Glossal pulse oximetry for anaesthetic management of patient with Raynaud’s phenomenon.
Raynaud’s phenomenon, is a disorder of the microvasculature due to vasoconstriction of the digital arteries, arterioles, and cutaneous arteriovenous shunts, affecting the fingers and toes but can present on nose, ears and nipples. Continuous pulse oximetry, a standard basic anaesthesia monitoring as per the American Society of Anaesthesiology (ASA) recommendation, could be challenging for these patients posted for surgery under anaesthesia. We discuss a case of Raynaud’s phenomenon where glossal pulse oximetry was used intraoperatively.
A 65-year-old female, case of adenocarcinoma of lung was posted for radiofrequency ablation under general anaesthesia.
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Pulse oximetry reading could not be recorded by standard finger probe. Ear lobe pulse oximeter showed room air saturation of 95% with weak tracing and poor plethysmograph. Patient was administered general anaesthesia, using injection fentanyl 100 µg, injection propofol 100 mg. The airway was secured using laryngeal mask airway supreme No. 3. No improvement was seen in the ear lobe tracing post anaesthesia. The probe was then placed on lingual surface of tongue, as shown in the picture. Anaesthesia was maintained with oxygen, nitrous oxide and sevoflurane, the case was continued using glossal pulse oximetry monitoring which was switched back to ear lobe prior to removal of supraglottic device. Rest of recovery was …show more content…
High cost of the Nasal Alar SpO2 sensor, £20.62 per unit may deter its use in resource limited settings. Alternatively, invasive arterial line could be inserted to measure arterial blood gases pre-induction, post intubation and following extubation, which could give a better information in such patients posted for major surgeries.
In this era of newer gadgets, we hope that reporting of this case will help in familiarizing the newer generation of anaesthesiologists to this old lost technique. In conclusion, glossal oximetry is a valuable method of intraoperative pulse oximetry in patient with Raynaud’s phenomenon.
References:
1. Coté CJ, Daniels AL, Connolly M, Szyfelbein SK, Wickens CD Tongue oximetry in children with extensive thermal injury: comparison with peripheral oximetry, Can J Anaesth, 1992; 39: 454–7.
2.Hickerson W, Morrell M, Cicala RS. Glossal pulse oximetry. Anesth Analg 1989;
REVIEW OF SYSTEMS;;12 point ROS was preformed and is negative except noted in above HIP, PMH and PSH. Careful attention was paid to endocrine, integumentary, pulmonary, renal and neurological exam
and the pulse oximeter reading is at 88% room air, so the physician ordered 2 to 4 L of
The patient may have a hard time breathing because she is in pain after having surgery. Since they patient doesn’t want to breath due to the pain it can cause atelectasis and later sepsis if not treated in time. It would be important to teach the patient about splinting and to use an incentive spirometry in order to help her be able to breath. Another risk factor for the patient not being able to oxygenate would be hypovolemia since there is less blood volume which can also lead to less oxygen being able to travel in the blood or able to perfuse throughout the body.
Sharon presented with symptoms 2 hours after administration of inhaled Halothane. She had a temperature that elevated to 105 degrees, increased heart rate of 120 bpm and low blood pressure of 60/56. All of these symptoms are abnormal for this patient but can be signs of the condition MH. This condition can be triggered by exposure to volatile inhaled anesthesia drugs like Halothane and signs can be seen during surgery or shortly after surgery (library book, pg510).
At 2330 T.B. spikes a temperature of 38.6° C (tympanic). His SaO2 on 2 L O2/NC is now 90%, so you immediately increase the flow rate to raise his O2 saturation. You inform the on-call surgeon, and she orders a STAT chest x-ray (CXR) and a broad-spectrum antibiotic—imipenem and cilastatin 500 mg IV q6h (check renal function; this medication must be dose adjusted if patient has renal impairment, or there is an increased risk for seizure).
Learning about the potential complications of epidural reinforced my knowledge in being able to choose the right anaesthetic monitoring equipment. Knowing that Spinal and epidural anaesthesia can cause unpredictable and profound arterial hypotension necessitate the use of adequate monitoring like the; Pulse oximetry, ECG and Blood pressure cuff. This knowledge will help me to be able to select appropriate monitoring devices during epidural catheter insertion. Also it goes without saying that an epidural must be performed in a work area that is equipped for airway management and resuscitation.
Both rapid, shallow breathing patterns and hypoventilation effect gas exchange. Arterial blood gases will be monitored and changes discussed with provider. Alteration in PaCO2 and PaO2 levels are signs of respiratory failure. Patient’s body position will be properly aligned for optimum respiratory excursion, this promotes lung expansion and improved air exchange. Patient will be suctioned as needed to clear secretions and maintain patent airways. The expected outcome is that the patient’s airway and gas exchange will be maintained as evidence by normal arterial blood gases (Herdman,
Patient outcome consisted of performing 10 deep breaths per hour. We have reviewed details that were difficult for the patient to remember, such as breathing out before placing the lips on the mouthpiece, and holding breath for 3 to 5 seconds at the top of each inhalation. With empathy, I provided understanding that being hospitalized is never easy due to sensory overload, pain and lack of privacy. Additionally, we have discussed the basic pathophysiology of lung inflammation and what it can do to a person. So overall, the outcome included enhanced disease knowledge with effective use of incentive spirometer.
