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- 2.A person did not get food for 3 days and had lost his weight, ketone bodies were revealed in urine. To explain this: a)describe the main metabolic pathways providing energy for the liver cells during starvation and explain its regulation; b) discuss the hormonal changes; c)predict the deleterious effect of prolonged starvation.22. Which hormone is NOT derived from cholesterol? a. oxytocin b. testosterone c. both a and b d. neither a nor b 23. Which lipoprotein mostly contains triacyglycerol (TAGs)? a. HDL b. VLDL c. LDL d. chylomicrons 24. Which enzyme in cholesterol synthesis is inhibited by statins? a. HMG-CoA synthase b. HMG-CoA reductase c. both a and b d. neither a nor b 25. Which lipoprotein prevents the formation of plaque in the artery? a. HDL b. VLDL c. LDL d. chylomicrons 26. In which test/s will triacylglycerol yield a positive result? a. acrolein b. translucent spot c. both a and b d. neither a nor b 27. In which solvent will corn oil be soluble? a. hexane b. benzene c. both a and b d. neither a nor b 28. In which test/s will cholesterol yield a positive result? a. acrolein b. Huble's test c. both a and b d. neither a nor b 29. Which sample would test negative in saponification test? a. cholesterol b. 18:2 fatty acid c. both a and b d. neither a nor b 30. What test could distinguish…1. A lipogenase antagonist would a. increase prostaglandins b. increase inflammation c. decrease leukotrienes d. decrease prostaglandins 2. Which of the following is true if you give a patient a drug that blocked exocytosis? a. increase protein hormone synthesis b. increase steroid hormone synthesis c. decrease levels of protein hormones in the bloodstream d. decree levels of steroid hormones in the bloodstream
- 1.if the human body is stressed, glucocorticoids: a decrease potassium secretion from the kidneys. b. decrease glucose uptake by cells in the nervous system. c. increase the amount of sodium reabsorbed from urine in the kidneys d. inhibit the synthesis of glucose from noncarbohydrate sources. e promote the breakdown of proteins in the muscles and bones.A diabetic who injects too much insulin can lose consciousness. Explain why injecting excess insulin could impair brain function. Glucagon reverses this effect. Explain how.A diabetic who injects too much insulin can lose consciousness. Explain how injecting excess insulin can impair brain function. Explain also why an injection of glucagon can restore normal function.
- If the human body is stressed, glucocorticoids: a. promote the breakdown of proteins in the muscles and bones. b. increase the amount of sodium reabsorbed from urine in the kidneys. c. decrease potassium secretion from the kidneys. d. decrease glucose uptake by cells in the nervous system. e. inhibit the synthesis of glucose from noncarbohydrate sources.Which is true of brain metabolism in starvation? a. The brain can use glucogenic amino acids for energy b. The brain can only use glucose as fuel c. Up to a quarter of the energy requirement of the brain can come from fatty acids. d. Up to half the energy requirement of the brain can be met by ketone bodiesAs a result of complete fasting for 3 days, a significant change in metabolism occurs. How will the level of fatty acids in the blood change? What hormone causes these changes? Justify your answer schematically.
- 1.a. Given what you know about glycolysis and gluconeogenesis, does it make sense that insulin activates PDH phosphatase? Why? b.How do vitamins increase to the breadth of chemical reactions available within a biological system?1. If obesity was purely related to the hormonal imbalance of these hunger/satiation hormones, what would we expect to see in obese individuals? What do we observe? 2. What is a common feature between food overconsumption, drug addiction, and dopamine receptor (D2) in certain individuals? Why is this significant for possible treatments? 3. Describe the drugs Belviq, and Rimonabant and their possible role in hunger control.Bile salts are responsible for the... A. transport of triacylglycerols to adipocytes. B. transport of fatty acids from adipocytes to myocytes. C. exposure of triacylglycerols to intestinal lipases. D. prevention of triacylglycerol to diacylglycerol degradation.