Patient is a 58-year-old right-hand white male interviewed in the presence of his wife. He was previously seen by me last month for admission for the acute onset of vertigo with possible TIA or stroke as etiology. He stated that he awoke on 06/18/2015 in his usual state of health and then went to work and while there, he had the sudden onset of marked spinning vertigo. He was unable to walk straight, but denied any bilateral lower extremity weakness. He did have nausea and vomited several times. He initially denied any diplopia, visual field cut, blurred vision, facial asymmetry, facial or body sensory changes, dysarthria, or focal weakness. He continued to have marked vertigo in the emergency room, …show more content…
He also had right more than left mastoid opacifications and states that he was recently treated for otitis media. He has hypertension, hyperlipidemia, coronary disease and had been noncompliant with his medications in the past. His exam was essentially normal except for the subjective vertigo. There was no nystagmus and no diplopia on the initial exam. On 06/19/2015, he gave a different history. He states that he had a strike to the left temporal on Tuesday 06/16/2015. This did not result in any vertigo or any other neurological symptoms at that time. It was two days later that he had the vertigo at work. The patient also claimed that he had been seeing double since the previous night and the morning of the 19th. However, his neurological exam at that time, failed to reveal any actual disconjugate gaze. The patient had an MRI MRA, which revealed old white matter ischemic disease and mild intracranial atherosclerosis, but no evidence for acute stroke or posterior circulation significant stenosis. His diagnosis was labyrinthitis, possibly due to his bilateral mastoiditis. He was treated with Augmentin for 10 days. His symptoms resolved prior to discharge on meclizine. On physical therapy on discharge, he had no
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The patient is a 59-year-old right-handed white female who was admitted in June to Portsmouth Regional Hospital for what was determined to be either transient global amnesia or complicated migraine. I did review those notes. She was seen by Galina Simkin, MD. The symptomatology discussed in the H&P is consistent with transient global amnesia. She was having problems asking questions inappropriately, repeating sentences, repeating questions over and over again, and seeming somewhat confused. There were no other neurological symptoms at that time. No evidence for seizure activity. No evidence for stroke. She was brought to the emergency room, where she underwent a CAT scan, which was
Patient states that she has had no head injuries, past or present. Patient states she has never suffered from vertigo or dizziness. BB states that she rarely suffers from generalized headaches. Patient avoids taking pain relievers of any kind when she does present with head pain.
L.H. report no concurrent or severe headaches; There was no head trauma, syncope or vertigo. Patient wears corrective lens with no difficulty of vision or diplopia; absent of inflammation, discharge or lesion. Last eye exam was in September of 2016 with no history of glaucoma, cataracts. L.H. denies having any frequent colds, sinusitis, epistaxis and trauma. Patient reports having obstruction stating, “it happens when I am lying down” with an occasional postnasal drip.
Patient is a 50-year-old-year-old left-handed white male who presents with his wife for evaluation of multiple symptoms that have been present since an MVA in 02/2013. At that time, he was T. boned and his car was totaled. He has amnesia for the event and is unaware whether or not he hit his head. The airbag did deploy. Afterwards, he was confused and noted significant pain in his neck and upper back. Since then, he has had multiple symptoms. He does have involuntary twitching on the right, more than left, both hands, legs, and sometimes feet. He demonstrates one of these twitches and it looks like a focal myoclonic jerk of a limb. These occur on a daily basis. He also has problems with his left thumb and index finger locking up. When he is fatigued, especially when his neck gets tight, he has some problems with word finding, paraphasic errors, and syntax errors. He did see Anthony P. Knox,
Patient is a 60-year-old Latin American female, who presents with her husband for evaluation of some spells. She seems to have two different things going on. She did report intermittent episodes of feeling of weakness with blurred vision, diffuse paresthesias and a sensation she is about to pass out or the sensation of before undergoing generalized surgery, feeling like she is being sucked down. There is no loss of consciousness with this. She is unable to give any further history, except these events have been going on for about a month. They are almost daily. She does note they happen after eating, at which point, she will fall asleep easily. Otherwise, she denies any loss of consciousness,
They give a long list of diagnoses but the most prevalent is the fact that she has a rapidly progressing dementia. Note that she has a rapidly progressing dementia as well as a B12 deficiency. They describe a subdural hematoma in the CT scan reports. The one on 01/03 shows a lot of microvascular changes, a lot of cortical atrophy, and apparently, she had bilateral subdural hematomas that had converted to hygromas, but apparently the larger one on the left side still had some blood in it. When they repeated the CT scan of the head on 01/19, they commented that the hygromas were still present but there was less blood in the larger subdural. She had extensive blood testing, which basically was unremarkable. It did not appear that she had a urinary tract infection. Appears that since she has been here her status has been fairly stable. She was weak, but apparently, underwent physical therapy and made some improvement to where she became ambulatory in her gait. It looked like from the very beginning she was having a day/night confusion, was having a lot of un purposeful movements that might be have been contributed to either delusions or hallucinations. They gave her some Risperdal for the behavioral problems, but according to Cynthia she had taken Risperdal in the past and had an allergic reaction, and today when I have seen the patient there is a marked amount of periorbital edema
Ms. Castellanos reports she suffers from arthritis, dizziness, and headaches due to a head injury, hypertension, and cataract (both eyes). Client reports she underwent cataract surgery 10/8/15 in the left eye and 10/25/15 in the right eye. Client continues to reports she is currently taking the following medications: Meclizine 25mg, Simvastatin 40 mg, Gabapentin 300mg, Oyster Shell Calcium tab 500-200mg, Omega-3 Ethyl Esters 1mg, Losartan-HCI 100-12.5mgG, Amitiza 24 mg, and Sertraline. Client continues to report she is seen by Dr. Molinas Alveris tel3 718-4264747 located at 9319 Roosevelt Ave Fl 1, Jackson Heights, NY 11372.
