Following administration of an opiate drug, "pain signals" are prevented from getting out of the spinal cord via two mechanisms. One inhibits spinal cord neurons that communicate with the parafascicular nucleus of the thalamus. What is the other way that pain signals are stopped? dendro-dendritic synapses that enhance IPSI's in somatosensory neurons. the disinhibition of long axons that start in the spinal cord and influence free nerve endings directly. feedback loop to the periaquedictal grey of the midbrain, which enhances neural activity in that area. axo-axonic synapses that prevents somatosensory neurons from releasing neurotransmitter.

Human Physiology: From Cells to Systems (MindTap Course List)
9th Edition
ISBN:9781285866932
Author:Lauralee Sherwood
Publisher:Lauralee Sherwood
Chapter6: The Peripheral Nervous System: Afferent Division; Special Senses
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Following administration of an opiate drug, "pain signals" are prevented from getting out of the
spinal cord via two mechanisms. One inhibits spinal cord neurons that communicate with the
parafascicular nucleus of the thalamus. What is the other way that pain signals are stopped?
dendro-dendritic synapses that enhance IPSI's in somatosensory neurons.
the disinhibition of long axons that start in the spinal cord and influence free nerve endings directly.
O a feedback loop to the periaquedictal grey of the midbrain, which enhances neural activity in that area.
axo-axonic synapses that prevents somatosensory neurons from releasing neurotransmitter.
Transcribed Image Text:Following administration of an opiate drug, "pain signals" are prevented from getting out of the spinal cord via two mechanisms. One inhibits spinal cord neurons that communicate with the parafascicular nucleus of the thalamus. What is the other way that pain signals are stopped? dendro-dendritic synapses that enhance IPSI's in somatosensory neurons. the disinhibition of long axons that start in the spinal cord and influence free nerve endings directly. O a feedback loop to the periaquedictal grey of the midbrain, which enhances neural activity in that area. axo-axonic synapses that prevents somatosensory neurons from releasing neurotransmitter.
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