Some human diseases are caused by mutations in mitochondrialgenes. Which of the following statements is false?a. Mitochondrial diseases may follow a maternal inheritance pattern.b. Mutations associated with mitochondrial diseases often affectcells with a high demand for ATP.c. The symptoms associated with mitochondrial diseases tend toimprove with age.d. Heteroplasmy plays a key role in the severity of mitochondrialdisease symptoms

Some human diseases are caused by mutations in mitochondrialgenes. Which of the following statements is false?a. Mitochondrial diseases may follow a maternal inheritance pattern.b. Mutations associated with mitochondrial diseases often affectcells with a high demand for ATP.c. The symptoms associated with mitochondrial diseases tend toimprove with age.d. Heteroplasmy plays a key role in the severity of mitochondrialdisease symptoms

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Description: Some human diseases are caused by mutations in mitochondrialgenes. Which of the following statements is false?a. Mitochondrial diseases may follow a maternal inheritance pattern.b. Mutations associated with mitochondrial diseases often affectcells wi

Biology: The Dynamic Science (MindTap Course List)

4th Edition

ISBN:9781305389892

Author:Peter J. Russell, Paul E. Hertz, Beverly McMillan

Publisher:Peter J. Russell, Paul E. Hertz, Beverly McMillan

Chapter4: Cells

Section: Chapter Questions

Problem 15TYK

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All mutations in mitochondrial genes ultimately affect(whether directly or indirectly) the key function ofmitochondria, which is to make ATP. Why then domutations in different genes cause different diseases,with specific symptoms? (Note: The answer to thisquestion is not known, but your speculations will helpyou think about the material in this chapter.)29. How could researchers have determined that
All mutations in mitochondrial genes ultimately affect (whether directly or indirectly) the key function of mitochondria, which is to make ATP. Why then do mutations in different genes cause different diseases, with specific symptoms?
Mitochondria contain their own genetic material. Mitochondrialmutations are inherited from mother to child, without anycontribution from the father. Why?
A principle of biology is that structure determines function.Explain how the invaginations of the inner mitochondrialmembrane are related to mitochondrial function.
If you had a protein X, which is a solubleenzyme found inside the peroxisome, and you wished to separateit from a similar protein Y, which is an enzyme found embeddedin the mitochondrial membrane, what would be your initial techniques for isolating those proteins?
In the 1930’s DNP was given to people to lose weight. DNP makes the mitochondrial membrane leaky to hydrogen ions. Why might this cause one to lose weight? Why would this cause death? How is this similar or different to cyanide which binds to complex IV in the mitochondria?
Mitochondria are critical for normal metabolism. From which parent did each human being’s original mitochondria come from at conception? In Luft’s syndrome the mitochondria are not producing sufficient amounts of ATP. What series of reactions could be most responsible for the deficiency? What are some other conditions that are thought to involve mitochondrial malfunction? What's your opinion on the 3-parent babies technique approved in the UK?
You are conducting a study of comparing human, chlamydomonas and Cyanobacteria. Which is a protein found in both chlymydomoans and humans but not in Cyanobacteria. a) mitochondrial receptor protein b) atp synthase c) citrate synthase of citric acid cycle d) ribosome protein
Release of cytochrome C from mitochondrial membrane is stimulated by which of the following (multiple answers)? A. Caspase 9 B. Bcl-W and Mcl-1 C. Bcl-2 and Bcl-XL D. BAX and BAD E. Bcl-2 alone
Consider a gene in the mitochondrial genome that codes for a channel protein in the inner membrane that allows magnesium ions to travel freely across. If a mutation in that gene resulted in this protein allowing H+ to pass through it rather than magnesium, what would be the consequence for cellular respiration?
In the 1930's a chemical known as DNP was touted as en effective diet pill. Weight loss was dramatic in patients taking this drug. However, to quote Efraim Racker (A New Look at Mechanisms in Bioenergetics, Academic Press, 1976, p. 155), ..."the treatment eliminated not only the fat but also the patients,...This discouraged physicians for awhile..." This drug works for weight loss, by causing the leakage of H+ across the inner mitochondrial membrane. Which of the following would you expect to see in patients taking DNP? Select the best answer. Question options: decreased ability to produce ATP increased ability to produce ATP decreased ability to use Krebs cycle to produce ATP slowing down the citric acid cycle.
How is the Inner Mitochondrial Membrane (IMM) different from the Outer Mitochondrial Membrane (OMM) ?