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- State 3 factors that affect Stroke VolumeWhat effect does sympathetic stimulation have on stroke volume if thevenous return remains constant? Dilation of the coronary blood vesselsoccurs in response to an increased heart rate and stroke volume. Explainthe functional advantage of that effect.Explain the relationship between stroke volume and venous return.
- There are three major factors that affect stroke volume and therefore affect cardiac output: afterload, preload, and contractility. Explain how and why each of these factors affects cardiac output. Each answer must explain what the factor is as well as how it affects cardiac output.There are three major factors that affect stroke volume and therefore affect cardiac output: afterload, preload, and contractility. Select two of these factors and explain how and why each affects cardiac output.What is the function of the pulmonary circulation In a resting state, explain what happened to the heart rate when the stroke volume decreased. Why does this occur? DescribetheFlank-Starlinglawintheheart. Compare the structural features of the different chambers and valves of the heart and how this relates to their function. Explain the flow of the blood as it passes through the heart. Describe how the heart alters stroke volume under sympathetic stimulation. please answer the queations into 2 paragraphs. thanks.
- Which of the following would increase flow? Vasoconstriction of the artery An increase in blood viscosity An increase in resistance An increase in stroke volumeStroke volume is thea. amount of blood pumped by the heart per minute.b. difference between end-diastolic and end-systolic volume.c. difference between the amount of blood pumped at rest and thatpumped at maximum output.d. amount of blood pumped from the atria into the ventriclesParasympathetic stimulation decreases the heart rate by Decreasing permeability of SA node cells to Ca2+ increasing ion influx, thus increasing the rate of depolarization. Increasing the permeability of SA node cells to Ca2+. Increasing the permeability of SA (sinoatrial) node cells to K+.