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- You are a studying the genetic basis of horn color in unicorns. Gene C determines the color of pigment destined for the horn by converting a pink precursor into a purple pigment. C- individuals make purple pigment whereas cc individuals accumulate pink precursor. Gene D ensures the deposition of the pigment into the horn cells, such that D- individuals have a colored horn (purple or pink), whereas dd individuals have a white horn. You breed two purple horned unicorns to each other (CcDd). Assuming Gene C and Gene D are unlinked, what is the probability that the baby unicorn has a purple horn? Please express your answer to 2 decimal places (e.g. 0.69).1. There are two different variations at STR locus THO1. Where did these variations come from? b) The STR locus D21S11 only has one variation. Why?2)You would expect liver cells and muscle cells to have the same gene expression. a)True b)False
- 1.Which of the following events is NOT involved in anteroposterior axis determination?* a. Dickkopf inhibits Xwnt8 at the posterior regionption 1 b. Cerberus inhibits BMP4 at the posterior region. c. Frizzled-related protein inhibits Xwnt8 at the anterior region d. Cerberus inhibits nodal-related protein at the anterior region 2. Which of the following statements BEST explains the dorsalizing function of the gurken gene?* a. The gurken ligands bind to torpedo receptors of follicles b. The gurken proteins inhibit pipe genes in the dorsal follicle cells. c. The gurken gene is activated at the dorsally located nucleus of the egg. d. The gurken mRNAs are synthesized at the dorsal portion of the egg celThe ABO blood groups in humans are expressed as the IAlB, and IAalleles. The allele encodes the A blood group antigen, lBencodes B, and i encodes O. Both A and B are dominant to O. If a heterozygous blood type A parent (IAi) and a heterozygous blood type B parent (IBi) mate, one quarter of their offspring will have AB blood type (IAIB) in which both antigens are expressed equally. Therefore, ABO blood groups are an example of: multiple alleles and incomplete dominance codominance and incomplete dominance incomplete dominance only multiple alleles and codominanceThe ability of humans to taste the bitter chemical phenylthiocarbamide (PTC) is a genetic trait. People with at least one copy of the normal, dominant allele of the PTC gene can taste PTC; those who are homozygous for a mutant, recessive allele cannot taste it. Could two parents able to taste PTC have a nontaster child? Could nontaster parents have a child able to taste PTC? A pair of taster parents, both of whom had one parent able to taste PTC and one nontaster parent, are expecting their first child. What is the chance that the child will be able to taste PTC? Unable to taste PTC? Suppose the first child is a nontaster. What is the chance that their second child will also be unable to taste PTC?
- The photos above show flowers from Arabidopsis plants. One plant is wild-type (unmutated); the other carries a mutation in one of its ABC floral identity genes. This mutation causes sepals and petals to form instead of stamens and carpels. Refer to Figure 10.8 to decide which gene (A, B, or C) has been inactivated by the mutation.3.Tay Sachs is a lysosomal storage disease that is inherited in an autosomal recessive manner (T= dominant allele; t = recessive allele). Individuals with Tay Sachs have a defect in the gene encoding hexosaminidase A, a lysosomal enzyme that breaks down cell membrane components called gangliosides. The inability to break down gangliosides causes harmful quantities of this lipid to accumulate in nerve cells of the brain, eventually leading to premature death of affected cells. There is no known cure for the disease, and death usually occurs by age 4. Jack and Jill have a child who has Tay-Sachs. What do you know about the genotypes of Jack and Jill? A.has the genotype DD B.has the genotype Dd C.is still of unknown genotype since there were offspring of both deaf and hearing phenotypes. D.has the genotype dd4. Marfan syndrome is a genetic trait caused by in a dominant allele. The trait causes a weakening of the aorta that can be fatal. A teenager whose mother has the syndrome (but whose maternal grandfather was not affected), and whose father was unaffected, is concerend that she may have the trait. a. What is the phenotype of each of her parents? Genotype? b. What is the chance that the teenager has Marfan syndrome?
- 4. Assuming the translation product is an enzyme, explain its role in the final expression of a phenotype.3. Albinism in humans is controlled by a recessive gene (c). From marriages between two normally pigmented carriers (Cc), what is the probability of having a normal child? An albino? Assume that from such a marriage (Cc x Cc) four children will be produced. (@ What is the probability that all four will be normal? (b) Three will be normal and one albino? (c) Two normal and two albino?1. The location of hemophilia C- factor XI is 4q35, where exactly is it? Describe.2. Referring to image of chromosome 16 provided, indicate the exact location of the geneslabeled a-k: