End-Tidal CO2 In Operating Room Environment Analysis

-Use of indiscriminate sedatives, benzos, and opioid’s, especially in post-op patient who retains Co2, may suppress the ventilator drive and lead to respiratory failure

Shock is a life-threatening condition with a variety of underlying causes. It is critical that the nurse apply the nursing process as the guide for care. Shock is unpredictable and rapidly changing so the nurse must understand the underlying mechanisms of shock. The nurse must also be able to recognize the subtle as well as more obvious signs and then provide rapid assessment and response to provide the patient with the best chance for recovery. Coping skills are important, but not the ultimate priority. Keeping the physician updated with the most accurate information is important, but the nurse is in the best position to provide rapid assessment and response, which gives the patient the best chance for survival. Monitoring for significant

Both rapid, shallow breathing patterns and hypoventilation effect gas exchange. Arterial blood gases will be monitored and changes discussed with provider. Alteration in PaCO2 and PaO2 levels are signs of respiratory failure. Patient’s body position will be properly aligned for optimum respiratory excursion, this promotes lung expansion and improved air exchange. Patient will be suctioned as needed to clear secretions and maintain patent airways. The expected outcome is that the patient’s airway and gas exchange will be maintained as evidence by normal arterial blood gases (Herdman,

As Jane was presenting with a symptom of a life threatening event it was important that treatment was immediate. Priority was initially made from assessment of the airways, breathing and circulation, level of consciousness and pain. Jane’s respirations on admission were recorded at a rate of 28 breaths per minute, she looked cyanosed. Jane’s other clinical observations recorded a heart rate of 105 beats per minute (sinus tachycardia), blood pressure (BP) of 140/85 and oxygen saturation (SPO2) on room air 87%. It is important to establish a base line so that the nurse is altered to sudden deterioration in the patient’s clinical condition. Jane’s PEWS score (Physiological Early Warning Score) was 4 and indicated a need for urgent medical attention (BTS 2006). Breathing was the most obvious issue and was the immediate priority.

18 study data were derived from the apnea database (PhysioNet) corresponding to MIT/BIH database. AirFlow, SaO2, Abdominal and Thoracic Movements were obtained from full PSG records. Each record includes a period of nearly eight hours and signals annotation is done minute-by-minute. A minute is labeled as an apnea case if it contains at least one condition of apnea or hypopnea, otherwise labeled as a non-apnea. The patients mean age and weight are between 27 to 63 years (4810.8) and 53 to 135 kg (86.322.2), respectively. PhysioNet data are classified in two categories: Apnea Group (Class A, more than 100 minutes of apnea, mean age: 50 years in range of 29 to 63 years) and Normal Group (class C, less than 5 minutes of apnea, mean age of 33 years

It is a beneficial treatment that should be implemented as early in patient care as possible, such as, within the EMS system. Through the last century this therapy has been accepted and rejected by many medical professionals. Since medicine is an ever-changing field, future research and practice of hypothermia will dictate if this therapy is more beneficial than harmful, and maybe one day could be a permanent major role, or it may never be used again. Hypothermia has been proved to decrease neurological impairment after cardiac arrest, but also has many limitations that can occur. A major limitation of this therapy is, if continued care cannot be guaranteed by receiving hospitals, therapeutic hypothermia is irrelevant for EMS to initiate. Likewise, if hypothermia is not begun in the field by EMS, then the receiving facilities now will have a delayed time in starting the therapy and anoxic brain injury could have already occurred. EMS agencies can drive the implementation of therapeutic hypothermia in the medical field. This therapy allows EMS providers to have a major role in the outcome of a cardiac arrest patient’s recovery and neurological outcome. With the progression of research and practice, medicine is evolving day after day, and patient mortality and morbidity have decreased over the years. Although, cardiac arrest patients have a poor

