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Evidence Question #5 1. Cholesterol is abundant in the plasma membrane. Would you expect cholesterol to flip between the two leaflets of the membrane faster than phospholipids, or slower, or at the same rate? Explain. 3. Many neurons contain "delayed" voltage-gated K+ channels. Compared to voltage- gated Na+ channels, the voltage-gated K+ channels respond more slowly to changes in membrane potential. When the voltage-gated K+ channels are open, they make the plasma membrane even more permeable to K+. a) Why does neuronal function require the voltage-gated K+ channels to open more slowly than the voltage-gated Na+ channels? b) How would you expect the voltage-gated K+ channels to affect the shape of the action potential? 3. Mitochondrial DNA encodes key proteins of the electron transport system in the mitochondrial inner membrane. Certain yeast strains carry mitochondrial DNA mutations that selectively inactivate the electron transport system, but these mutant cells are viable when grown in the presence of glucose. a) How do yeast cells lacking the electron transport system make ATP? b) Yeast cells lacking the electron transport system still have a weak membrane potential across the mitochondrial inner membrane. How do these cells generate a membrane potential?

Evidence Question #5 1. Cholesterol is abundant in the plasma membrane. Would you expect cholesterol to flip between the two leaflets of the membrane faster than phospholipids, or slower, or at the same rate? Explain. 3. Many neurons contain "delayed" voltage-gated K+ channels. Compared to voltage- gated Na+ channels, the voltage-gated K+ channels respond more slowly to changes in membrane potential. When the voltage-gated K+ channels are open, they make the plasma membrane even more permeable to K+. a) Why does neuronal function require the voltage-gated K+ channels to open more slowly than the voltage-gated Na+ channels? b) How would you expect the voltage-gated K+ channels to affect the shape of the action potential? 3. Mitochondrial DNA encodes key proteins of the electron transport system in the mitochondrial inner membrane. Certain yeast strains carry mitochondrial DNA mutations that selectively inactivate the electron transport system, but these mutant cells are viable when grown in the presence of glucose. a) How do yeast cells lacking the electron transport system make ATP? b) Yeast cells lacking the electron transport system still have a weak membrane potential across the mitochondrial inner membrane. How do these cells generate a membrane potential?

Biochemistry
Biochemistry
6th Edition
ISBN:9781305577206
Author:Reginald H. Garrett, Charles M. Grisham
Publisher:Reginald H. Garrett, Charles M. Grisham
Chapter32: The Reception And Transmission Of Extracellular Information
Section: Chapter Questions
Problem 20P
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Evidence Question #5 1. Cholesterol is abundant in the plasma membrane. Would you expect cholesterol to flip between the two leaflets of the membrane faster than phospholipids, or slower, or at the same rate? Explain. 3. Many neurons contain "delayed" voltage-gated K+ channels. Compared to voltage- gated Na+ channels, the voltage-gated K+ channels respond more slowly to changes in membrane potential. When the voltage-gated K+ channels are open, they make the plasma membrane even more permeable to K+. a) Why does neuronal function require the voltage-gated K+ channels to open more slowly than the voltage-gated Na+ channels? b) How would you expect the voltage-gated K+ channels to affect the shape of the action potential? 3. Mitochondrial DNA encodes key proteins of the electron transport system in the mitochondrial inner membrane. Certain yeast strains carry mitochondrial DNA mutations that selectively inactivate the electron transport system, but these mutant cells are viable when grown in the presence of glucose. a) How do yeast cells lacking the electron transport system make ATP? b) Yeast cells lacking the electron transport system still have a weak membrane potential across the mitochondrial inner membrane. How do these cells generate a membrane potential?
Transcribed Image Text:Evidence Question #5 1. Cholesterol is abundant in the plasma membrane. Would you expect cholesterol to flip between the two leaflets of the membrane faster than phospholipids, or slower, or at the same rate? Explain. 3. Many neurons contain "delayed" voltage-gated K+ channels. Compared to voltage- gated Na+ channels, the voltage-gated K+ channels respond more slowly to changes in membrane potential. When the voltage-gated K+ channels are open, they make the plasma membrane even more permeable to K+. a) Why does neuronal function require the voltage-gated K+ channels to open more slowly than the voltage-gated Na+ channels? b) How would you expect the voltage-gated K+ channels to affect the shape of the action potential? 3. Mitochondrial DNA encodes key proteins of the electron transport system in the mitochondrial inner membrane. Certain yeast strains carry mitochondrial DNA mutations that selectively inactivate the electron transport system, but these mutant cells are viable when grown in the presence of glucose. a) How do yeast cells lacking the electron transport system make ATP? b) Yeast cells lacking the electron transport system still have a weak membrane potential across the mitochondrial inner membrane. How do these cells generate a membrane potential?
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