Mr. B’s ECG and respirations. Early detection of critical ECG and respiratory changes could have initiated medical interventions that would have saved Mr. B’s life. One hazard is the emergency room nurses’ heavy patient load at the time of Mr. B’s sentinel event. Another hazard is having a licensed
All the pre-operative checks I did are important for patient safety and are derived from the World Health Organisation (WHO) surgical safety checklist (2008).The WHO Surgical Safety Checklist was developed after extensive consultation aiming to decrease errors and adverse events, and increase teamwork and communication in surgery (WHO, 2014). The ward staff had already put on the anti –embolism stockings on the patient.
Pleural pigtail catheter placement associated with many complications including pneumothorax, hemorrhage, and chest pain. Air embolism can rarely be a complication of pigtail catheter insertion and has a high risk of occurrence with positive pressure ventilation (PPV).
Deep vein thrombosis (DVT) happens when a blood clot forms in one or more of the deep veins in your body, it usually takes place in your legs. Deep vein thrombosis can cause swelling or leg pains, and you may not get any symptoms at all. Deep vein thrombosis can arise if you have certain medical conditions that affect how your blood clots. Deep vein thrombosis can also happen if you don't move for a long time. I can happen sitting in a car for too long, after surgery, following an accident, or when you are confined to a hospital or nursing home bed. DVT is a serious condition because blood clots in your veins can break loose, travel through your bloodstream and lodge in your lungs. It can block the blood flow and cause a pulmonary embolism.
The authors used quite an extensive list of references, ranging in time frame from the years 1982-2014, which were used appropriately throughout the article and were aimed to give readers evidence on the background and complications of Central Venous Access Devices. The authors followed APA guidelines and used credible resources.
The purpose of this paper is to discuss pressure ulcers (PUs) and their prevention. The National Pressure Ulcer Advisory Panel (NPUAP), European Pressure Ulcer Advisory Panel (EPUAP), and Pan Pacific Pressure Injury Alliance (PPPIA) define pressure ulcers as a ‘localized injury to the skin and/or underlying tissue usually over a bony prominence, as a result of pressure, or pressure in combination with shear’ (Brown, 2016, p. S6). Pressure ulcers are a healthcare problem that can have detrimental effects on patients’ quality of life and can be regarded as an indicator of poor nursing practice. Hospital-acquired pressure ulcers (HAPUs) result in costly settlements
367). On the side of expert opinion, the CMS has had discourse regarding the potential broadening of events related to inadequate VTE prevention for both surgical procedures and medical patients (Maynard, 2016, p. 4).
[3] The nurse’s experience and the emergency medical care provider can explain some difficulties in cannula insertion. Success rate and time to apply intra-venous cannula is mandatory to obtain the best resuscitation of critical patients. Difficult intra-venous cannulation can be challenging even to the most experienced emergency nurse. Central venous catheterization (CVC) is an alternative route for cannulation in patients with difficult intra-venous access. CVC provides a fast effective intra-venous access for flued resuscitation and for Central Venous Pressure monitoring. However, venous thrombosis, catheter associated bloodstream infection, arterial puncture and pneumothorax are most common Central Venous Catheterization (CVC)
There are numerous reasons why one would look within the paramedicine professions rich history, a case can be made that it allows us to discover what practises were successful and improve upon them. Take the sheer size of Claude S. Beck’s defibrillator for instance, a cumbersome 18x18x24 inches (Eisenberg, 1998). Today there are iterations smaller than a loaf of bread, this increase in portability has led to monumental developments in health outcomes.
Pulmonary embolism (PE) is a blockage in a lung artery due to a clot.1 These clots are a result of a condition called deep venous thrombosis (DVT), the clots travel from veins usually in lower extremities through the bloodstream to the lungs, and block an artery.1 If not treated, this condition may lead to death depending on the size of the clot; therefore rapid treatment must be administered.1 The aim of this article is to analyze multiple peer reviewed research studies about the effects of thrombolytic therapy on the mortality and treatment escalation during the management of patients with an acute PE. The assumptive benefit of using thrombolytic therapy is that it can potentially break up clots instead of only preventing them from getting larger and dissolving clots will lead to an improved hemodynamic status in a short time period.2 However the effects of thrombolytic therapy are relatively unclear compared to the standard anticoagulation therapy which is prescribed more often.2
\textit{Pneumatic compression therapy is one of the most effective treatments for venous and lymphatic disorders, yet these devices are expensive and difficult to use in daily life due to its restriction on patient mobility. Consequently, we have developed an autonomous wearable compression therapy (AWCT) device that can reconfigure the treatment pressure automatically based on user activity and provides the required treatment without the active involvement of the patient or the carer. However, there is no available evaluation system to validate the performance of the AWCT. This paper therefore presents development of a low-cost robot arm for the evaluation of wearable devices by simulating kinetic parameter changes during walking and running
Stawicki SP et al(6) studied the correlation between inferior vena cava collapsibility index(IVC-CI) and central venous pressure(CVP). They used intensivist-performed bedside ultrasonography(INBU) for assessing IVC-CI. They divided the patients into three groups based on IVC-CI (<0.20,0.20-0.60 and >0.60). They concluded that IVC-CI correlate well with CVP in low(<0.20) and high(>0.60) collapsibility ranges.
The first parameter of intermittent compression is inflation pressure. The inflation pressure level has all been loosely correlated with blood pressure and patient comfort. One of the most common treatment protocols is using the pressure approximating the patient’s diastolic blood pressure has been used as a baseline measurement. Normally, the pressure that exceeds 30 mmHg encourages reabsorption of edema and also helps promote movement of the lymph. One must be carefully when using high pressure because if too high it would shut off arterial blood flow and therefore create a potentially uncomfortable tissue response as the result of low blood flow. This is just further proof that more is not necessarily better and you only need another pressure