Case Studies on Cardiac Function

You need to explain to him the s/s of blood clotting (since he may have too low an INR d/t treatment and he needs to know this). Explain that his a-fib puts him at risk for blood clots.

12 lead EKG: It is one of the tools for initial evaluation of patients suspected of coronary syndromes such as MI. It as a sensitivity of 80% (Kreatsoulas et al., 2016). ST segment changes of elevation or depression, left bundle branch block, presence of Q waves, new onset of T wave inversion are suggestive of ischemic changes of heart. In this case, then Intervention for cardiac catheterization with stent placement may be required (McConaghy & Oza, 2013).

At April 2015, two labs will pay as settlement $47 million and Singulex will pay $1.5 million. The government also intervened in the lawsuits as to similar allegations against another laboratory, Berkeley HeartLab Inc.; a marketing company, BlueWave Healthcare Consultants Inc., and its owners, Floyd Calhoun Dent and J. Bradley Johnson; and former CEO Latonya Mallory of HDL. Two of the lawsuits against former CEOs are declined the government to intervene in the allegations against Goix and

Mrs. Lee will require blood work. Cardiac enzymes will be drawn, including a troponin and a creatine kinase. All cardiac patients should have biomarkers

The characteristics of pain from myocardial infarction and pericarditis can help to differ both the conditions, and rule out the actual problem of the patient. The characteristics of myocardial infarction include pain duration- 30 minutes to 1 hour; pain intensity and type- severe, crushing, occurs on exertion; and pain does not relieve by the rest or taking nitroglycerine. However, pericarditis, the inflammation of pericardium causes pain that lasts for hours to days; pain intensity and type- mild to severe, asymptomatic, sharp or cutting; pain increases with breathing, swallowing, belching, neck or trunk movement; and relieved by leaning forward, kneeling, sitting upright, or breath holding (Goodman & Snyder, 2013). Therefore, the therapist should ask the patient questions about his or her pain duration, type, intensity, aggravating factors, and relieving factors to rule out whether it is MI or an acute onset of

Choice "D" is not the best answer. The vignette states that the QRS length is less than 120 milliseconds. Ventricular tachycardia consists of wide QRS waves that are greater than 200

Cardiology is the study of the heart and the Cardiovascular system and its functions and disorders.(2)

Other desired outcomes are that ECG results shows the client has a normal sinus rhythm and troponin lab result is less than

Working in a facility where majority of the patients or residents have cardiac related health problems, I perform a shorter version of cardiac and peripheral vascular system assessments in day-to-day basis. Most of my residents have signs and symptoms of cardiovascular disease and daily monitoring is important. I have one resident in particular that I have been monitoring and regularly update the doctor of his status. He has congestive heart failure (CHF), COPD, and other co-morbidities. He has 4+ pitting edema to the lower extremities, elevated heart rate, and crackles to the lungs, which are the presenting symptoms of CHF (Jarvis, 2016). He is alert and oriented x3 with episodes of forgetfulness. I have to constantly reminding him to elevate

Heart failure describes the heart’s inability to function properly, meaning the heart is unable to pump efficiently throughout the body. Thus causing the heart to work extra hard in order to compensate the body’s needs, but this ultimately leads to failure. And due to Mrs. Harris’s hypertension and alcohol consumption, she is now displaying signs and symptoms of congestive heart failure, as both are major risk factors. Heart failure can be seen in the left side, which is also known as congestive heart failure, and the right side of heart. The left side is typically the first side to fail, as the left ventricle is the heart’s largest chamber and the most powerful.

828). Early identification is important and it is most commonly caused by aortic stenosis, genetics and hypertension. According to Kupper and Mitchell, “the four main characteristics of hypertrophic CMP are 1) massive ventricular hypertrophy; 2) rapid, forceful contraction of the left ventricle; 3) impaired relaxation; and 4) obstruction to aortic outflow,” (Kupper & Mitchell, 2014, p. 828). Patients can appear asymptomatic or may present with dyspnea that is caused by an elevated diastolic pressure (Kupper & Mitchell, 2014, p. 828). Fatigue, angina, and syncope may occur due to a decrease in cardiac output, an increase in left ventricular muscle mass, or an increase in obstruction to aortic

The diagnostic value of ausculating the heart mainly depends on the sounds that one hears from the heart and hence may be different. According to Roxette in 1988, the diagnostic value was determined by one though listening to his or her own heart. The type of information one can get when listening to the heart includes what his or her heart sounds like, how often each sound occurs and how loud is the sound of the

Thrombolytic therapy is the use of drugs to break up or dissolve blood clots, which are the main cause of both heart attacks and stroke.

PJ’s symptoms including, a history of angina, mild hypertension which is controlled and chest pain that subsides with rest and or nitroglycerin tablets is consistent with the diagnosis of stable angina with underlying coronary artery disease. According to McCance and Huether (2014), stable angina is a condition that is the result of myocardial ischemia or the lack of oxygen needed related to the hardening of the arteries and their walls making them ineffectively dilate to increase the needed blood flow and oxygen to the myocardium for exertional efforts. The decrease in adequate blood flow and delivery of oxygen in turn creates an anaerobic metabolism by the cells

One of the main fibrinolysis enzymes is plasmin, this is an enzyme that comes from plasminogen, the conversion of plasminogen to plasmin will incorporate 2 serine proteases. These serine proteases are tPA and uPA. The enzyme tPA, is made and released from the endothelial cells, while uPA is made by monocytes and macrophages. Two enzymes also differ in the fact that uPA has a lower affinity for plasminogen compared to tPA. This process will stop and inhibit the buildup of fibrin, it also allows the thrombus to be removed. The activation of plasminogen will form plasmin and this will degrade the thrombus. Plasmin will terminate the clotting process. During this process, fibrin degradation products (FDPs) will begin to form. These FDPs will include fibrinopeptide B and other fibrin degradation dimers. These products will be released and will degrade the fibrin18. This process will remove the