Assume that a person was born with a defective NADH dehydrogenase complex, the complex that allows electron transport chain (ETC) to make ATP from NADH. Following consumption of a diet rich in carbohydrates and fats, would you expect this person to be able to produce any ATP by oxidative phosphorylation? Explain your answer?

Assume that a person was born with a defective NADH dehydrogenase complex, the complex that allows electron transport chain (ETC) to make ATP from NADH. Following consumption of a diet rich in carbohydrates and fats, would you expect this person to be able to produce any ATP by oxidative phosphorylation? Explain your answer?

Human Physiology: From Cells to Systems (MindTap Course List)

9th Edition

ISBN:9781285866932

Author:Lauralee Sherwood

Publisher:Lauralee Sherwood

Chapter2: Cell Physiology

Section: Chapter Questions

Problem 2TAHL

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Similar questions

Suppose a liver cell developed a mutation such that all of its ATP synthase complexes are now super efficient, and they now only require 1 H+ to pass through the complex to produce one molecule of 1 ATP! How many net ATP would be produced in these liver cells from the complete oxidation of 1 molecule of glucose?
Maximal production of ATP from glucose involves the reactions of glycolysis, the citric acid cycle, and the electron transport chain. Which of these reactions requires O2, and why? Which, in certain organisms or physiological conditions, can proceed in the absence of O2?
Would you expect the citric acid cycle to bemore or less active when a cell has a high ATP/ADP ratio and ahigh NADH/NAD1 ratio? Give the reason for your answer.
For each molecule of glucose that enters cellular respiration, what would be the combined products of glycolysis, pyruvate oxidation, and the citric acid cycle? A) 4 ATP, 6 CO2, 10 NADH, and 2 FADH2 B) 1 ATP, 2 CO2, 3 NADH, and 1 FADH2 C) 4 ATP, 4 CO2, 6 NADH, and 3 FADH2 D) 2 ATP, 6 CO2 10 NADH, and 2 FADH2
Assuming that the glycerol 3-phosphate shuttle is operative, a complete oxidation of five molecules of glucose (by a eukaryotic cell) into carbon dioxide and water would produce approximately _________ ATP molecules via oxidative phosphorylation only. (Consider NADH = 2.5 ATP and FADH2 = 1.5 ATP)
If ATP synthase completely stopped working, could glycolysis or the citric acid cycle still produce any ATP? Explain.
If cellular respiration were the stock market (you’re investing glucose and pyruvate, and receiving ATP Dividends, where would you get the most return on your investments (glycolysis, Krebs cycle, or electron transport chain) explain why.
The energy investing phase of glycolysis produces 4 ATP and 2 NADH molecules. true or false?
You know that during oxidative phosphorylation, 1 NADH creates 2.5 ATP and 1 FADH2 creates 1.5 ATP. From this information, how many total ATP molecules will be produced from the complete oxidation of pyruvate?
The energy stored in one molecule of NADH is used to synthesize how many molecules of ATP?
How is energy carried from glucose into the mitochondrial electron transport chain? Why is oxygen necessary for oxidative phosphorylation and cellular respiration? Compare and contrast the metabolic fate of pyruvate when oxygen is present or absent.
Discuss the operation of the citric acid cycle and the electron transport chain in the aerobic production of ATP. What is the function of NAD+ and FAD in these pathways?