i need the answer quickly 7. A 22-year-old patient suffering argininosuccinic aciduria from little wasprescribed the treatment by administration of keto-analogs of valine, leucine,isoleucine and phenylalanine against the background of reduced protein in thediet during two weeks. The doctor noted the decrease of ammonia concentrationin plasma from 0.09 mg/dL to 0.04 mg/dL as a result, and arginine succinateexcretion was reduced from 2 to 0.8 g/24h. What is the molecular mechanism ofthe curative effect of amino acids keto-analogs? For the answer:a) write down the scheme of ammonia detoxification in the liver;b) explain the causc of argininosuccinic aciduria;c) make up the scheme of present amino acid keto-analogs usage for thereduction of hyperammoniemia.

Argininosuccinic aciduria: This is an inherited disorder. In this condition, the ammonia gets…

Answered: argininosuccinic aciduria from little…

Phlebotomy Essentials

6th Edition

ISBN:9781451194524

Author:Ruth McCall, Cathee M. Tankersley MT(ASCP)

Publisher:Ruth McCall, Cathee M. Tankersley MT(ASCP)

Chapter1: Phlebotomy: Past And Present And The Healthcare Setting

Section: Chapter Questions

Problem 1SRQ

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3. What is the significance of the levelling off in the titration curves in terms of the buffering action of amino acids?
3 i. Define Biological Value (BV) of Proteinsii. What are the advantages of this protein evaluation method when compared to Nbalance and chemical score?5iii. A bull consumes 7 kg DM of feed containing 60 g N/kg. The bull excretes 120 g N inthe faeces and 50 g N in the urine. The feacal and urinary N had 20 g metabolic feacalnitrogen (MFN) and 6 g endogenous urinary nitrogen (EUN) respectively.Calculate the BV of the protein in the diet.
i. Explain the behaviour of the amino acids (Alanine, Lysine, aspartic acid) during the ion exchangeprocedure.ii. Explain the behaviour of the amino acids (Alanine, Lysine, aspartic acid) during TLC. How does thesolvent affect the rate of migration iii. How does the column chromatography the amino acids (Alanine, Lysine, aspartic acid) with fraction eluted with HCl and phosphate buffer have an effect in cellulose TLC and the actual TLC?
1. What are the different methods in determining BUN? 2. What are the principles of the colorimetric and enzymatic methods for quantitating urea? 3. What is the clinical significance associated with detecting abnormal level of urea in the blood?
1. A physician orders 800 mg of acyclovir IV q8h x10 days for a patient with shingles. Acyclovir comes in a powdered form in a 0.5 g vial, 10 mL. The directions state it should be reconstituted with 5 mL of normal saline. How many mL would contain the ordered dosage?
1. Explain the biochemical formation of creatinine, urea, and uric acid. 2. State the principle of the Jaffe (end-point, and kinetic) and the enzymatic methods for measuring creatine.
1. How much glucose ( MW: 180) we have to add to 2 L normal saline to make 300mOsmol/L?
1) Anemia, hemorrhage, and chronic obstructive pulmonary disease can all cause metabolic acidosis. The best explanation is that the lack of oxygen causes: A) a decrease in insulin that, in turn, increases anaerobic glycolysis in the brain B) a decrease in oxidative phosphorylation so the cells have to rely upon anaerobic glycolysis C) a decrease in the oxidation of tyrosine to epinephrine which decreases gluconeogenesis in muscle D) a decrease in the removal of CO2 from the blood. The resulting decrease in pH causes an increase in glycolysis in most cells E) an increase in glycolysis in red blood cells 2) Which of the following is NOT TRUE following β-oxidation in the liver? a) The formation of propionyl CoA could lead to the production of glucose from B-oxidation b) The formation of acetyl CoA could lead to ketone bodies synthesis c) Acetyl CoA could be converted to pyruvate allowing for the production of glucose d) Acetyl CoA could be used for the synthesis of HMG CoA e) Acetyl CoA…
Consider the decapeptide angiotensin I. Treatment of angiotensin I with ACE (the angiotensin-convertingenzyme) cleaves only the amide bond with the carbonyl group derivedfrom phenylalanine to afford two products. The larger polypeptide isangiotensin II, a hormone that narrows blood vessels and increasesblood pressure. Give the amino acid sequence of angiotensin II usingthree-letter abbreviations. ACE inhibitors are drugs that lower bloodpressure by inhibiting the ACE enzyme
Make a figure that includes the hydropathy profile analysis, the TMHMM analysis and the TOPCONS analysis of AlaE attached. Describe the purpose of these analyses, how they work, and what your analyses suggest with regards to the topology of AlaE.
An amino acid mixture consisting of lysine,leucine, and glutamic acid is to be separated by ion-exchangechromatography, using a cation-exchange resin at pH 3.5, with theeluting buffer at the same pH. Which of these amino acids will beeluted from the column first? Will any other treatment be neededto elute one of these amino acids from the column?
7. During amino acid catabolism, how is amino group removed and excreted in the form of urea? Please write the reactions starting from a generic amino acid to the formation of urea. Please indicate the names of enzymes, cofactors, names of substrates, and products of each reaction and the location of the reaction i.e. whether in cytoplasm or mitochondria.

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