During prolonged starvation or in uncontrolled diabetes mellitus, the formation of acetyl-CoA exceeds oxaloacetate supply, forming ketone bodies Hypoglycaemia may be observed in Cushing syndrome patients The range of albuminuria that is considered as a reversible and treatable is 30 – 300 mg/24hr (20 – 200 μg/min)

During prolonged starvation or in uncontrolled diabetes mellitus, the formation of acetyl-CoA exceeds oxaloacetate supply, forming ketone bodies Hypoglycaemia may be observed in Cushing syndrome patients The range of albuminuria that is considered as a reversible and treatable is 30 – 300 mg/24hr (20 – 200 μg/min)

Ebk:Nutrition & Diet Therapy

10th Edition

ISBN:9780357391747

Author:DEBRUYNE

Publisher:DEBRUYNE

Chapter21: Carbohydrate- Controlled Diets For Diabetes Mellitus

Section: Chapter Questions

Problem 6SC

See similar textbooks

Similar questions

The presence of compounds in urine, depending on the level or range, are indications of underlying disorders. Using glucose as an example, what does the presence of this compound in the urine indicate? And after seeing this compound consecutively in the result, what would be the best course of action to take?
A newborn develops severe vomiting and symptoms of metabolic acidosis. Urinalysis results are positive for ketones and negative for glucose and other reducing substances. If the urine had an odor of “sweaty feet,” what metabolic disorder would be suspected? If the newborn were producing dark brown urine with a sweet odor, what disorder would be suspected? Would an MS/MS screen be helpful for the diagnosis?
Match the bold specific genetic metabolic disorders to their category: adrenal hyperplasia Albinism Cystinuria Galactosaemia Phenylketonuria Pompe Wilson Disease Chromosomal abnormality Excessive amounts of metabolites in a metabolic pathway Storage disease Disrupted regulatory system Disrupted Vitamin and/or co-factor absorption or processing Transport disorder Accumulation of substrates in a metabolic pathway Absence of end product
Indicate what will happen ( increase, decrease or no effect) a. release of glucagon in the blood to the activity of carnitine acyl transferase 1 b. high malonyl CoA to the activity of carnitine acyl transferase I C. Epinephrine to the activity og glycogen synthase d. high citrate to the activity of acetyl CoA carboxylase e. high acetyl CoA to ketogenesis
Discuss the 3 mechanisms of abnormal carbohydrates metabolism that result in the development of ketone bodies in the urine and give examples of the condition to each.
Which clinical manifestations are associated with metabolic syndrome? give specific rational A.Polyuria, polydipsia, polyphagia B.High urea plasma levels and hypotension C.Large abdominal girth, hypertriglyceridemia, insulin resistance D.Fever and acidosis
In people with diabetes mellitus Type 1, explain the reason for:(a) ketoacidosis, and (b) ketonuria.
Which of the following is a clinical manifestation consistent with diabetic ketoacidosis? Question 63 options: a) Glucose < 14 mmol/L b) pH > 7.51 c) Bicarbonate > 30 mmol/L d) Acetone breath
Define the following terms:a. Cori cycleb. glucagonc. glucose-alanine cycled. hypoglycemiae. antioxidant
List all the inborn errors of Carbohydrate metabolism. Explain their pathophysiologies
Explain how excessive ketone bodies may form in the following: During starvation In patients with diabetes mellitus
How does the pathophysiology of diabetes ketoacidosis differ from hyperosmolar non-ketonic coma? (easy and simple)