Hypermobility

in Parctice (n.d.) reports that diagnosis of EDS is hampered by the fact that symptoms of EDS such as joint hypermobility, can mimic normal pre-adolescent growth. Ehlers-Danlos syndrome hypermobility type, also known as Joint Hypermobility Syndrome (EDS-HT/JHS), is the most common hereditary disorder of the connective tissue (HDCT). It is characterized by tissue fragility, joint hypermobility and a wide range of articular and non-articular manifestations, which often appear in infancy (Baeza-Velascoa

not really a part of the body that is left unaffected. Ehlers Danlos can be described as, “widespread manifestations in skin, ligaments, joints, blood vessels and internal organs” (De Paepe, Malfait 2012). Ehlers Danlos Syndrome, or EDS, causes hypermobility in the joints and connective tissues of the body, especially those of the musculoskeletal system. EDS can also cause “velvety” skin that is highly stretchable and elastic. Since the walls of the vascular structures

Classical Type (further divided in gravis and mitis types, the former being a milder version of the former), Hypermobility Type, Vascular Type, Kyphoscoliosis Type, Arthrochalasia Type, and Dermatosparaxis Type (Ehlers-Danlos syndrome, 2015) . For the former three types, current

Joint hypermobility (JH), which is when the joint has an exaggerated range of motion, may present in several ways and is involved in a variety of conditions. Joint Hypermobility Syndrome (JHS) is when the joint has increased range of motion in conjunction with musculoskeletal pain(73, 74). This entity can exist in conditions such as Generalized Joint Hypermobility Syndrome, Benign Joint Hypermobility Syndrome, and Ehlers Danlos Syndrome (Hypermobile Type)(73, 75, 76). Children with JHS may complain

gene abnormality responsible is either COL5A1 or COL5A2. The disease is diagnosed through clinical examination using the beighton diagnostic scale, a test to determine joint hypermobility as well as family history. Classical type is determined by in order of occurrence; skin hyperelasticity, Wide atrophic scars, joint hypermobility, velvety skin, easy bruising, subluxations and dislocations, flat feet, muscle hypotonia, gross motor disease, postoperative complications, tissue fragility. Although there

The most dominant type, hypermobility type, causes subluxation, dislocation and even tears in the joints. Vascular type, which is regarded as the most serious type, can cause organ or arterial rupture. Classical type has most of the qualities of Hypermobility type as well as hyperextendability of the skin. This is just to name a few of the six predominant types of EDS. Lots of people have Hypermobility type without even knowing it. One of the symptoms for hypermobility type of EDS is Cronin pain

Classical, Hypermobility, Vascular and Arthrochalasia forms of EDS are transmitted as an autosomal dominant trait. Kyphoscoliosis and Dermatosparaxis type of EDS are transmitted as an autosomal recessive trait. Classical EDS results from a faulty collagen V genes. A decrease

The manifestations in EDS continues to be difficult in characterizing and quantifying the prevalence of the many clinical symptoms seen in this syndrome. Study subjects are often diagnosed later in the disease process which causes disproportionate groups with few subjects in the early symptomatic stages. The groups that have been studied demonstrate the following clinical issues. Skin hyper-extensibility is the primary Dermatologic feature seen in EDS. To test the skin doctors find a neutral area

Ehlers-Danlos Syndrome (Hypermobility Type) Ehlers-Danlos Syndrome (Hypermobility Type) is a genetic connective tissue disorder characterized by joint laxity, velvety skin, joint pain, and other widespread complications of the body. It can be inherited from a parent with the same faulty gene, or it can be a newly developed mutation. Connective tissue is infused with collagen protein that provides strength and elasticity. It is spread abundantly throughout the human body in skin, muscles, tendons

various types and causes including, hypermobility, hypomobility, anterior or posterior rotation, or up slip of the pelvis. Types of Dysfunction Hypermobility – hypermobility within the joint causes decreased stability. Causes may be weak supporting muscles, trauma, or increased ligament laxity (hormonal). Often seen with pregnant women as the ligaments relax around the hip joint due to protein hormone Relaxin in preparation for birthing, this in turn causing hypermobility within the hips which can lead