Part C Suppose that you discover cels containing a mutation in a second protein and learn that this mutation also prevents mitotic cyclin from being degraded the onset of mitosia, even when mitotic cyclin is normal. The degradation of other proteins appears to proceed normally in such cells. A mutation in what kind of protein might explain such results? O Amutution in an activating enzyme that specifically binds to a degron present in mitotic cyclin would explain such observations. O A mutation in an ubiquitylated protoin that specifically binds to a degron esent in mitotic cyclin would explain such observations. A mutation in ubiquitin would explain such observations. O Amutation in a recognition protein that specifically binds to a degron present in mitotic cyclin would explain such observations. Submit Reauest Anawet

Part C Suppose that you discover cels containing a mutation in a second protein and learn that this mutation also prevents mitotic cyclin from being degraded the onset of mitosia, even when mitotic cyclin is normal. The degradation of other proteins appears to proceed normally in such cells. A mutation in what kind of protein might explain such results? O Amutution in an activating enzyme that specifically binds to a degron present in mitotic cyclin would explain such observations. O A mutation in an ubiquitylated protoin that specifically binds to a degron esent in mitotic cyclin would explain such observations. A mutation in ubiquitin would explain such observations. O Amutation in a recognition protein that specifically binds to a degron present in mitotic cyclin would explain such observations. Submit Reauest Anawet

Biology: The Dynamic Science (MindTap Course List)

4th Edition

ISBN:9781305389892

Author:Peter J. Russell, Paul E. Hertz, Beverly McMillan

Publisher:Peter J. Russell, Paul E. Hertz, Beverly McMillan

Chapter10: Cell Division And Mitosis

Section: Chapter Questions

Problem 1ITD: Biologists have long been interested in the effects of radiation on cells. In one experiment,...

See similar textbooks

Similar questions

At the checkpoints of the cell cycle, proteinscalled cyclins bind to________________ and activatereactions that enable the cell to proceed tothe next stage of the cycle.
Biologists have long been interested in the effects of radiation on cells. In one experiment, researchers examined the effect of radium on mitosis of chick embryo cells growing in culture. A population of experimental cells was examined under the microscope for the number of cells in telophase (as a measure of mitosis occurring) before, during, and after exposure to radium. The results are shown in the Figure. What is the effect of radium exposure on mitosis? Source: R. G. Canti and M. Donaldson. 1926. The effect of radium on mitosis in vitro. Proceedings of the Royal Society of London, Series B, Containing Papers of a Biological Character 100:413419.
Figure 28.11 depicts the eukaryotic cell cycle. Many cell types â€œexitâ€� the cell cycle and donâ€™t divide for prolonged periods, a state termedG0; some, for example neurons, never divide again. a. In what stage of the cell cycle do you suppose a cell might be when it exits the cell cycle and enters G0? b. The cell cycle is controlled by checkpoints, cyclins, and CDKs. Describe how biochemical events involving cyclins and CDKs might control passage of a dividing cell through the cell cycle.
When the DNA of a human cell becomes damaged, the p53 geneis activated. What is the general function of the p53 protein?Is it an enzyme, transcription factor, cell-cycle protein, orsomething else? Describe three ways in which the synthesisof the p53 protein affects cellular function. Why is it beneficialfor these three things to happen when a cell’s DNA hasbeen damaged?
Progression through the cell cycledepends on the phosphorylationof hundreds of different proteins bycyclin–Cdk complexes. What are themolecular mechanisms ensuring thatthese proteins are phosphorylated atprecisely the right time and place?
What cellular mechanism(s) ensure that passage through the cell cycle is unidirectional and irreversible? What mo-lecular machinery underlies these mechanism(s)?
EVOLUTION CONNECTION The result of mitosis is that thedaughter cells end up with the same number of chromosomesthat the parent cell had. Another potential way to maintain thenumber of chromosomes would be to carry out cell divisionfirst and then duplicate the chromosomes in each daughtercell. Assess whether this would be an equally good way oforganizing the cell cycle. Explain why evolution has not ledto this alternative.
Match the process with the correct phase of the cell cycle (G1, S, G2, M). If the process occurs in M phase indicate subphase (ex. telophase). E) ___________________Extra cell membrane is synthesized by the smooth ER. F) ___________________DNA is compacted into chromatin.
What is the order in which the following events occur during cell division: A. anaphase B. metaphase C. prometaphaseD. telophase E. lunar phaseF. mitosis G. prophaseWhere does cytokinesis fit in?
The cell cycle of an average mammalian cell is illustrated below. How long did it take for the above-illustrated mammalian cell to complete one cell cycle?How long did the mitosis/cytokinesis phase (M phase) last? What % of the total cell-cycle timedoes that constitute to? What phase takes the longest time to complete during cell cycle and what % of the total cellcycle time does that constitute to? Explain why you think this might be the longest phase of thecell cycle.How long was the S phase? What is happening in the cell during the S phase?
Beginning with the G1 phase of the cell cycle, describe the levelof compaction of the eukaryotic chromosome. How does thelevel of compaction change as the cell progresses through thecell cycle? Why is it necessary to further compact the chromatinduring mitosis?
Pleassseeee describe the process of mitosis