Open with -Mutation: Incomplete Wing VeinsP generation Phenotypes:Normalfemale XIncompletemaleFi generationPhenotvoeFemalesMalesTotalRatioNormal0.Incomplete8.8.F, XF, Phenotypes:IncompleteIncompletefemale XmaleF2 generationPhenotvoeFemalesMalesTotalRatioNormalIncompleteF. Punnett squareFEPunnett squarePars399FOMCO

Name: Open with -Mutation: Incomplete Wing VeinsP generation Phenotypes:Nor
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Description: Open with -Mutation: Incomplete Wing VeinsP generation Phenotypes:Normalfemale XIncompletemaleFi generationPhenotvoeFemalesMalesTotalRatioNormal0.Incomplete8.8.F, XF, Phenotypes:IncompleteIncompletefemale XmaleF2 generationPhenotvoeFemalesMalesTotal

The alleles are the alternative forms of a gene that are located on the same locus of a homologous…

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Open with Mutation: Incomplete Wing Veins P generation Phenotypes: Normal female X Incomplete male Fi generation Phenotvoe Females Males Total Ratio Normal 0. Incomplete 8. F, XF, Phenotypes: Incomplete Incomplete temale X male F2 generation Phenotvoe Females Males Total Ratio Normal Incomplete FPunnett square F. Punnett square Peps 399F

Open with Mutation: Incomplete Wing Veins P generation Phenotypes: Normal female X Incomplete male Fi generation Phenotvoe Females Males Total Ratio Normal 0. Incomplete 8. F, XF, Phenotypes: Incomplete Incomplete temale X male F2 generation Phenotvoe Females Males Total Ratio Normal Incomplete FPunnett square F. Punnett square Peps 399F

Biology: The Unity and Diversity of Life (MindTap Course List)

15th Edition

ISBN:9781337408332

Author:Cecie Starr, Ralph Taggart, Christine Evers, Lisa Starr

Publisher:Cecie Starr, Ralph Taggart, Christine Evers, Lisa Starr

Chapter31: Animal Tissues And Organ Systems

Section: Chapter Questions

Problem 1CT: IPSCs are nearly identical to human embryonic stem cells in terms of gene expression, but there may...

