Dop Psychology Research Paper

My gene is located at chromosome 9 on the long (q) arm at position 34, 9q34.

The mapping of human genes has allowed for certain genetic disorders to be identified according to the genes that it is affecting. This has created a map that other individuals' genes can be compared to in order to determine any mistakes or any alterations that may lead to the development of a disease. Any changes in epigenetic

Genetics can cause differences in brain chemistry and biochemistry may be important in the development and maintenance of schizophrenia. The dopamine

Genetics can help answer questions about our traits and why we look different and advance in different ways from each other in the world. Chapter 1 explains the basics about how genes work, and the portrayal of DNA and RNA. Chapter 2 describes RNA more in detail and it consist of the explanation of the human genome. Specifically, Chapter 3 clarifies how evolution works and how it relates to genetic and medical research. Furthermore, Chapter 4 and 5 explains the knowledge researchers have about genes role in health and diseases, and how society is affected with the advances in medicine and science given approximate credit to these researchers.

It just may happen to be true that the greatest threats to the safety of young adolescents is young adolescents themselves or perhaps even society’s way of dealing with adolescents. At any age throughout the psychological development of the brain, the thought process is bound to be affected by environmental factors which later appear in manifestations attributed singularly to young adolescents such as binge drinking and higher mobile vehicle fatalities. In teenagers an important environmental factor often dictating their behavior is one simple somewhat expected part of life that modifies their behavior when compared to other age groups: peers. An individual at any age shows the same level of irrationality despite preconceived notions that teenagers and young adults have a higher propensity for such risky behavior. This

Five other gene disorder that contributes to autism are (1) "EN2 (Engrailed 2) involved in cerebellum development. (2) GABR (Gamma Amino Butyric Acid Receptor) regulates brain cell migration. (3) OXTR (Oxytocin Receptor) participating in the response to stress and social skills. (4) RELN (Reelin) involved in neuronal migration in the developing brain. (5) SLC6A4, a serotonin transporter gene” (Johnson, Giarelli, Lewis, & Rice, 2013). As a result of all the researches done several chromosomal loci have been shown to be linked to Autistic Spectrum disorder including those at 2q24-2q31, 7q22-7q31, 7q34-7q36, and 17q11-17q21. Structural chromosomal changes involving deletions and duplication at 7q11, 15q11-15q13, 17p11.2, 22q11.2, and 22q13 have also been associated with forms of autism. However, the most common chromosomal abnormalities currently associated with autism include the fragile X mutation, other sex chromosome abnormalities, and abnormalities of 15q11-q13. “Evidence has shown that duplications of 15q11–q13 have led to higher risks of Autism Spectrum Disorder and developmental and cognitive deficits” (Flashner, Russo, Boileau, Leong, & Gallicano, 2013). Chromosome 15q11-q13.1 region is subject to genomic imprinting, which is an epigenetic process that results in monoallelic gene expression. Duplications lead to autism and are usually maternal in origin. Deletion of the maternal allele of chromosome 15q11-q13 cause Angelman syndrome (AS) a neurodevelopmental disorder

The purpose of this paper is to discuss the effects of the disorder and how genetics and biochemistry work together to create this

The recently completed human genome sequence has greatly assisted the detection of QTLs and polymorphisms (2). It must also be emphasized that genes do not directly dictate action, but rather are mediated by the proteins that they code for. It is necessary to examine not only the genes but also the assortment of proteins responsible for expression of particular traits (3)It is anticipated that detailed analysis of the human genome will contribute to understandings about gene organization and transcription, and hence regulatory elements that control expression. By utilizing genomic and proteomic tools, the relationship between gene/protein and behavior may be more accurately described.

The genome is the complete set of an individual’s inheritable traits or it’s DNA. As a fetus develops, signals are received that cause incremental change in the gene expression patterns. The DNA in our bodies is wrapped around proteins called histone. The histone and DNA are covered in chemical tags. This structure is called an epigenome. The epigenome shapes the structure of the genome. Epigenetic marks are modifications of DNA and histones. The epigenome tightly wraps inactive genes and allows active genes to be more easily accessible. The epigenome adjusts specific genes in response to our changing environment. The programming of neurons through epigenetic mechanisms is critical in neural development. A type of cellular memory is formed when those changes occur. These are epigenetic tags. Each tag records the cell’s experiences on the DNA. This is to help stabilize gene expression. Over time, and with thousands of different experiences, an epigenetic profile forms for each cell type. Each one is unique, with a distinct identity and a specialized function. A flexible epigenome allows us to adjust and learn from our mistakes. The epigenome responds to signals. These signals come from a variety of places. From fetal development to old age, our epigenome is effected by our environmental factors.

Molecular genetics is the study of the chemical nature of the gene itself while genetics, as a whole, is the study of heredity and inherited characteristics. Molecular genetics is more so a branch of the genetics study and expresses how genes and genetic information is encoded, replicated, and expressed. There is a depth induction in the study of molecular genetics to how the processes of replication, transcription, translation, and gene regulation happen and are completed. The study exemplifies how genes are units of heredity that passed down from generation to generation and they encode certain characteristics (Pierce, 2014).

DNA contains the genetic information that makes us who we are, from our genotype to our phenotype. Our genetic makeup can contribute to our behaviour and so much more. However, because we are very complex beings the ways in which our genes contribute to our behaviour also works in a complex manner (Nelson & Israel, 2015). Numerous studies have been conducted to examine the correlation between genetics and behavioral disorders such as ADHD. The purpose of this essay is to examine the role genetics plays in the development of ADHD, the ways in which ADHD can affect development, and the importance of understanding that ADHD has a dimensional nature of diagnosis and works in a continuum.

The gene that we are interested in for this project and for the duration of this semester is DMAP1. Not a lot is known about this is a gene, however in other organisms, it is involved in the methylation of DNA. DMAP1 is responsible for encoding a protein that associates with another protein that methylates DNA. The methylation of DNA alters the DNA structure and makes it unavailable to transcription factors, so the mutation of DMAP1 could lead to changes in transcription and gene regulation. For example, if DMAP1 were mutated, less DNA methylation might occur and more areas of DNA could be accessible to transcription factors and ‘active’, making more genes transcribed than should be. Gene alterations are important as they can lead

Dopamine is a chemical that allows two neurons to communicate (5). When genetic malformations occur, excessive amounts of dopamine enter the synapses resulting in malfunction of the cells in areas of the brain such as the thalamus and the striatum (8). The results can include deficiencies in motor, cognitive, and emotional function (8).

Smoller also explains, “[t]hese resources create opportunities for focusing genetic studies on biological pathways…[in] evaluating the functional significance of risk loci that may [identify] in the future” (Smoller, p. 310). He also distinguished,

It appears, however, that one single gene is not solely responsible for a particular behavior. Instead it seems to take many genes that are combined together to produce both functional and dysfunctional behaviors.