Why is that if A, B, and D neurons were all activated, an action potential in neuron C would not occur. But if A and D were activated, an action potential in C would occur?
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Q: ntinuously?
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Q: Explain why the strength of an action potential doesn’t decrease as it travels down an axon.
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Q: What would happen if summation of graded potentials in a neuron did not occur?
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Q: Which are the three different phases of action potential and what characterizes each?
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Q: What is a graded action potential ,when talking about the nervous system, in human anatomy?
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A: Action potential develops only when the membrane acquires a more positive charge inside.
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Q: Why there is a refractory period in the action potential?
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Why is that if A, B, and D neurons were all activated, an action potential in neuron C would not occur. But if A and D were activated, an action potential in C would occur?
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- Which factors contribute to an axon’s conduction velocity? comments: Please understand the question well than answer . Im using bartbly to hence my knowledgeCell bodies of UMN that synapse on LMN lie in all of these areas EXCEPT a) Medulla b) Thalamus c) Pre-motor cortex d) Pons e) Primary motor cortexFor a neuron to fire an action potential, its membranemust reach ________.a. hyperpolarizationb. the threshold of excitationc. the refractory periodd. inhibitory postsynaptic potential
- IPSP differs from EPSP in :-a- being of shorter durationb- being unable o summate spatiallyc- moving the membrane potential away from thresholdd- depending upon opening of voltage K + channelsDescribe how resting potential is form in a neuron. Key difference from in and out a neuron and how they are maintain and proteins involved.When a postsynaptic neuron is adequately stimulated, action potentials develop at the initial segment of the axon because :-a- it contains many voltage-gated Na + channelsb- it contains the highest concentration of ligand-gated cation channelsc- it contains many voltage-gated Ca ++ channelsd- it is more sensitive to the direct stimulant effect of neurotransmitters
- When EPSP and IPSP occur simultaneously the postsynaptic membrane :-a- becomes depolarizedb- becomes hyperpolarizedc- initiates an action potentiald- shows potential changes that depend upon the summation of their effectsA loss-of-function mutation in the ion channel responsible for thegeneration of end-plate potentials results in a diminished permeabilityof the ion channel. 1) How would you experimentally test the effect of the mutation on thefunctional properties of the cell (synapse)? You should discuss the typesof postsynaptic responses and physiological parameters you couldmeasure.The giant axon of the squid (Figure Q11–3) occu-pies a unique position in the history of our understandingof cell membrane potentials and nerve action. When anelectrode is stuck into an intact giant axon, the membranepotential registers –70 mV. When the axon, suspended in abath of seawater, is stimulated to conduct a nerve impulse,the membrane potential changes transiently from –70 mVto +40 mV.
- Contrast the somatic nervous system with the ANS foreach of the following:a. number of neurons between the CNS and the effectororganb. location of neuron cell bodiesc. structures each innervatesd. inhibitory or excitatory effectse. conscious or unconscious controlf. neurotransmitter(s) usedPresynaptic inhibition depends upon :-a- augmented release of chemical transmitter from presynaptic terminalsb- continued depolarization of presynaptic terminalsc- GABA receptors in presynaptic terminalsd- opening of voltage-gated Ca ++ channels in presynaptic terminalsSummarize the steps in generating an action potential as a flowchart. You can make your flowchart on paper and take a picture of it, or make it electronically. Be sure you’ve included: the location in the neuron and components of the neuron involved, the types of cellular transport and ions involved, how action potentials can be stimulated and inhibited. you can get the information from this: https://youtu.be/HYLyhXRp298