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1. A patient has an inborn mutation causing the inability to produce succinate dehydrogenase required for the oxidation of FADH2. Will this person still produce ATP? Explain your answer. 2 Mutations in the genes that encode certain mitochondrial proteins are associated with a high incidence of some type of cancer. How might defective mitochondria lead to cancer? 3. How is cytochrome c related to apoptosis? 4. Fill in the table below Number of ATP produced from one glucose molecule Pathway Number of H carriers produced from one glucose molecule NADH FADH2 Glycolysis Krebs Cycle Electron Transport Chain Total ATP Produced

1. A patient has an inborn mutation causing the inability to produce succinate dehydrogenase required for the oxidation of FADH2. Will this person still produce ATP? Explain your answer. 2 Mutations in the genes that encode certain mitochondrial proteins are associated with a high incidence of some type of cancer. How might defective mitochondria lead to cancer? 3. How is cytochrome c related to apoptosis? 4. Fill in the table below Number of ATP produced from one glucose molecule Pathway Number of H carriers produced from one glucose molecule NADH FADH2 Glycolysis Krebs Cycle Electron Transport Chain Total ATP Produced

Biochemistry
Biochemistry
6th Edition
ISBN:9781305577206
Author:Reginald H. Garrett, Charles M. Grisham
Publisher:Reginald H. Garrett, Charles M. Grisham
Chapter19: The Tricarboxylic Acid Cycle
Section: Chapter Questions
Problem 19P
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ACTIVITY 10.6.2 Provide what is being asked. 1. A patient has an inborn mutation causing the inability to produce succinate dehydrogenase required for the oxidation of FADH2. Will this person still produce ATP? Explain your answer. 2 Mutations in the genes that encode certain mitochondrial proteins are associated with a high incidence of some type of cancer. How might defective mitochondria lead to cancer? 3. How is cytochrome c related to apoptosis? 4. Fill in the table below Pathway Number of ATP produced from one glucose molecule Number of H carriers produced from one glucose molecule NADH FADH2 Glycolysis Krebs Cycle Electron Transport Chain Total ATP Produced
Transcribed Image Text:ACTIVITY 10.6.2 Provide what is being asked. 1. A patient has an inborn mutation causing the inability to produce succinate dehydrogenase required for the oxidation of FADH2. Will this person still produce ATP? Explain your answer. 2 Mutations in the genes that encode certain mitochondrial proteins are associated with a high incidence of some type of cancer. How might defective mitochondria lead to cancer? 3. How is cytochrome c related to apoptosis? 4. Fill in the table below Pathway Number of ATP produced from one glucose molecule Number of H carriers produced from one glucose molecule NADH FADH2 Glycolysis Krebs Cycle Electron Transport Chain Total ATP Produced
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