In an epic battle, the Green Goblin was able to wound Spider-Man aind collect a samplle of his cellis. Usllizing a HuMiouse modiel organism, the Goblin got the mutated chromosomes into the animals and began breeding them to determine the basis of their chromosomal inheritance. In the process of conducting his research, he discovered the gene associated with spider-like powers are actually three distinct genes: one for spider sense: one for super strength: one for enhanced agility. All genes appear to be dominant, so the Goblin runs a test cross and gets the following results: Phenotype Agility Number of Progeny 94 Agility and Sense 3 All 3 Powers 580 Sense 40 Strength and Agility Strength and Sense Strength 45 89 15 592 No Powers What parental genotypes were crossed to get this data? O Heterozygous with a homozygous recessive D Homozygous dominant with a homozygous recessive O Heterozygous with another heterozygous Cannot determine with this information Heterozygous with a homozygous dominant

In an epic battle, the Green Goblin was able to wound Spider-Man aind collect a samplle of his cellis. Usllizing a HuMiouse modiel organism, the Goblin got the mutated chromosomes into the animals and began breeding them to determine the basis of their chromosomal inheritance. In the process of conducting his research, he discovered the gene associated with spider-like powers are actually three distinct genes: one for spider sense: one for super strength: one for enhanced agility. All genes appear to be dominant, so the Goblin runs a test cross and gets the following results: Phenotype Agility Number of Progeny 94 Agility and Sense 3 All 3 Powers 580 Sense 40 Strength and Agility Strength and Sense Strength 45 89 15 592 No Powers What parental genotypes were crossed to get this data? O Heterozygous with a homozygous recessive D Homozygous dominant with a homozygous recessive O Heterozygous with another heterozygous Cannot determine with this information Heterozygous with a homozygous dominant

Human Heredity: Principles and Issues (MindTap Course List)

11th Edition

ISBN:9781305251052

Author:Michael Cummings

Publisher:Michael Cummings

Chapter6: Cytogenetics: Karyotypes And Chromosome Aberrations

Section: Chapter Questions

Problem 17QP

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Similar questions

The karyotype of a young girl who is affected with familialDown syndrome revealed a total of 46 chromosomes. Her olderbrother, however, who is phenotypically unaffected, actually had45 chromosomes. Explain how this could happen. What wouldyou expect to be the numbers of chromosomes in the parents ofthese two children?
Describe when X-chromosome inactivation occurs and how thisleads to phenotypic results at the organism level. In your answer,you should explain why XCI causes results such as variegated coatpatterns in mammals. Why do two different calico cats have theirpatches of orange and black fur in different places? Explainwhether or not a variegated coat pattern due to XCI could occur inmarsupials.
On rare occasions, people are born with a condition known as uniparental disomy. It happens when an individual inherits both copies of achromosome from one parent and no copies from the other parent. Thisoccurs when two abnormal gametes happen to complement each otherto produce a diploid zygote. For example, an abnormal sperm thatlacks chromosome 15 could fertilize an egg that contains two copies ofchromosome 15. In this situation, the individual has maternal uniparental disomy 15 because both copies of chromosome 15 were inheritedfrom the mother. Alternatively, an abnormal sperm with two copies ofchromosome 15 could fertilize an egg with no copies. This is known aspaternal uniparental disomy 15. If a female is born with paternal uniparental disomy 15, would you expect her to be phenotypically normal,have Angelman syndrome (AS), or have Prader-Willi syndrome(PWS)? Explain. Would you expect her to produce normal offspring oroffspring affected with AS or PWS?
Female fruit flies homozygous for the X-linked white-eye alleleare crossed to males with red eyes. On very rare occasions, an offspringof such a cross is a male with red eyes. Assuming these rareoffspring are not due to a new mutation in one of the mother’s Xchromosomes that converted the white-eye allele into a red-eyeallele, explain how a red-eyed male arises.
Is it more indicated for ageneticist desiring to map theX chromosome of the motherof a given family (theresearcher does not haveaccess to her DNA, only accessto the genetic material of theoffspring) to analyze thechromosomes of herdaughters or of her sons?
Genes that cause Prader-Willi syndrome and Angelman syndromeare closely linked along chromosome 15. Although people withthese syndromes do not usually reproduce, let’s suppose that acouple produces two children with Angelman syndrome. The oldest child (named Pat) grows up and has two children with PraderWilli syndrome. The second child (named Robin) grows up andhas one child with Angelman syndrome.A. Are Pat and Robin’s parents both phenotypically normal ordoes one of them have Angelman or Prader-Willi syndrome? Ifone of them has a disorder, state whether it is the mother or thefather, and explain how you know.B. What are the sexes of Pat and Robin? Explain.
The maternal-effect mutation bicoid (bcd) is recessive. Inthe absence of the bicoid protein product, embryogenesis isnot completed. Consider a cross between a female heterozygousfor the bicoid mutation (bcd+/ bcd-) and a homozygousmale(bcd-/ bcd-). Predict the outcome (normal vs. failed embryogenesis) inthe F1 and F2 generations of the cross described.
A couple planning their family are aware that through the past threegenerations on the husband's side a substantial number of stillbirthshave occurred and several malformed babies were born who diedearly in childhood. The wife has studies genetics and urges herhusband to visit a genetic counseling clinic, where a completekaryotype-banding analysis is performed. Although the tests showthat he has a normal complement of 46 chromosomes, bandinganalysis reveals that one member of the chromosome 1 pair (ingroup A) contains an inversion covering 70 percent of its length.The homolog of chromosome 1 and all other chromosomes showthe normal banding sequence.(a) How would you explain the high incidence of past stillbirths?predictabouttheprobabilityof(b) What can you abnormality/normality of their future children?(c) Would you advise the woman that she will have to bring each pregnancy to term to determine whether the fetus is normal? Ifnot, what else can you suggest?
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Genes that cause Prader-Willi syndrome and Angelman syndromeare closely linked along chromosome 15. Although people withthese syndromes do not usually reproduce, let’s suppose that acouple produces two children with Angelman syndrome. The oldestchild (named Pat) grows up and has two children with Prader-Willi syndrome. The second child (named Robin) grows up andhas one child with Angelman syndrome.A. Are Pat and Robin’s parents both phenotypically normal ordoes one of them have Angelman or Prader-Willi syndrome? Ifone of them has a disorder, state whether it is the mother or thefather, and explain how you know.B. What are the sexes of Pat and Robin?