All mutations in mitochondrial genes ultimately affect(whether directly or indirectly) the key function ofmitochondria, which is to make ATP. Why then domutations in different genes cause different diseases,with specific symptoms? (Note: The answer to thisquestion is not known, but your speculations will helpyou think about the material in this chapter.)29. How could researchers have determined that

All mutations in mitochondrial genes ultimately affect(whether directly or indirectly) the key function ofmitochondria, which is to make ATP. Why then domutations in different genes cause different diseases,with specific symptoms? (Note: The answer to thisquestion is not known, but your speculations will helpyou think about the material in this chapter.)29. How could researchers have determined that

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Biology: The Dynamic Science (MindTap Course List)

4th Edition

ISBN:9781305389892

Author:Peter J. Russell, Paul E. Hertz, Beverly McMillan

Publisher:Peter J. Russell, Paul E. Hertz, Beverly McMillan

Chapter4: Cells

Section: Chapter Questions

Problem 15TYK

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Similar questions

All mutations in mitochondrial genes ultimately affect (whether directly or indirectly) the key function of mitochondria, which is to make ATP. Why then do mutations in different genes cause different diseases, with specific symptoms?
Some human diseases are caused by mutations in mitochondrialgenes. Which of the following statements is false?a. Human mitochondrial diseases follow a maternal inheritance pattern.b. Mutations associated with mitochondrial diseases oftenaffect cells with a high demand for ATP.c. The symptoms associated with mitochondrial diseases tendto improve with age.d. Heteroplasmy plays a key role in the severity of mitochondrial disease symptoms
Mitochondria contain their own genetic material. Mitochondrialmutations are inherited from mother to child, without anycontribution from the father. Why?
Some human diseases are caused by mutations in mitochondrialgenes. Which of the following statements is false?a. Mitochondrial diseases may follow a maternal inheritance pattern.b. Mutations associated with mitochondrial diseases often affectcells with a high demand for ATP.c. The symptoms associated with mitochondrial diseases tend toimprove with age.d. Heteroplasmy plays a key role in the severity of mitochondrialdisease symptoms
The DNA located inside of mitochondria exhibits approximately ten times the mutation rate seen in nuclear DNA. Provide an explanation as to why this is the case and what are the effects of this higher mutation rate of mitochondrial DNA on disease processes?
Mitochondria are critical for normal metabolism. From which parent did each human being’s original mitochondria come from at conception? In Luft’s syndrome the mitochondria are not producing sufficient amounts of ATP. What series of reactions could be most responsible for the deficiency? What are some other conditions that are thought to involve mitochondrial malfunction? What's your opinion on the 3-parent babies technique approved in the UK?
A principle of biology is that structure determines function.Explain how the invaginations of the inner mitochondrialmembrane are related to mitochondrial function.
In the mitochondrial electron-transport chain, which complex accepts electrons from FADH2? Complex II. Complex I. Complex V. Complex IV. Complex III.
If you had a protein X, which is a solubleenzyme found inside the peroxisome, and you wished to separateit from a similar protein Y, which is an enzyme found embeddedin the mitochondrial membrane, what would be your initial techniques for isolating those proteins?
In the 1930’s DNP was given to people to lose weight. DNP makes the mitochondrial membrane leaky to hydrogen ions. Why might this cause one to lose weight? Why would this cause death? How is this similar or different to cyanide which binds to complex IV in the mitochondria?
Provide what is being asked 2. Mutations in the genes that encode certain mitochondrial proteins are associated with a high incidence of sometype of cancer. How might defective mitochondria lead to cancer?
How is the function of the thylakoid membrane similar to that of the inner mitochondrial membrane?