Right Ventricle Lab Report

The backward leak of blood from the aorta to left ventricle during diastole increases left ventricular volume. The left ventricle accommodates extra volume of blood by increasing ventricular size. This regurgitation leads to impaired forward systemic blood flow reducing cardiac output. Left ventricle increases ejection during early part of systole to compensate this. In increased regurgitation, left ventricular pressure increases, which may leads to increased left atrial pressure and pulmonary congestion.

Both the right and left atrium contract causing blood to flow though the two valves, and then into the left ventricle. The left ventricle pumps blood into the systemic circulation through the aorta. This systemic circulation system is much bigger than the pulmonary circulation system, which is why the left ventricle is so big. The blood on the left side of the heart is oxygenated. It becomes oxygenated when the deoxygenated blood passes through the right atrium and then flows into the left ventricle. It is then pumped along the pulmonary artery into the lungs where it is oxygenated. It then travels through the pulmonary veins back into the heart. It enters through the left atrium and then travels to the left ventricle. This process is repeated over and over again, to make blood continuously flow through the heart, lungs and body. This process ensures that there is always enough oxygen for the body to work

Likewise, Blood flows from the right atrium to the right ventricle, and then is pumped to the lungs to receive oxygen. From the lungs, the blood flows to the left atrium, then to the left ventricle, forming the complete circulation.

After a period of time, the heart muscles of the left ventricle begin to weaken. The weakening of the left ventricle will lead to decreased empting of the heart (systolic heart failure) which results in decreased cardiac output again. Since the left ventricle does not empty completely, blood begins to back up into the left atrium and then to the pulmonary circulation thus resulting in pulmonary congestion and dyspnea (Story 2012, 104). If left untreated, the blood will back up and affect the right side of the heart causing biventricular heart failure (both right and left heart failure). In right sided heart failure, the right ventricle weakens and cannot empty completely. This incomplete emptying causes blood to back up into the systemic circulation causing systemic edema (Lewis et al. 2014, 771).

1. The pulmonary circuit is supplied by which ‘side” of the heart? The systemic circuit? The right atrium

Deacreased vascular resistance and increased arterial pressure causes an increase in blood flow. This is important to supply organs with oxygen. 4. Restate your predictions that were correct and give data from your experiment that support them. Restate your predictions that were not correct and correct them with supporting data from your experiment. MAP would increase due to increase in activity, SVR would decrease due to decrease in resistance, CO would increase due to more force of blood being expelled.

In systolic ventricular dysfunction or systolic heart failure the heart is not able to produce enough output for adequate tissue perfusion. Heart rate and stroke volume produce cardiac output. Contractility, preload, and afterload influence the heart’s stroke volume. These factors are important in understanding the pathophysiologic consequences of this syndrome and possible treatments. Patients with systolic heart failure usually have dilated, large ventricles and impaired systolic function.

In a normal human being the heart correctly functions by the blood first entering through the right atrium from the superior and inferior vena cava. This blood flow continues through the right atrioventricular valve into the right ventricle. The right ventricle contracts forcing the pulmonary valve to open leading blood flow through the pulmonary valve and into the pulmonary trunk. Blood is then distributed from the right and left pulmonary arteries to the lungs, where carbon dioxide is unloaded and oxygen is loaded into the blood. The blood is returned from the lungs to the left

Oxygen poor blood fills the right atrium from either the superior or inferior vena cava.

The increased resistance of blood flow through the pulmonary semilunar valve from the right ventricle backs up the pressure of blood

It involves the tightening of blood vessels connected to and within the lugs. This makes it harder for the heart to pump blood thorough the lungs, much as it is harder to make water flow through a narrow pipe as opposed to a wide one. Over time, the affected blood vessels become both stiffer and thicker, further increasing the blood pressure within the lungs and impairing blood flow. In addition, the increase workload of the heart causes thickening and enlargement of the right ventricle, making the heart less able to pump blood through the lungs, causing right heart failure. As the blood flowing through the lungs decreases, the left side of the heart receives less blood. This blood may also carry less oxygen than normal. Therefore it becomes harder and harder for the left side of the heart to pump to supply sufficient oxygen to the rest of the body, especially during physical activity.

The S-A node signal is delayed by the atrioventricular node to allow the full contraction of the atria that allows the ventricles to reach their maximum volume. A sweeping right to left wave of ventricular contraction then pumps blood into the pulmonary and systemic circulatory systems. The semilunar valves that separate the right ventricle from the pulmonary artery and the left ventricle from the aorta open shortly after the ventricles begin to contract. The opening of the semilunar valves ends a brief period of isometric (constant volume) ventricular contraction and initiates a period of rapid ventricular ejection.

Blood is one of the most vital components of the human body. The blood carries many functions such as to supply oxygen to the bodies tissues, remove metabolic waste products, regulate our core temperature as well as fighting infection and foreign bodies (Glover, 1997). The cardiovascular system is composed of the heart and its vessels. The heart is an involuntary muscle which receives blood to the atrias, which is then pumped via the ventricles. The vessels are composed of three main types. Arteries, veins and capillaries; all which transport blood throughout the entirety of the body. The constant action of both the vessels and heart ensure that the body receives a continuous supply of blood, keeping us within our homeostatic limits.

reports the amount of force exerted by the blood into the arteries during ventricular contraction.

The amount of blood pumped out during systole is called the stroke volume and is less than the end diastolic volume because the ventricles do not completely empty themselves during systole. At all levels of physical activity stroke volume is increased. There is an improvement in ventricular performance with an increase of plasma volume [4] and a faster peak lengthening the rate of the left ventricle during diastole [6]. Training can improve stroke volume but by no more then about 20%. Due to the decreased heart rate an increase of ventricular filling will result and an increase in ventricular volume and thickening of ventricular walls thus