How does the Na+/ K+ ATPase play a role in glucose transport? phosphorylates GLUT1 to activate it pumps Na+ across plasma membranes into cytoplasm to activate the sodium-glucose transporter pumps Na+ across basal plasma membrane to the outside of intestinal cells so that co-transport of glucose with Na+ into cytoplasm is facilitated allows for the diffusion of K+ inside of cell so that co-transport with glucose is facilitated

How does the Na+/ K+ ATPase play a role in glucose transport? phosphorylates GLUT1 to activate it pumps Na+ across plasma membranes into cytoplasm to activate the sodium-glucose transporter pumps Na+ across basal plasma membrane to the outside of intestinal cells so that co-transport of glucose with Na+ into cytoplasm is facilitated allows for the diffusion of K+ inside of cell so that co-transport with glucose is facilitated

Human Physiology: From Cells to Systems (MindTap Course List)

9th Edition

ISBN:9781285866932

Author:Lauralee Sherwood

Publisher:Lauralee Sherwood

Chapter2: Cell Physiology

Section: Chapter Questions

Problem 10RE: Using the answer code on the right, indicate which form of energy production is being described: 1....

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Digoxin, a toxin derived from the foxglove (shown), can be used to treat heart disorders such as atrial fibrillation. Digoxin’s mechanism of action is to inhibit the Na+/K+ ATPase. Which of the following is the most likely side effect of Digoxin treatment? Failure to transport glucose into cells during an extended fast Failure to transport glucose into cells after after eating a large meal Enhanced response of muscle cells to Acetylcholine Failure to transport Cl- out of the cell using direct active transport
If a mutation in a sodium channel in an Intestinal epithelial cell caused a marked decrease In the concentration gradient of sodium across the plasma membrane, which of the following would you expect to observe? a decrease in Na/K ATPase activity. an increase in the movement of glucose across the basolateral membrane to the extracellular space an increase in sodium/glucose co-transport into the cell across the apical membrane a decrease in cytoplasmic glucose concentration
what type of glucose transport protein is found in pancreatic beta cells? SGLUT-1 GLUT-2 GLUT-3 GLUT-4 GLUT-5
Which of the following statements best describes the absorption of glucose? Absorption of glucose requires digestion by aminopeptidases on absorptive epithelial microvilli before absorption of the carbohydrate smaller units such as glucose. Absorption of glucose requires an electrochemical gradient of high extracellular K+ set up by primary active transport. Absorption of glucose occurs at villi and crypts mainly in the duodenum where amylases break down carbohydrates into glucose. Absorption of glucose occurs at the transporting epithelia in the small intestine and is a secondary active transport that requires a Na+gradient
Insulin signals activate all of the following enzymes except: Phosphoinositide 3-kinase (PI3K) Glycogen synthase kinase 3 (GSK3) Protein kinase B (PKB; or Akt) All of the above enzymes are activated by insulin signals Citrate move in and out of mitochondria via a specific transport protein. What would happen if aninhibitor completely inhibited this transport system? Fatty acid biosynthesis would slow down Ketone body synthesis would slow down Both A and B Neither A nor B
Order the steps (from 1-16) by which a person can generate ATP from dietary fat. ATP synthesis coupled to the movement of protons down the proton concentration gradient occurs. NADH and FADH2 electrons are transferred to O2 Acetyl-CoA are degraded to GTP, NADH and FADH2 in citric acid cycle. Fatty acids are degraded to acetyl-CoA, NADH…
A chemical called ouabain inhibits the Na/K ATPase (the sodium-potassium pump). What would happen to glucose concentrations in the extracellular fluid (not the gut lumen) after exposure to ouabain, and why? Ouabain would cause intracellular sodium levels to rise, which would reduce the active transport of glucose from the gut, thereby decreasing glucose in the extracellular fluid. Ouabain would cause extracellular sodium levels to rise, which would reduce the active transport of glucose from the gut, thereby decreasing glucose in the extracellular fluid. Ouabain would cause intracellular sodium levels to rise, which would reduce the active transport of glucose from the cell to the extracellular fluid. Ouabain would cause intracellular sodium levels to rise, which would enhance the active transport of glucose from the gut, thereby increasing glucose in the extracellular fluid.
What is the advantage of using ATP as a common energy source?Another way of asking this question is, “Why does ATP provide anadvantage over using a bunch of different food molecules?” For example,instead of just having a Na+/K+-ATPase in a cell, why not have manydifferent ion pumps, each driven by a different food molecule, like aNa+/K+-glucosase (a pump that uses glucose), a Na+/K+-sucrase (a pumpthat uses sucrose), a Na+/K+-fatty acidase (a pump that uses fatty acids),and so on?
If our body lack of insulin, is it mean the glucose in our blood is low concentration now. So is it will effect pathways in mucles cell like glycolysis, fatty acids synthesis, glycogen synthesis, gluconeogenesis (increase or decrease)? I know the liver can regconize that low or high concentration of blood glucose so insulin or glucagon can act as the signal for liver can know, but is the muscle cell can do it to? If not, how they know to adjust their pathways. And if our body lack of insulin, is the liver cells response same as glucagon signaling.
An increase in calcium concentration can be associated with an increase in stomach acidity. This process is similar to the processes involved in neurotransmitter and insulin release. How is calcium associated with increased stomach acid? a. Calcium binds to the alpha subunit of a G protein activating proton pumps already on the membrane. b. Calcium is pumped out of the cell by an ATPase and as a result this lowers the pH of the gut. c. Calcium influx into the cell allows vesicles containing proton pumps on their membranes to fuse with the cellular membrane. d. Calcium influx into the cell allows vesicles filled with protons to fuse with the cellular membrane. e. Calcium binds to acetylcholine receptors on Parietal cells and opens gated proton channels releasing protons from the cytoplasm to outside the cell.
If sucrase breaks down sucrose and a mystery sugar inhibits sucrase from breaking down sucrose allowing sucrose to mobilize in the body, what kind of inhibition is the mystery sugar? i.e. uncompetitive, competitive, non-competitive
Which of the following statements is TRUE regarding regulation of glycolyis? hexokinase regulates glycolysis by degrading glucose-6-phosphate low levels of ATP inhibit phosphofructokinase-1 because ATP preferentially binds to the allosteric inhibition site inhibition of glucose-6-phosphate by hexokinase is an example of feedback inhibition none of these statements are correct I selected option C and got it wrong, what is the correct answer with an explanation to why it is and also how was my answer wrong?

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		phosphorylates GLUT1 to activate it
		pumps Na+ across plasma membranes into cytoplasm to activate the sodium-glucose transporter
		pumps Na+ across basal plasma membrane to the outside of intestinal cells so that co-transport of glucose with Na+ into cytoplasm is facilitated
		allows for the diffusion of K+ inside of cell so that co-transport with glucose is facilitated