Clindamycin

types of C. botulinum because it can advance the release of the toxins (Mayo Clinic Staff, 2015). There are three different antibiotics that can be used on wound C. botulinum patients, Penicillin G (Pfizerpen), Chloramphenicol (Chloromycetin), and Clindamycin (Cleocin). Penicillin G is the preferred drug for wound C. botulinum the other two are an alternative to penicillin (Chan-Tack, 2015). “Penicillin G interferes with synthesis of cell wall mucopeptide during active multiplication, resulting in bactericidal

Presenting Symptoms: Johnny is a 5 year old Asian boy presented with runny nose that started a week ago but has not resolved. He also developed sores around the nose that started as a blister and ruptured. A scab formed with crust that looks like dried maple syrup but continues seep and drain. He also has a lesion on his forearm and also been febrile. Physical examination reveals moderate purulent rhinorrhea and 0.5- to 1-cm diameter weeping lesions around the nose and mouth and on the radial surface

to the emergency room on February 21, 2011, with ongoing chest pain, shortness of breath, and a productive cough. He was evaluated at that time the Northeast Methodist’s emergency room and was admitted. His antibiotic therapy was changed to IV Clindamycin and Azithromycin and he was referred to pulmonary specialist, Alison Brown, MD for consultation on February 24, 2011. Following consultation, blood studies were ordered which showed an ongoing elevated white cell count. Blood cultures showed

enlargement of the right tonsil without abscess (pocket of infection). You received intravenous treatment (through the vein) fluid bolus (fast) in the amount of 2 liters, Morphine (pain medicine) for pain, Decadron (medicine that decreases swelling), Clindamycin (antibiotic) and Unasyn (antibiotic) all intravenous (through the vein) stat (immediately). Your blood cultures showed no growth. You were admitted with intravenous (through the vein) fluid at 60 an hour (rate), Zosyn (antibiotic) intravenous (through

Pathophysiology Necrotizing enterocolitis (NEC) is the most common gastrointestinal (GI) medical emergency that occurs in neonates. Although it commonly affects premature babies, it can happen in term babies as well. It usually occurs during the first two to four weeks of life in premature babies and in one to three days for up to one month in term babies. For the premature babies, their organs are not fully developed and it puts them at risk for disease. It is an acute inflammatory disease that

Introduction The increased rates of infection may be attributed to the 3 primary risk factors: hospitalization, changing patterns of antibiotics use, Age > 65 year and more susceptible population. Classically, any exposure to antibiotics (particularly clindamycin, ampicillin or amoxicillin, cephalosporins and fluoroquinolones) was a major risk factor for the development of CDI. However, Hospitalization provides not only a reservoir, but also a vector for transmission. Also, second risk factors may be attributed

HISTORY This is a 67-year-old female who is being admitted at this time for elective port-a-cath removal. The patient had bilateral mastectomies in July of this year by Dr. McMillan; left side was malignant, right side was prophylactic. She underwent a port-a-cath insertion on the right side and underwent radiation and chemotherapy and did reasonably well. Left side was radiated, the right side was not. She came in to see me in clinical follow-up. Clinically her port-a-cath was intact and she has

Every time I purchased tampons I read Toxic Shock Syndrome on the box but never saw the value in furthering my knowledge about it. However, after a lecture where it was briefly brought up along with its intense effects, I decided to interest myself in this infection that has repeatedly crossed my mind. Toxic Shock Syndrome is a dangerous and unstable disease caused by the toxin producing strains of Staphylococcus aureus and Streptococcus pyogenes (group A streptococcus).1 TSS is notoriously associated

Scenario C The patient in this scenario C was suffering with food poisoning caused by Salmonella sp. Non-typhoid Salmonella sp. causes inflammation of the digestive tract (enterocolitis). Common symptoms such as abdominal pain, fever, myalgia and watery non-bloody diarrhoea usually appear between 8 and 72 h after the ingestion of a pathogen (D 'Aoust, 1994). Human salmonellosis is self-limiting infection, nevertheless patients are advised to replace fluid and electrolytes (D 'Aoust, 1994). Sometimes

01/15/2015 S: The patient is a 28y/o (years old) c/o myalgia (pain in muscles), episodes of syncope (loss of consciousness), fever, rash of the face and chest, N and V x 2 days (nausea and vomiting for 2 days), and leukorrhea (a discharge of mucus from the vagina). Patient states she experienced dysmenorrhea (painful ministration with abdominal cramps) for the last 5 days and diarrhea (frequent bowel movements in a liquid form) started today. O: BP: 105/62, T: 100.7, R: 22, P: 96, weight 155, height