Additionally, the care environment developed a hazard when the patient population increased both in number and acuity with the admission of the acute respiratory distress patient and increasing patient load in the lobby without note of available back up staff being called in. Examples of errors from the flow chart comparison might include failure to assess and monitor when Nurse J initiates blood pressure and SpO2 measurements, fails to initiate ECG with respiration monitoring, fails to administer supplemental O2, and leaves the room without apparently noting the baseline of the patient2. Furthermore, there appears to be an error in the lack of communication collaboration between the RN and LPN regarding Mr. B’s post procedure status and monitoring needs, and there is a failure to rescue when the LPN notes the low SpO2 value, fails to respond, and instead re-initiates another blood pressure reading without noting the results. As Mr. B’s condition deteriorates and a code is called, an ACLS error is observed in the timeline when the patient is noted first to have absent pulse and respirations and that a monitor is next applied and the patient and displays ventricular fibrillation. Chest compressions appear to not have been the first action in this scenario, nor is end tidal CO2 monitoring noted as initiated to monitor the quality of compressions. These are examples of hazards and errors in the care of Mr. B and in an actual RCA the level of detail would likely turn up

interrupted and so the number of rotations can be measured. The frequency of rotations depends on the flow of air so a flow signal can be generated.

Nitric oxide is a molecule that our body produces that helps cells communicate with each other. Nitric oxide has previously been used for respiratory failure in term and preterm babies. Nitric oxide is commonly known as an environmental pollutant because it is a component of the smog we breath during bad air days. Nitric oxide is naturally produced in the body in the upper and lower airway but can also be present in cigarette smoke. This paper will be looking at the effects and use of inhaled nitric oxide in acute respiratory distress syndrome (ARDS). ARDS can be triggered by other things such as pneumonia, a serious injury to the chest, inhaling vomit, and many other things. Having ARDS means there is inflammation throughout the lungs

Spirometry is the most popular lung function test. The patient performs a maximal inhalation and then forcefully exhales as quickly and as long as they are able. The spirometer measures the volume of the air exhaled by patients. These measurements are taken at two intervals. The first measurement is the forced expiratory volume in one second (FEV1), records the volume of air exhaled after one second. The second measurement is taken at the point where the patient has fully exhaled the volume of inhaled air; this measurement is the forced vital capacity (FVC) (Harpreet Ranu et al.,

Shock is a common condition in critical care, affecting about one third of patients in the intensive care unit(ICU).

The American Heart Association (AHA) updated the 2005 cardiopulmonary resuscitation clinical practice guidelines after the New England Journal of Medicine published two landmark studies in 2002. The HACA and the Bernard, et al. study found significant improvement in neurological outcomes with therapeutic hypothermia. Additionally, the Bernard, et al. study also revealed reduced mortality after cardiac-arrest survivors received therapeutic hypothermia (2002). In 2010, the AHA strengthened its position based on the growing body of research. Therapeutic hypothermia was the only intervention shown to improve neurological outcomes (Peberdy, et al., 2010). The most updated guidelines, set by the AHA in 2015, recommended that all comatose,

The baseline value of this experiment was the PO2, PCO2 and Hb-O2 saturation during normal breathing. These baseline value (table 1) allow the comparison between different breathing patterns to normal breathing. The results (Figure 1, 2, 3 & table 1) of this experiment shows that breath-hold compare to normally breathing, mean PO2 decrease, mean PCO2 increase (figure 1) and mean Hb-O2 saturation slightly decrease (Figure 2). Moreover, comparing p values of breath-hold to breath-hold after hyperventilation, which is less than 0.05, meaning there is significant changes in mean PO2 and PCO2 (table 2), mean PO2 decrease, mean PCO2 increase (figure 1), and mean oxygen haemoglobin saturation increase (figure 2), the mean duration of breath hold decrease

Lung function test is essential to monitor client’s lung function, however, since a peak flow meter or a spirometer may not be affordable for the client, the method for making a self-made spirometer could be demonstrated so as to allow the client perform lung function test, and monitor the lung function daily. And in order to facilitate a better understanding, a video will be given to the client to revise.

Assessment of hemodynamic status in a shock state remains a challenging issue in Emergency Medicine. Early recognition and appropriate treatment of shock have been shown to decrease mortality (Moore and Copel, 2011; Volpicelli, 2011). In case of medical emergencies there may not be enough time to perform detailed diagnostic procedures and eliminating any imminently life-threatening conditions from the possibilities is always challenging in a busy ED.