Livedoid Vasculopathy (LV)

Lupus may also affect the blood. Anemia is common in individuals with Lupus, with this condition there is a low read blood cell count, the red blood cells are coated with antibodies that cause them to break down and break apart. Leupkopenia is also common, with this condition there is a low white blood cell count. This can lead to an increased susceptibility to infections. Thrombocytopenia is also common; with this condition people have trouble

BB’s skin presents as pink, warm and dry. No obvious signs or symptoms of abnormal bruising or lesions present however, the patient states that the skin has of late has

The working diagnosis is that this patient is suffering from Angioedema as a result of Anaphylaxis and developing Urticaria. Angioedema is the swelling of deep layers of the skin due the accumulation of fluid, symptoms of Angioedema include swelling of the eyes, lips, hands and feet.

Skin (history of skin disease, pigment or color change, change in mole, excessive dryness or moisture, pruritus, excessive bruising, rash

Lucci the patient had systemic lupus erythematosis, his silver eyebrows pointed to heaven, and he broke out into a big smile. When he asked me to explain why the patient had lupus, I named the fourteen manifestations of lupus and told Dr. Lucci the diagnosis of lupus was contingent on a patient's having at least four of the fourteen manifestations. His patient did indeed have five of the fourteen manifestations: non-deforming arthritis, Raynaud's phenomenon, in which the hands exhibit a two-phase color change when placed in cold water, alopecia or hair loss, oral ulcerations, and what appeared on the chest X-ray to be a pericardial effusion which suggested pericarditis.

My patient is a patient that is leaving the E.R. after being diagnosed with the shingles virus. The patient came into the E.R. with a rash on the right side of the trunk. The rash presented as clear blisters and was painful to the touch. The physician diagnosed the patient with

Vitiligo is a chronic skin disorder that affects the pigmentation of the skin. Pigment is what gives the skin color and is produced by cells called melanocytes. In the disorder, the body attacks the melanocytes resulting in the loss of color in patchy areas. When the color is lost, the affected area becomes a white irregular shaped patch on the outer layer of the skin. Vitiligo can affect a very small portion of the body or affect up to fifty percent of the skin. The most common areas of the body affected by the disorder include the hands, feet, arms, face and sometimes the genitalia. This illness can be treated but it cannot be cured

Peripheral vascular. Client denies cold numb hands or feet, color change, pain in legs while walking, any swelling of the extremities, or hair loss on her peripheral extremities. She denies non-healing sores on the extremities.

“Systemic Lupus Erythematosus is a chronic, inflammatory autoimmune disease in which the immune system attacks the body’s own structures” (Beattie, 2013). This means that the disease that has a slower onset, but it also has a long-term effect and the signs and symptoms will be continuous. Systemic Lupus Erythematosus (SLE) can affect the entire body or any organ in the body, including: the kidneys, swelling of the joints, affecting the blood vessels and the skin.

Systemic Lupus Erythematosus is chronic disease that affects almost all body systems and may result in renal failure. The body produces proteins called autoantibodies that causes inflammation and tissue damage.

Discoid lupus erythematosus (DLE) is a chronic, scarring, atrophic, photosensitive dermatosis. DLE may occur in patients with systemic lupus erythematosus (SLE), and <5% patients with DLE progress to SLE. The primary lesion is an erythematous papule or plaque with slight-to-moderate scaling. As the lesion progresses, the scale may thicken and become adherent, and pigmentary changes may develop. Patients with DLE rarely fulfill four or more of the criteria used to classify SLE. Serologic abnormalities are uncommon. Therapy with sunscreens, topical corticosteroids, and antimalarial agents is usually

Lambert-Eaton myasthenic syndrome, also known as Eaton Lambert syndrome, is an autoimmune disease that is characterized by the onset of severe muscle weakness. The muscle weakness involved with Lambert-Eaton myasthenic syndrome (LEMS) typically occurs in the pelvis and thigh muscles. Approximately 60 percent of LEMS cases are associated with a small cell lung cancer (SCLC), and the onset of LEMS symptoms often precedes the detection of the cancer (Gozzard). Patients who have LEMS that also have cancer tend to have a history of smoking and are usually older adults. LEMS occurs often in men with tumors in their chests, especially lung cancer. Individuals without cancer can develop LEMS at any age. However, LEMS is rarely seen in children and this disease is not hereditary. This disease is also more common in men than in women and the progression depends on whether it occurs with cancer.

Systemic Sclerosis (SSc) is an autoimmune connective tissue disease of unknown etiology resulting in a complex interplay of vasculopathy, inflammation, and fibrosis.[1] More recently, lymphatic microangiopathy was also described.[2,3] Lymphatic vessel involvement seems to start in an early phase of SSc and progresses to a significant loss of lymphatic capillaries in later stages.[3,4] This lymphatic microangiopathy in addition to increased vascular permeability may underlie the de pathophysiology of the early edematous phase frequently observed in SSc, which is most often observed on hands. In patients with diffuse SSc, this feature might be extensive and severe cases of lymphedema have been described.[5,6] The unusual feature of this case

Systemic Lupus Erythematosus is a chronic autoimmune disease which causes inflammation of your joints, tissues, and organs. The inflammation presents itself as heat, pain, swelling and redness. SLE is a variable disease that doesn’t take any one particular course; therefore its unpredictability makes it even more devastating. No two people will experience the same disease symptoms or severity level. As S.L.E progresses there will be periods of very subtle to no symptoms at all called remission or an exacerbation of symptoms called flares.

A nearly significant association between the epidermal nucleo-cytoplasmic expression of LXR-α and the high VIDA scores was noted in studied cases of the present work (P=0.09). It might through a light on the value of LXR-α expression as prognostic markers evaluating the disease severity. Unfortunately, this finding had not been noted before, therefore it is considered as an area requiring additional research.