What did Enterobacter aerogenes to do with the Lactose negative go to the Serratia liquefaciens? What does Enterobacter aerogenes causes disease in humans? What did Enterobacter aerogenes to do with oxidase?
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What did Enterobacter aerogenes to do with the Lactose negative go to the Serratia liquefaciens? What does Enterobacter aerogenes causes disease in humans? What did Enterobacter aerogenes to do with oxidase?
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- Would gut fermentation syndrome best be described as anabolic or catabolic? Besides the alcoholic pathway, what other pathway exists for the fermentation of carbohydrates?Why is gluconeogenesis important in the cori cycle in 3 sentences onlyWhat does the negative of the Methyl Red for the Enterobacter aerogenes to do?
- Is Oxaloacetate, a TCA cycle intermediate also a metabolite in gluconeogenesis? If not, which ones of these TCA cycle intermediates are also a metabolite in gluconeogenesis? Succinate Oxaloacetate Pyruvate CitrateIn the liver, the relative activity of ornithine transcarbamylase is high, that of argininosuccinate synthetase is low, and that of arginase is high. Why is it important that ornithine transcarbamylase activity be high in the liver? What might be the consequence if arginase activity is low or defective?In the intestine, the triacylglycerols must be converted to fatty acids and glycerol by hydrolytic enzymes before transport into enterocytes. Afterward, fatty acids and glycerol are reconverted into triacylglycerols and then packaged into chylomicrons. Suggest why this energy-requiring process is used instead of a direct transport of triacylglycerols into enterocytes.
- All of the following are true with respect to alpha-glucosidase inhibitor, except:A. It inhibits intestinal alpha-glucosidase B. It decreases enzymatic conversion of oligo- to monosaccharidesC. It lowers postprandial glucoseD. It causes gastrointestinal disturbances.E. It may cause development of hypertension.Why is oxaloacetate depletion from the TCA cycle important for the initiation of ketogenesis? What causes this oxaloacetate depletion? What tissue type in our body is this oxaloacetate depletion found and why is it not found in other tissues?The peroxisomal enzyme b-ketoacyl–CoA-thiolase does notbind medium-chain acyl-CoA, in contrast to the analogousmitochondrial enzyme. Explain why this phenomenon is anadvantage to the cell.
- salvia does not contain amylase until babies are two months old. How could thu affect an infant's digestve requirements?Amino acids that degrade, yielding acetyl-CoA or acetoacetylCoA, are referred to as _________________.How does placing the solution in a boiling bath terminate the hydrolysis reaction mediated by salivary α-amylase?