Read the clues and classify as Virus, Archaebacteria, Eubacteria, VIRUSES ARCHEABACTERIA EUBACTERIA Have a protein coat Source of antibiotics Are heat lovers Infect and destroy bacterial cells Live in the our small intestine and help with digestion Have a polyhedral shape Produce poisonous chemicals to get us sick. No peptidoglycan in cell wall Causes strepthroat O O о о о 11 points BOTH (EU & ARCHEABACTERIA) O O O O O

Read the clues and classify as Virus, Archaebacteria, Eubacteria, VIRUSES ARCHEABACTERIA EUBACTERIA Have a protein coat Source of antibiotics Are heat lovers Infect and destroy bacterial cells Live in the our small intestine and help with digestion Have a polyhedral shape Produce poisonous chemicals to get us sick. No peptidoglycan in cell wall Causes strepthroat O O о о о 11 points BOTH (EU & ARCHEABACTERIA) O O O O O

Concepts of Biology

1st Edition

ISBN:9781938168116

Author:Samantha Fowler, Rebecca Roush, James Wise

Publisher:Samantha Fowler, Rebecca Roush, James Wise

Chapter17: The Immune System And Disease

Section: Chapter Questions

Problem 4RQ: Which statement is true? a. A virion contains DNA and RNA. b. Viruses are acellular. c. Viruses...

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There have been recurring cases of mad-cow disease in the United Kingdom since the mid-1990s. Mad-cow disease is caused by a prion, an infectious particle that consists only of protein. In 1986, the media began reporting that cows all over England were dying from a mysterious disease. Initially, there was little interest in determining whether humans could be affected. For 10 years, the British government maintained that this unusual disease could not be transmitted to humans. However, in March 1996, the government did an about-face and announced that bovine spongiform encephalopathy (BSE), commonly known as mad-cow disease, can be transmitted to humans, where it is known as variant Creutzfeldt-Jakob disease (VCJD). As in cows, this disease eats away at the nervous system, destroying the brain and essentially turning it into a spongelike structure filled with holes. Victims experience dementia; confusion; loss of speech, sight, and hearing; convulsions; coma; and finally death. Prion diseases are always fatal, and there is no treatment. Precautionary measures taken in Britain to prevent this disease in humans may have begun too late. Many of the victims contracted it over a decade earlier, when the BSE epidemic began, and the incubation period is long (VCJD has an incubation period of 10 to 40 years). A recent study concluded that 1 in 2,000 people in Great Britain carry the abnormally folded protein that causes VCJD. In spite of these numbers, the death rate from VCJD remains low. It is not clear whether this means that the incubation period for the disease is much longer than previously thought, or whether they may never develop the disease. How can a prion replicate itself without genetic material?
la celula animal presenta, excepto A)lisosomas B)cloroplastos C)mitocondrias D)nucleo E)ribosomas
A 4-month-old infant had been running a moderate fever for36 hours, and a nervous mother made a call to her pediatrician.Examination and tests revealed no outward signsof infection or cause of the fever. The anxious mother wanteda prescription for antibiotics, but the pediatrician recommendedwatching the infant for two days before making a decision. Heexplained that decades of rampant use of antibiotics in medicineand agriculture has caused a global surge in antibiotic-resistantbacteria, drastically reducing the effectiveness of antibiotic therapyfor infections. He pointed out that bacteria can exchange antibioticresistance traits and that many pathogenic strains are nowresistant to several antibiotics. The mother was not placatedbythese explanations and insisted that her baby receive antibioticsimmediately. This situation raises several issues. If you were an anxious parent of the patient, would it change your mind if you learned that a woman died in 2016 from a bacterial infection…
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