The patient is an 85-year-old female who is brought to the ED by her family because of increasing confusion and supposedly she had a degree of altered mental status of two hours previous to presentation. In the ED she is completely worked up. CT shows advanced atrophy with microvascular changes and several lacunar infarcts nothing acute. Specific gravity in the urine reveals her to be markedly dehydrated. She culture completely, started on IV antibiotics, IV fluids and B12. On the day after admission she still presents as persistently confused. She is evaluated by PT. The patient who was formerly ambulating with a walker and allegedly driving a car is unable to be ambulated. Before the history indicates that she has a slow downward
Blood pressure -138/88, HR 71, Lung sounds –clear, temperature 98.8 F, radial pulse and pedal pulses +1 bi-laterally, normoactive bowel sounds. No history of smoking, drugs, alcohol use or diabetes; takes no daily medications. Surgical history: Hernia surgery September 2016 and cataract surgery September 2013. Moderately active, walks every day, sometime incorporating hand weights. Patient presents with minimal trembling unilaterally, (left side) when fingers stretched out, reports movements have been slower than normal. Patient’s wife reports “He’s been eating more slowly and it has been taking longer for him to get dressed in the morning.”. Upon examination it was determined that patient has reduced arm swing, slight stiffness in neck, difficulty rising from sitting position in the chair, masked facial features and deteriorated balance. No signs or symptoms of stroke.
I did receive a significant amount of records from this patient's PCP, as well as neurology consult in followup and infectious disease notes. In short, he is a 67-year-old right-handed white male who while living in Alaska developed some hip pain, as well as medial right hand numbness. He did have an EMG on 10/2000 that showed ulnar neuropathy with cubital tunnel syndrome on the right. Ulnar nerve transposition was considered, but the patient deferred this. He did have an MRI of the cervical spine, which revealed most significantly C3-4, moderate bilateral neuroforaminal narrowing, C4-5 severe left and moderate right neuroforaminal narrowing, C5-6 severe bilateral neuroforaminal narrowing. He did
The Alfred Hitchcock film; Vertigo is a narrative film that is a perfect example of a Hollywood Classical Film. I will be examining the following characteristics of the film Vertigo: 1)individual characters who act as casual agents, the main characters in Vertigo, 2)desire to reach to goals, 3)conflicts, 4)appointments, 5)deadlines, 6)James Stewart’s focus shifts and 7)Kim Novak’s characters drives the action in the film. Most of the film is viewed in the 3rd person, except for the reaction shots (point of view shot) which are seen through the eyes of the main character.(1st person) The film has a strong closure and uses continuity editing(180 degree rule). The stylistic (technical) film form of Vertigo makes the film much more
Based on the medical report dated 03/25/16, the patient continues to have significant headaches and bilateral neck and shoulder pain. IW has numbness and tingling in both arms with neck pain.
While BPPV may be the most common, Dr. Herman Jenkins, Professor and Chair of the Department of Otolaryngology at the University of Colorado School of Medicine and Health Science Center warns, "It can be many things in the elderly, from loss of function from a viral infection to dislodged crystals in the ear." Since vertigo may be caused by decreased blood flow to the base of the brain, blood clots,
Vertigo is a condition that causes everything to start spinning out of nowhere. This is caused by an inner ear condition. If you move so fast a certain way it will cause your body to be off balance which will cause you to feel a spinning sensation. This is caused by the calcium particles in your ears to block the canals. My dad has vertigo and there are sometimes when he has to go to the doctor to get his head shaken. The reason for this is to help get the calcium particles in your ears back on balance. When he first noticed something was wrong was when he moved and he almost fell due to everything spinning. He had almost all of the symptoms of vertigo which are nausea, eye jerking, headache, sweating, and ringing of the ear. Sadly there in
Vertigo is the sensation of motion or spinning while not in motion. Vertigo can cause inner ear disorders and central nervous system disorders. Some symptoms of vertigo are the sensation of moving and spinning, tinnitus; which is ringing in the ears, nausea, disequilibrium; imbalance, clumsiness;and ataxia, or loss of coordination in the muscles. Vertigo and it’s symptoms last depending on cause and may be episodic. Vertigo treatment usually depends on the cause,but here are some treatments they have. Otolith Repositioning, Inner Ear Surgery, Low-Salt Diet, Medications, Vestibular Rehabilitation Therapy, and Treatment of Underlying