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IPSCs are nearly identical to human embryonic stem cells in terms of gene expression, but there may be other ways in which they are not equivalent. For example, the telomeres of IPSCs often vary in length, with many IPSCs cells having telomeres shorter than those of embryonic. How might shortened telomeres affect the life-span of IPSCs or of differentiated cells derived from them?
Enhanced Spatial Learning in Mice With an Autism Mutation Autism is a neurobiological disorder with symptoms that include impaired social interactions and stereotyped patterns of behavior. Around 10 percent of autistic people have an extraordinary skill or talent such as greatly enhanced memory. Mutations in neuroligin 3, an adhesion protein that connects brain cells to one another, have been associated with autism. One mutation changes amino acid 451 from arginine to cysteine. In 2007, Katsuhiko Tabuchi and his colleagues genetically modified mice to carry the same arginine-to-cysteine substitution in their neuroligin 3. Mice with the mutation had impaired social behavior. To test spatial learning ability, the mice were placed in a water maze: a deep pool of warm water in which a platform is submerged a few millimeters below the surface. The platform is not visible to swimming mice. Mice do not particularly enjoy swimming, so they locate a hidden platform as fast as they can. When tested again, they can remember its location by checking visual cues around the edge of the pool. How quickly they remember the platforms location is a measure of spatial learning ability (FIGURE 15.18). FIGURE 15.18 spatial learning ability in mica mutation in neuroligin 3 (R451C), compared with unmodified (wild-type) mica. 1. In the first, how many days did it take unmodified mice to learn to find the location of the hidden platform within 10 seconds?
Need explanation on #4 of my genetic question
12.please match with the type of mutation
In cladograms, sister groups are _______ . a. inbred b. the same age c. represented by nodes d. in the same family
Identify five (5) errors in the figure below. Draw a corrected version below it and encircle all thecorrections that you made.
Enhanced Spatial Learning Ability in Mice Engineered to Carry an Autism Mutation Autism is a neurobiological disorder with symptoms that include impaired social interactions and repetitive, stereotyped patterns of behavior. Around 10 percent of autistic people also have an extraordinary skill or talent such as greatly enhanced memory. Mutations in the gene for neuroligin 3, an adhesion protein that connects brain cells, have been associated with autism. One of these mutations is called R451C because the altered gene encodes a protein with an amino acid substitution: a cysteine (C) instead of an arginine (R) in position 451. In 2007, Katsuhiko Tabuchi and his colleagues introduced the R451C mutation into the neuroligin 3 gene of mice. The researchers discovered that the genetically modified mice had impaired social behavior and superior spatial learning ability. Spatial learning in mice is tested with a water maze, which consists of a small platform submerged a bit below the surface or a pool of water so it is invisible to a swimming mouse. Mice do not particularly enjoy swimming, so they try to locate the hidden platform as quickly as they can. When tested again later, they remember the platforms location by checking visual cues around the edge or the pool. How quickly they remember is a measure of their spatial learning ability. FIGURE 15.14 shows some or Tabuchis result. FIGURE 15.14 Spatial learning ability in mice. Mice with a mutation in neuroligin 3(R451C) were tested for learning performance: as compared with unmodified (wild-type) mice. In the first test, how many days did it take unmodified mice to learn to find the location of a hidden platform in 10 seconds?
need explaination in detail also explain why other options are incorrect too or else I will downvote need sonn
A small fraction—2 to 3%—of all cancers, acrossmany subtypes, displays a quite remarkable phenome-non: tens to hundreds of rearrangements that primarilyinvolve a single chromosome, or chromosomal region.The breakpoints can be tightly clustered, with several in afew kilobases; the junctions of the rearrangements ofteninvolve segments of DNA that were not originally closetogether on the chromosome. The copy number of varioussegments within the rearranged chromosome was foundto be either zero, indicating deletion, or one, indicatingretention.You can imagine two ways in which such multi-ple, localized rearrangements might happen: a progressiverearrangements model with ongoing inversions, deletions,and duplications involving a localized area, or a cata-strophic model in which the chromosome is shattered intofragments that are stitched back together in random orderby nonhomologous end joining (Figure Q20–2).A. Which of the two models in Figure Q20–2 accountsmore readily for the features of…
6. A diploid strain of yeast was made by mating a haploidstrain with a genotype w−, x−, y−, and z− with a haploidstrain of opposite mating type that is wild type for thesefour genes. The diploid strain was phenotypically wildtype. Four different X-ray-induced diploid mutantswith the following phenotypes were produced fromthis diploid yeast strain. Assume a single new mutation is present in each strain.Strain 1 w− x+ y− z+Strain 2 w+ x− y− z−Strain 3 w− x+ y− z−Strain 4 w− x+ y+ z+When these mutant diploid strains of yeast go throughmeiosis, each ascus is found to contain only two viablehaploid spores.a. What kind of mutations were induced by X-rays tomake the listed diploid strains?b. Why did two spores in each ascus die?
Glioblastoma multiforme (GBM) is the most commonand aggressive form of brain cancer in humans.Without any treatment, the mean survival rate is aboutthree months. Even with standard treatments such assurgical resection, radiation, and chemotherapy, themean survival rate is between seven and 14 months.GBM tumors differ in their spectrum of geneticchanges, and these changes may influence the effectof particular treatments. Answer the following questions about the relevance of particular mutations toparticular treatments and outcomes.a. Biopsies of about 20% of GBMs show the expression of a certain mutational variant of the EGFR(epidermal growth factor receptor) protein calledEGFRvIII. The same cancerous cells of theseGBMs also show the expression of normal, wildtype EGFR. Is the gene encoding EGFR a tumorsuppressor gene or a proto-oncogene?b. It is very difficult to induce cells expressingEGFRvIII to undergo apoptosis. If you werea radiologist treating a patient with a GBMthat expresses…
Retinoblastoma: The Hits Just Keep Coming Part IV – Time to Reconvene Question Since Julie is indeed a carrier of the mutated RB1 allele and Chris is homozygous for the wild-type allele, what is the likelihood that their next child will inherit Julie’s RB1 mutation? Part V – A Different Kind of Hit Question Chris does not have an RB1 mutation, and is therefore homozygous wild-type. Julie is heterozygous for the mutation. However, Kay has inherited a different RB1 mutation than the one her mother carries. Therefore, Kay did not receive her mother’s mutant allele. Assuming that Chris really is the father, what other explanation might there be for how she got a germline mutation?