Mutated genes that promote cell proliferation are called: Question 23 options: Proto-oncogenes Oncogenes
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Question 23 options:
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Proto-oncogenes
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Oncogenes
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Solved in 2 steps
- Match the following terms with their definition. Note that some of the terms will not be used. 1. S phase 2. Oncogene 3. G0 phase 4. Cyclin 5. Metaphase checkpoint 6. G2 phase 7. G1 checkpoint 8. Cyclin-dependent kinase 9. Mitosis ______The stage a cell is in if it is not going to divide ______The part of the cell cycle during which newly replicated DNA is checked for mistakes ______The part of the cell cycle during which DNA replication occurs ______An enzyme that adds phosphate groups to other proteins only when it is bound to another protein ______A protein that is present at high levels only during certain times of the cell cycle _____Point at which the cell dies if all the chromosomes are not correctly attached to the mitotic spindle_____________________ describes the imbalance in genetic information due to the removal of controls at cell cycle checkpoints. Group of answer choices Tumor mosaicism Oncogene Benign tumor Genomic instabilityAnswer each question with a paragraph; 3-5 sentences each. Explain the difference between a proto-oncogene and a tumor-suppressor gene. Explain the difference between prokaryotic and eukaryotic cell division. Explain the difference between positive regulation of the cell cycle and negative regulation of the cell cycle. Explain the difference between normal p53 and mutated p53. Find two differences and two similarities between binary fission and mitosis.
- O Tumors of the blood A tumor with limited growth Explain why radiation and 5-fluorouracil are effective cancer treatments. Please include a few sentences about how these treatments interact with the cell cycle. Your answer Any comments please use this space (e.g. to clarify an answer or point o issues on questions, etc.)Carcinogens generate mutations in: Question 17 options: anti-oncogenes and tumor activation genes proto-oncogenes and tumor suppressor genes proto-oncogenes and tumor activation genes anti-oncogenes and tumor suppressor genesCan you please answer the following three questions (one paragraph for each question). Your answers need to be in your own words. Please make sure everything is from your own words 1. In a paragraph, explain what is cancer? 2. In a paragraph, explain why cancer exists, what causes it, and other important things that relates to cancer in general? 3. In a paragraph, explain how different cancers affect different population? Explain also the stages of cancer Remember your work should be in your own words. For anything you use, please cite it in APA.
- p53 is a gene / protein often associated with cancer. Why? What does p53 do? What kind of gene is it? Is it associated more with any one particular type of cancer or all cancers? Tell me more about p53, but please do not exceed one typed page.D Question 18 Your patient's biopsy report indicates they have "cancer in situ". This means: Abnormal cells are present, they have not invaded local cells O Cells are normal, they will become cancer cells in the near future Abnormal cells are present, they have invaded a small number of local cells Cells are normal, they will not become cancer cells unless stimulated « PreviousThe direct cause of cancer is DNA damage in genes that control the cell cycle. Question 13 options: True False
- Please explain how cancer is a disease of the cell cycle checkpoints. you must explain how the function of p53 and proto-oncogenes, how alterations in p53 or proto-oncogenes can cause cell cycle alteration, and how this leads to the typical phenotypes seen in cancer cells.You are in charge of a new gene therapy clinic. Two cases have been referred to you for review and possible therapy. Case 1. A mutation in the promoter of a proto-oncogene causes the gene to make too much of its normal product, a receptor protein that promotes cell division. The uncontrolled cell division has caused cancer. Case 2. A mutation in an exon of a tumor-suppressor gene makes this gene nonfunctional. The product of this gene normally suppresses cell division. The mutant gene cannot suppress cell division, and this has led to cancer. What treatment options can you suggest for each case?12/ cell determination is the expression of the cell's specialized role true or false
