The Pathophysiology Of Bacterial Meningitis

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1. The pathophysiology of bacterial meningitis is an infection of the bloodstream from an infected area or by commonly acquired through the respiratory system. Next, the bacteria gains access to the subarachnoid space. The body next responds with increased neutrophils to the subarachnoid space. Furthermore, “the release of cytotoxic inflammatory agents and bacterial toxins alter the blood-brain barrier and damage brain tissue (Huether & McCance, 2012)”. As a result, the meningeal vessels become engorged and gradually become permeable. “The cerebral spinal fluid is thicken by inflammatory exudate and inhibits normal cerebral spinal fluid circulation around the brain and spinal cord (Huether & McCance, 2012)”. Furthermore, arachnoid villi may become congested and produce an effect of the enlargement of the skull and compression of the brain. Increased intracranial pressure is experienced from the combination of thicken exudate and swollen meningeal cells. Engorged blood vessels and thrombi can disrupt blood flow, causing further injury. 2. Neisseria meningitides is a form of bacteria that infects the meninges of the brain, in which, affects the brain membrane. The meninges become edematous and affects cerebral spinal fluid. As a result, increased cerebral pressure is exhibited. This form of bacteria is “transmitted from person-to-person through droplets of respiratory or throat secretions from carriers (WHO, 2015)”. Examples of transmission is exhibited by close and prolonged
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