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Compare and contrast viral proteins with anti-immune system properties and their targets of SARS-COV-2 and Influenza Type A Virus.
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Solved in 2 steps
- Compare and contrast the receptor-binding proteins of the envelope and the receptor used by the virus to enter cells of SARS-COV-2 and Influenza Type A Virus.Compare and contrast SARS COV-2 and. Influenza Type A Virus with respect to the following parameters: Type of genetic material. Receptor binding proteins of the envelope and receptor used by the virus to enter cells. Effects of virus on interferon induction and innate Inflammatory response.Compare and contrast the effects of virus on interferon induction and innate Inflammatory response of SARS-COV-2 and Influenza Type A Virus.
- Compare and contrast the genetic material of SARS-COV-2 and Influenza Type A Virus.If antibody responses are not elicited by a Covid-19 vaccine, are there other types of immune response that could provide protection from the SARS-Co-V2 virus following vaccination? Explain why or why not.Explain the multifaceted role of interferons as first responders to viral infection: Identify important target ISGs. Discuss the general mechanisms by which interferon induced genes block virus replication.
- Compare and contrast host mechanisms that restrict virus infection by specifically interacting with viral gene products of SARS-COV-2 and Influenza Type A Virus.Explain : Vpr counteracts LAPTM5 to promote HIV-1 infection in macrophagesBriefly list three mechanisms by which monoclonal antibodies that bind to viruses or virally infected cells, such as anti-Spike protein antibodies, cause reduction of viral infection and spread
- please help explain would SARS-CoV-2 N-nucleocasid protein be good target for neutralizing antibodies and vaccine development ?and whyGiven what we know about HIV, describe the impact of this virus on humoral and cellular immunity. [hint - HIV targets CD4 cells; how will this impact an immune response]“Type I interferons serve as the first line of defence against viral infections”. Give a schematic representation of their mechanism of action and also explain how they interfere with viral replication.