A recently emerged virus has been transmitting and mutating rapidly in humans throughout the world. If a vaccine that stops transmission of all current circulating variants of the virus in humans is utilized and herd immunity is achieved throughout the world, would this essentially stop 1) further transmission of the virus; 2) further mutation of the virus, respectively? Why/why not
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This is a hypothetical scenario.
A recently emerged virus has been transmitting and mutating rapidly in humans throughout the world. If a vaccine that stops transmission of all current circulating variants of the virus in humans is utilized and herd immunity is achieved throughout the world, would this essentially stop 1) further transmission of the virus; 2) further mutation of the virus, respectively? Why/why not? Please answer the question succinctly and in bullet points!
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- According to the article which was written before the COVID-19 vaccine was created, when do people turn to magic? How can this tendency, which happens across cultures and throughout history, contribute to the current spread of the novel coronavirus and its variants?Efforts to produce an HIV vaccine have met with limited success. What aspects of the virus and its replicative strategy make it difficult to produce a vaccine against HIV? What other kind of virus might be similarly different to vaccinate against? What similarities and differences exist between the two types of virus that account for the differences in vaccine production?What is the Coronavirus? Is it a living organism? How does it reproduce? The current vaccines that are being distributed in the United States are made by Pfizer, Moderna, and Johnson and Johnson. Explain how they work and how health officials believe they will aid in the pandemic? What should we expect as more and more people get vaccinated? Will things immediately go back to the way they were before? What is the Delta variant and how is it impeding on getting things back to "normal"?
- Which of the following is the most likely explanation for an individual who lacks CCR5 as a result of a homozygous defect in the CCR5 gene becoming infected with HIV? a. The mutated CCR5 genes reverted to the normal form, rendering macrophages susceptible to macrophagetropic HIV variants. b. The macrophage-tropic HIV variant entered host cells using CD4 alone. c. The viral nucleic acid alone was taken up by cells, as in cell transformation by bacterial DNA. d. The individual had received a transplant of HIV-infected cells expressing normal CCR5. e. The primary infection involved a lymphocyte-tropic strain of HIV that used CXCR4 as its co-receptor.Despite our advancement in Science and Technology, thanks to the invention of the early scientists Robert Hooke and Antonie Philips van Leeuwenhoek that paved the way to the discovery of cells and the cure of many diseases, why is it that there are still many who are hesitant to have themselves immunized by COVID-19 vaccines? Please answer with 500 wordsLet’s say there are two closely related viruses, let’s call them Guernsey virus and Micro virus. These two viruses only infect epithelial cells. Their surface antigens are sufficiently similar that there are a number of shared epitopes between the two viruses. Choose one of the following: A) If you get infected and successfully recover from one virus, it’s quite possible that you’ll have neutralizing antibodies against the other. B) If you get infected and successfully recover from one virus, you’ll still be susceptible to the other one because the antigens aren’t exactly the same. C) If you get infected and successfully recover from one virus, it’s likely that getting infected with the other virus will be worse because of the common phenomena of antibody-dependent enhancement. D) Not enough information has been given.
- Even though the oral polio vaccine is not used in the developed world, it is still widely used in the developing world, in part because it confers what might be called “accidental” herd immunity. Can you speculate on what this is?It used to be that our only method of creating vaccines was to use dead or weakened pathogens. That is no longer the case - what are some newer options that are available to researchers?Would it be effective to use an inhibitor of RNA polymerase to block the HIV replication cycle and subsequent infection of human T cells? Yes, this would stop the transcription of viral genes and prevent HIV from being able to replicate. No, these types of inhibitors do not exist and would be impossible to make. No, this would block transcription of T cell genes, leading to the death of T cells and compromised immune systems in individuals. Yes, inhibitors of RNA polymerase are easy and relatively inexpensive to produce and make good therapeutics.
- Vaccines have effectively prevented many viral diseases. Attempts over many years to develop an effective vaccine against HIV disease and AIDS, however, have so far met with little success. Why is this so?Two newly developed vaccine candidates (A and B) are tested in mice for their ability to elicit high concentrations of anti-meningococcal antibodies that would provide mucosal as well as bloodstream protection. Also, the ideal candidate vaccine should also provide long-lasting immunity to the infection. The figure below shows the responses to a primary, followed by a secondary immunization to each of the two candidate vaccines. a) Which candidate vaccine elicits the preferred response? What are the three aspects of the preferred response that make it the candidate vaccine of choice? b) What is the likely composition of each vaccine and what evidence from the information above are used to lead to your conclusions? c) To confirm the choice of the preferred candidate vaccine, what type of additional information from the vaccine trials in mice shown above would support this conclusion? Name two additional features of the secondary antibody response to each candidate vaccine that could be…What part of the SARS-CoV-2 virus is a potential target site for a vaccine? Explain your reasoning; include in your discussion the following terms: antigen, epitope, and antibody.