The primary assessment of this patient is the Airway, Breathing, Circulation, Disability, Exposure (ABCDE) approach. According to the ABCDE approach, it is crucial to check the airway first to see if the airway is patent. Then, check the patient’s breathing. Determining the respiratory rate, inspecting movement of the chest wall, measuring pulse oximeter and using the stethoscope to auscultate the lung sounds are the possible way to check if the breathing of the patient is sufficient (Thim et al., 2012). Next, check the circulation. Skin changes, such as check the colour and sweating, relate to the circulatory problems. Also, heart auscultation, blood pressure measuring and electrocardiography monitoring should be involved in this procedure (Thim et al., 2012). When measure the blood pressure, using the AVPU score (A for ‘alert’, V for ‘reacting to vocal stimuli’, P for ‘reacting to pain’, U for ‘unconscious’) to assess the consciousness, which is for the disability part. Moreover,
Although when it happens, there can be a devastating impact on patients as well as to the multidisciplinary theatre team involved. Consequently, the DAS has produced a consensus set of guidelines for managing failed intubations in adult and paediatric patients, but there are as yet no such nationally-agreed guidelines in obstetrics, therefore each obstetric unit should have their own flowchart with regards to management of failed intubation (Brien and Conlon, 2013). Furthermore, in light of the latest DAS guidelines, several aspects of clinical anaesthetic practise have changed over recent years (Frerk at al, 2015). Amongst the changes are the use of new drugs such as rocuronium and suggamadex and using electronic video-laryngoscopes (Frerk et al, 2015). Further work had also looked at extending the period of apnoea without causing desaturation by optimising the preoxygenation process and adequate patient positioning (Frerk et al, 2015). As a result, updated guidelines for difficult intubations in adult patients were published in 2015; these guidelines provide a flowchart to be used when endotracheal intubation proves difficult or impossible and focus on the central importance of oxygenation while reducing the amount of airway interventions in order to minimize trauma to the delicate airway (Frerk et al, 2015). The main message of the revised guidelines is
Vasoplegic syndrome is a severe vasodilatory shock characterised by hypotension, tachycardia, normal or elevated cardiac output, decrease in systemic vascular resistance, poor or no response to fluid resuscitation and vasopressor administration [1]. Though it is commonly seen during cardiac surgery, it is also been reported during non cardiac surgery [2,3]. The incidence of vasoplegic syndrome is 8-10% in cardiac surgery [4], and its risk factors include intravenous heparin, beta-blockers, calcium channel blockers, renin angiotensin system antagonists, protamine use, myocardial dysfunction, diabetes mellitus, presence of pre-cardiopulmonary bypass hemodynamic instability, increased duration of cardiopulmonary bypass and ventricular device insertion [5]. We present a case of successful management of vasoplegic syndrome the developed perioperatively following Whipple’s procedure.
The primary nursing diagnosis for this patient is impaired gas exchange, related to abnormal ventilation and perfusion ratio, as evidenced by restlessness, irritability, anxiety, decreased level of consciousness, abnormal arterial blood gases, and abnormal skin color (Gulanick & Myers, 2014, p. 82). A.C. has an endotracheal tube (ETT), and there is a note for the next day to have surgery to put in a tracheostomy. She is currently a smoker, her C02 is 74.6mEq/L which is high, her pH is low at 7.19, and the bicarbonate is 28.6mEq/L which is high. Her oxygen saturation is maintaining at 90%. Her PA02 is 56mm Hg and FI02 is 0.60. The patient is very anxious and restless in the bed, despite sedation and pain medication, and her skin is pale in color and she is diaphoretic.
The nurse found Mrs Smith to be tachypnoeic, her respirations were recorded as 24 breaths per minute it was observed as being fast and it appeared that her accessory muscles were being used. Mrs Smith’s pallor also appeared flushed and her saturations were documented as 93%. The nurse used the stethoscope to check for wheeze the patient’s lungs were clear and chest rise was symmetrical. Mrs Smith was commenced on 100% oxygen through a non-rebreathe mask, oxygen as an intervention is necessary as Creed & Spiers (2010) highlight ‘metabolic demand for oxygen throughout the body is hugely increased by sepsis and is essential to ensure the supply of oxygen is maximized’ .The nurse monitored the patient closely because in her confused state the patient may try to remove the oxygen mask.
Side effects of radiographic contrast media extent from a moderate incommode, such as itching, to a life-threatening emergency. The well-known unpropitious reaction associated is contrast-induced nephropathy (CIN) with the use of intravenous or intra-arterial contrast material. Delayed allergic reactions, anaphylactic reactions, and cutaneous reactions is the other configuration of deleterious reactions encompass. The possibility of deteriorate adverse reactions to contrast agents elevating due to antecedent allergic reactions. Hypersensitivity reactions and thyroid dysfunction is the primarily significant adverse effects of contrast media. Moderate hypersensitivity reactions subsist of immediate skin rashes, redden, rhinorrhoea and
After being reminded by the instructor, I was aware of my mistakes and noticed that I failed to maintain patient’s safety. An oxygen below 90% can be very dangerous for the patient, especially for a post-op day #1 patient, because prolonged hypoxemia can cause fatigue, headache, acute respiratory failure, cardiac problems (increased heart rate,